We are analyzing https://link.springer.com/article/10.1007/s40336-014-0098-3.

Title:
Amyloid PET imaging: applications beyond Alzheimer’s disease | Clinical and Translational Imaging
Description:
As a biomarker of beta-amyloid, positron emission tomography (PET) amyloid imaging offers a unique opportunity to detect the presence of this protein in the human body during life. Besides Alzheimer’s disease (AD), deposits of beta-amyloid in the brain are also present in other neurodegenerative diseases associated to dementia, such as Parkinson’s disease and dementia with Lewy bodies, as well as in other processes affecting brain function, such as cerebral amyloid angiopathy, brain trauma, Down’s syndrome and meningiomas, as shown by post-mortem pathology studies. Furthermore, in systemic amyloidosis other organs besides the brain are affected, and amyloid PET imaging may be suitable for the identification of these extra-cerebral amyloid depositions. Finally, the potential use of amyloid PET tracer accumulation in cerebral white matter (WM) as a marker of myelin is being investigated, leading to some promising results in patients with WM lesions and multiple sclerosis. In this article, a review of the ongoing research pointing to a broader application of amyloid PET imaging in clinical practice beyond AD is provided.
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Keywords {🔍}

amyloid, pet, imaging, article, pubmed, google, scholar, cpib, patients, betaamyloid, brain, disease, dementia, cas, clinical, dlb, uptake, deposition, reported, studies, cerebral, neurol, alzheimers, caa, med, tbi, binding, central, lewy, amyloidosis, found, patient, pathology, lesions, study, potential, tracer, subjects, table, results, diagnosis, nucl, higher, body, angiopathy, pdd, positive, age, diseases, syndrome,

Topics {✒️}

article download pdf full size image perivascular beta-amyloid depositions 18f-fdg pet scans 18f-labeled amyloid tracers extra-cerebral amyloid depositions registered 11c-pib slices positron emission tomography pro-caa position statement blood–brain barrier disruption show beta-amyloid plaques cortical beta-amyloid deposition 11c-pib retention index striatal 11c-pib retention 11c-pib positive scans global 11c-pib retention positive 18f-florbetapir uptake amyloid-ss-directed immunotherapy lower 11c-pib retention c-pib pet imaging magnetic resonance imaging 11c-pib pet imaging peptide-related cerebral amyloidosis anti-beta-amyloid immunotherapy regional beta-amyloid depositions brain beta-amyloid depositions cerebral beta-amyloid deposition 18f-flutemetamol pet/ct parenchymal beta-amyloid plaques differential 11c-pib retention 3h-pib autoradiography performed regional 11c-pib binding negative 11c-pib scan 11c-pib uptake patterns reduced 11c-pib uptake substantial beta-amyloid deposition jack cr jr brain beta-amyloid deposition global 11c-pib binding global 11c-pib ratio efficacious disease-modifying drugs 11c-pib uptake compared brain β-amyloid deposition diffuse beta-amyloid deposition beta-amyloid deposition patterns progressive beta-amyloid accumulation higher 11c-pib uptake cortical gray matter cerebral white matter [18f]florbetaben pet study

Questions {❓}

Aarsland D, Ballard CG, Halliday G (2004) Are Parkinson’s disease with dementia and dementia with Lewy bodies the same entity?
Chen W, Dilsizian V (2012) Molecular imaging of amyloidosis: will the heart be the next target after the brain?
Jellinger KA, Attems J (2006) Does striatal pathology distinguish Parkinson disease with dementia and dementia with Lewy bodies?
Lhommel R, Sempoux C, Ivanoiu A, Michaux L, Gerber B (2014) Is 18F-flutemetamol PET/CT able to reveal cardiac amyloidosis?

Schema {🗺️}

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      headline:Amyloid PET imaging: applications beyond Alzheimer’s disease
      description:As a biomarker of beta-amyloid, positron emission tomography (PET) amyloid imaging offers a unique opportunity to detect the presence of this protein in the human body during life. Besides Alzheimer’s disease (AD), deposits of beta-amyloid in the brain are also present in other neurodegenerative diseases associated to dementia, such as Parkinson’s disease and dementia with Lewy bodies, as well as in other processes affecting brain function, such as cerebral amyloid angiopathy, brain trauma, Down’s syndrome and meningiomas, as shown by post-mortem pathology studies. Furthermore, in systemic amyloidosis other organs besides the brain are affected, and amyloid PET imaging may be suitable for the identification of these extra-cerebral amyloid depositions. Finally, the potential use of amyloid PET tracer accumulation in cerebral white matter (WM) as a marker of myelin is being investigated, leading to some promising results in patients with WM lesions and multiple sclerosis. In this article, a review of the ongoing research pointing to a broader application of amyloid PET imaging in clinical practice beyond AD is provided.
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