We are analyzing https://link.springer.com/article/10.1007/s13539-011-0049-z.

Title:
Myostatin blockage using actRIIB antagonism in mice bearing the Lewis lung carcinoma results in the improvement of muscle wasting and physical performance | Journal of Cachexia, Sarcopenia and Muscle
Description:
Background Cachexia is a multiorganic syndrome associated with cancer, characterized by body weight loss, muscle and adipose tissue wasting and inflammation. Methods The aim of this investigation was to examine the effect of the soluble receptor antagonist of myostatin (sActRIIB) in cachectic tumor-bearing animals analyzing changes in muscle proteolysis and in quality of life. Results Administration of sActRIIB resulted in an improvement in body and muscle weights. Administration of the soluble receptor antagonist of myostatin also resulted in an improvement in the muscle force. Conclusions These results suggest that blocking myostatin pathway could be a promising therapeutic strategy for the treatment of cancer cachexia.
Website Age:
28 years and 1 months (reg. 1997-05-29).

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Keywords {🔍}

muscle, article, google, scholar, pubmed, cas, myostatin, cachexia, mice, cancer, treatment, skeletal, sactriib, lung, table, wasting, animals, carcinoma, lewis, weight, tumor, growth, bearing, results, physical, busquets, protein, increase, barcelona, body, tumorbearing, force, activity, system, access, receptor, proteolysis, role, effects, spain, reverse, lopezsoriano, full, resulted, expression, analysis, journal, performance, open, loss,

Topics {✒️}

greater ubiquitin–proteasome-dependent proteolysis amino-terminal pro-peptide carboxy-terminal mature region ubiquitin-dependent proteolytic pathway atp-ubiquitin-dependent proteolysis tgf-β family members ubiquitin-dependent proteolytic system myostatin anti-sense rna regular light–dark cycle calcium-dependent proteases participate article download pdf lewis lung carcinoma tgf-beta superfamily member pcr real-time analysis transcriptor reverse transcriptase open access license ubiquitin-proteasome pathway comparative ct method suppressing foxo3 action atrophy-specific gene transcription total physical activity curr opin pharmacol departament de bioquímica universitat de barcelona tumor-bearing mice treated treated tumor-bearing mice mitochondria-mediated pathway full-length precursor activin iib receptor privacy choices/manage cookies ternary signaling complex open access ministerio de ciencia metastatic lung area myostatin signaling acts drug discov today full access total traveled distance argiles jm facultat de biologia sactriib sequesters activin c8 proteasome subunit measure physical performance common systemic manifestations high glycolytic rate physiol cell physiol single-step method grip strength meter blocking myostatin pathway triangular pull bar

Questions {❓}

DNA fragmentation occurs in skeletal muscle during tumor growth: a link with cancer cachexia?
Why do cancer cells have such a high glycolytic rate?

Schema {🗺️}

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         description:Cachexia is a multiorganic syndrome associated with cancer, characterized by body weight loss, muscle and adipose tissue wasting and inflammation. The aim of this investigation was to examine the effect of the soluble receptor antagonist of myostatin (sActRIIB) in cachectic tumor-bearing animals analyzing changes in muscle proteolysis and in quality of life. Administration of sActRIIB resulted in an improvement in body and muscle weights. Administration of the soluble receptor antagonist of myostatin also resulted in an improvement in the muscle force. These results suggest that blocking myostatin pathway could be a promising therapeutic strategy for the treatment of cancer cachexia.
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            Molecular Medicine
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      headline:Myostatin blockage using actRIIB antagonism in mice bearing the Lewis lung carcinoma results in the improvement of muscle wasting and physical performance
      description:Cachexia is a multiorganic syndrome associated with cancer, characterized by body weight loss, muscle and adipose tissue wasting and inflammation. The aim of this investigation was to examine the effect of the soluble receptor antagonist of myostatin (sActRIIB) in cachectic tumor-bearing animals analyzing changes in muscle proteolysis and in quality of life. Administration of sActRIIB resulted in an improvement in body and muscle weights. Administration of the soluble receptor antagonist of myostatin also resulted in an improvement in the muscle force. These results suggest that blocking myostatin pathway could be a promising therapeutic strategy for the treatment of cancer cachexia.
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         Cancer cachexia
         Skeletal muscle
         Physical activity
         Muscle force
         Ubiquitin
         Internal Medicine
         Oncology
         Geriatrics/Gerontology
         Clinical Nutrition
         Molecular Medicine
         Pharmacology/Toxicology
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      name:Institut de Biomedicina de la Universitat de Barcelona (IBUB), Barcelona, Spain
      name:Departments of Metabolic Disorders and Protein Science, Amgen Research, Thousand Oaks, USA
      name:Cancer Research Group, Departament de Bioquímica i Biologia Molecular, Facultat de Biologia, Universitat de Barcelona, Barcelona, Spain
      name:Institut de Biomedicina de la Universitat de Barcelona (IBUB), Barcelona, Spain

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