We are analyzing https://link.springer.com/article/10.1007/s13105-020-00736-2.

Title:
Impact of adipokines and myokines on fat browning | Journal of Physiology and Biochemistry
Description:
Since the discovery of leptin in 1994, the adipose tissue (AT) is not just considered a passive fat storage organ but also an extremely active secretory and endocrine organ that secretes a large variety of hormones, called adipokines, involved in energy metabolism. Adipokines may not only contribute to AT dysfunction and obesity, but also in fat browning, a process that induces a phenotypic switch from energy-storing white adipocytes to thermogenic brown fat–like cells. The fat browning process and, consequently, thermogenesis can also be stimulated by physical exercise. Contracting skeletal muscle is a metabolically active tissue that participates in several endocrine functions through the production of bioactive factors, collectively termed myokines, proposed as the mediators of physical activity–induced health benefits. Myokines affect muscle mass, have profound effects on glucose and lipid metabolism, and promote browning and thermogenesis of white AT in an endocrine and/or paracrine manner. The present review focuses on the role of different myokines and adipokines in the regulation of fat browning, as well as in the potential cross-talk between AT and skeletal muscle, in order to control body weight, energy expenditure and thermogenesis.
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Keywords {🔍}

pubmed, article, google, scholar, cas, central, adipose, tissue, cell, brown, leptin, fat, frühbeck, obesity, white, muscle, physiol, metab, human, gómezambrosi, browning, adipocytes, rodríguez, endocrinol, catalán, mice, clin, thermogenesis, metabolic, nitric, expression, myokines, cells, oxide, nature, becerril, skeletal, med, adipocyte, beige, spiegelman, energy, nat, diabetes, endocrine, metabolism, exercise, role, prdm, fgf,

Topics {✒️}

cocaine-amphetamine-regulated-transcript ck month download article/chapter leptin-deficient ob/ob mice β3-adrenergic receptor agonist pgc-1α isoform induced fibroblast growth factor-21 calsyntenin 3β-s100b axis surgically-induced weight loss adipokine zinc-α2-glycoprotein short-term interval training pgc1-α-dependent myokine leptin-induced lipolysis opposes peroxynitrite-mediated oxidative stress diet-induced insulin resistance fgf21 regulates pgc-1α obese insulin-resistant subjects white-brown adipocyte plasticity reduces diet-induced obesity hfd-induced obese mice high-fat-diet feeding high-intensity interval exercise potential cross-talk cold-inducible coactivator regulates immune-adipose interactions cold-induced ucp1 expression full article pdf nitric oxide-dependent mechanisms energy-storing white adipocytes zinc-α 2-glycoprotein brown adipogenic differentiation drives brown-fat leptin-stat3-prdm16 signal alternatively activated macrophages zinc-α2-glycoprotein lipid mobilizing factor gobierno de navarra human skeletal muscle obesity-related metabolic syndrome proopiomelanocortin ppar-γ contracting skeletal muscle diet-induced resistance beige adipose tissue van der heide privacy choices/manage cookies fgf21 regulates metabolism adipocyte hyperplasia induced resistance exercise-induced increasing pgc-1α fisher fm major risk factor

Questions {❓}

Frühbeck G, Becerril S, Sáinz N, Garrastachu P, Garcia-Velloso MJ (2009) BAT: a new target for human obesity?
Gomez-Ambrosi J, Catalan V, Rodriguez A, Salvador J, Fruhbeck G (2015) Does body adiposity better predict obesity-associated cardiometabolic risk than body mass index?
Kolb H, Kolb-Bachofen V (1998) Nitric oxide in autoimmune disease: cytotoxic or regulatory mediator?
Kristiansen OP, Mandrup-Poulsen T (2005) Interleukin-6 and diabetes: the good, the bad, or the indifferent?
Rodríguez A, Catalán V, Gómez-Ambrosi J, Frühbeck G (2007) Visceral and subcutaneous adiposity: are both potential therapeutic targets for tackling the metabolic syndrome?
Wilcox CS (2005) Oxidative stress and nitric oxide deficiency in the kidney: a critical link to hypertension?

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