We are analyzing https://link.springer.com/article/10.1007/s12035-013-8416-8.

Title:
Glaucoma: An Extension of Various Chronic Neurodegenerative Disorders | Molecular Neurobiology
Description:
The eyes are the window of the brain; various pathologies of the eyes can be explained on the basis of neurological disorders and vice versa. Today, our only approach for the management of glaucoma is intraocular pressure (IOP) reduction, whether by pharmacological or surgical methods. Available methods may effectively control IOP but fall short of stopping the progression of glaucoma. IOP is just one of the risk factors our therapy addresses, so we need to work towards shifting our approach towards addressing other factors involved in the etiopathogenesis of the disease. Pathogenesis of glaucoma at molecular level can be compared with that of common neurodegenerative diseases like Alzheimer’s and Parkinsonism. Targets for therapeutic intervention in chronic neurodegenerative diseases and glaucoma include apoptosis, axonal transport, free radical damage, role of polyunsaturated fatty acids, chaperones, gene regulation, etc. p38MAPK is a common pathway involved in all the neurodegenerative disease and glaucoma as well, and more research in this direction can be really beneficial for the development of novel diagnostic and therapeutic measures.
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28 years and 1 months (reg. 1997-05-29).

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Keywords {🔍}

google, scholar, article, pubmed, cas, glaucoma, disease, neurodegenerative, alzheimers, ophthalmol, diseases, brain, acid, fatty, vis, sci, patients, parkinsons, privacy, cookies, content, jindal, access, glaucomatous, retinal, amyloid, precursor, protein, salem, publish, research, search, molecular, vishal, polyunsaturated, optic, ocular, mol, curr, activation, ferrari, neurol, mouse, signaling, neurochem, kinase, calon, docosahexaenoic, dietary, function,

Topics {✒️}

month download article/chapter stress-activated map kinase toxin-induced neuronal degeneration optic nerve head rat ocular hypertension glaucomatous optic neuropathy staurosporine-induced apoptosis quigley ha chronic neurodegenerative diseases common neurodegenerative diseases normal-tension glaucoma polyunsaturated fatty acids glaucoma include apoptosis open-angle glaucoma full article pdf omega-3 fatty acids retinal diseases phosphatidylinositol-3 kinase pathway map kinase pathways amyloid precursor protein amyloid precursor protein privacy choices/manage cookies mckinnon sj neurodegenerative diseases glaucomatous neurodegeneration retinal tau pathology chronic neurodegenerative disorders related subjects reduced retinal function transgenic mouse model calcineurin/nfat signaling article jindal docosahexaenoic acid protects european economic area free radical damage apoptotic cell death cortical motor neurons early presymptomatic stages amyotrophic lateral sclerosis manning-bog ab decreases nmda receptors targeting amyloid-β amyloid-β immunoreactivity conditions privacy policy kerrigan-baumrind la common pathway involved high occurrence rate vitreous fluid levels increased tau levels decreased β-amyloid1–42

Questions {❓}

Halliwell B (2006) Oxidative stress and neurodegeneration: where are we now?

Schema {🗺️}

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         description:The eyes are the window of the brain; various pathologies of the eyes can be explained on the basis of neurological disorders and vice versa. Today, our only approach for the management of glaucoma is intraocular pressure (IOP) reduction, whether by pharmacological or surgical methods. Available methods may effectively control IOP but fall short of stopping the progression of glaucoma. IOP is just one of the risk factors our therapy addresses, so we need to work towards shifting our approach towards addressing other factors involved in the etiopathogenesis of the disease. Pathogenesis of glaucoma at molecular level can be compared with that of common neurodegenerative diseases like Alzheimer’s and Parkinsonism. Targets for therapeutic intervention in chronic neurodegenerative diseases and glaucoma include apoptosis, axonal transport, free radical damage, role of polyunsaturated fatty acids, chaperones, gene regulation, etc. p38MAPK is a common pathway involved in all the neurodegenerative disease and glaucoma as well, and more research in this direction can be really beneficial for the development of novel diagnostic and therapeutic measures.
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