We are analyzing https://link.springer.com/article/10.1007/s12026-008-8049-6.

Title:
Neutrophil apoptosis and the resolution of infection | Immunologic Research
Description:
Polymorphonuclear leukocytes (PMNs) are the most abundant white cell in humans and an essential component of the innate immune system. PMNs are typically the first type of leukocyte recruited to sites of infection or areas of inflammation. Ingestion of microorganisms triggers production of reactive oxygen species and fusion of cytoplasmic granules with forming phagosomes, leading to effective killing of ingested microbes. Phagocytosis of bacteria typically accelerates neutrophil apoptosis, which ultimately promotes the resolution of infection. However, some bacterial pathogens alter PMN apoptosis to survive and thereby cause disease. Herein, we review PMN apoptosis and the ability of microorganisms to alter this important process.
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Keywords {🔍}

google, scholar, pubmed, cas, apoptosis, human, neutrophils, immunol, cell, neutrophil, infect, macrophages, death, immun, biol, cells, macrophage, apoptotic, infection, receptor, mycobacterium, blood, role, phagocytosis, expression, med, activation, tuberculosis, necrosis, microbiol, clin, inflammation, leukoc, exp, leukocytes, regulation, caspase, deleo, polymorphonuclear, bacterial, induces, induction, protein, tolllike, ligand, immune, granulocytes, invest, tumor, induced,

Topics {✒️}

p38-mitogen-activated protein kinase tnf-related apoptosis-inducing ligand tnf-relating apoptosis-inducing ligand type iii secretion-dependent early-onset pma-induced apoptosis extracellular signal-regulated kinase methicillin-resistant staphylococcus aureus fe-superoxide dismutase mutants apoptosis-inducing factor participation month download article/chapter tumor necrosis factor-α polyglutamine-induced lc3 conversion growth factors g-csf fourteen-member macrolides promote panton-valentine leukocidin mycobacterium tuberculosis-induced apoptosis pubmed google scholar aspirin-triggered lipoxins override pseudomonas aeruginosa-infected macrophages tumor necrosis factor-alpha heat-killed staphylococcus aureus mannheimia haemolytica leukotoxin nuclear factor-κb activation superoxide anion-generating system bad-mediated signalling pathway induces nf-κb activation mycobacterium bovis bcg mitochondria-dependent death pathway staphylococcal phenol-soluble modulin induced bax-independent apoptosis ifn-gamma independent manner reactive oxygen intermediates-independent fas ligand-induced apoptosis interferon-activated neutrophils store ifn-γ-inducible protein-10 mek1-erk1/2 pathways downstream gutierrez-pabello ja caspase-independent death pathway p2z-receptor-mediated necrosis clostridium difficile toxin pro-resolution lipid mediators human monocyte-derived macrophages myeloperoxidase––h2o2––halide system cytokine-mediated bax deficiency classical protein kinase il-1 beta-converting enzyme host-pathogen interactions adenylate cyclase-hemolysin adenylate cyclase hemolysin tumor necrosis factor

Questions {❓}

Apoptosis: a possible tactic of Haemophilus somnus for evasion of killing by bovine neutrophils?
How do neutrophils and pathogens interact?
Increased expression of Fas ligand on Mycobacterium tuberculosis infected macrophages: a potential novel mechanism of immune evasion by Mycobacterium tuberculosis?
Is Panton-Valentine Leukocidin the major virulence determinant in community-associated methicillin-resistant Staphylococcus aureus disease?
What can we learn from highly purified neutrophils?

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         Allergology
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