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Title:
The enzymatic poly(gallic acid) reduces pro-inflammatory cytokines in vitro, a potential application in inflammatory diseases | Inflammation
Description:
Cytokines like IL-6, TNF-α, and IL-1β are important mediators of inflammation in many inflammatory diseases, as well as in cellular processes like cell proliferation and cell adhesion. Finding new molecules that decrease cell proliferation, adhesion (inflammatory infiltrate), and pro-inflammatory cytokine release could help in the treatment of many inflammatory diseases. The naturally derived poly(gallic acid) (PGAL), produced enzymatically from gallic acid in aqueous medium, is a non-toxic, thermostable multiradical polyanion that is antioxidant and has potential biomedical uses. Experimental evidence has demonstrated that PGAL reduces pro-inflammatory cytokines, which are the target of some inflammatory diseases. PGAL decreased IL-6, TNF-α, and IL-1β production in human monocytes exposed to PMA without affecting cell viability. Additionally, PGAL reduced cell proliferation by affecting the transition from the S phase to the G2 phase of the cell cycle. Cell adhesion experiments showed that PMA-induced cell adhesion was diminished with the presence of PGAL, particularly at a concentration of 200 μg/mL. These properties of PGAL show a potential use for treating inflammatory diseases, such as psoriasis or arthritis.
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Keywords {🔍}
article, pubmed, google, scholar, cas, acid, cell, méxico, ibarra, inflammation, proinflammatory, cytokines, inflammatory, potential, pgal, ciudad, journal, research, polygallic, diseases, proliferation, adhesion, gallic, nacional, instituto, privacy, cookies, content, data, gimeno, arthritis, international, immunopharmacology, cells, information, publish, search, vitro, zamudiocuevas, miquel, access, biological, rheumatoid, wang, antiinflammatory, author, rehabilitación, luis, guillermo, springer,
Topics {✒️}
karina martínez-flores cristina velasquillo-martínez valentín martínez-lópez roberto sánchez-sánchez reduces pro-inflammatory cytokines pro-inflammatory cytokines tnfalpha pro-inflammatory cytokine release month download article/chapter pma-induced cell adhesion article zamudio-cuevas miquel gimeno author information authors pro-inflammatory cytokines pi3k/akt/mtor pathway proyectos de investigación decrease cell proliferation clemente ibarra fluoxetine-induced oxidative stress enzyme-mediated polygallic acid ciudad de méxico topical anti-inflammatory potential ariadna aparicio-juárez cell proliferation programa de apoyo facultad de química full article pdf induce cell death cell adhesion privacy choices/manage cookies thp-1 cell line affecting cell viability edson aguillón-solís roeb garcía-arrazola polyconjugated semiconducting material article inflammation aims vitro cell model check access related subjects redox-sensitive kinase instant access treating inflammatory diseases anti-inflammatory activity anti-inflammatory potential european economic area erika ruvalcaba-paredes thermostable multiradical polyanion human monocytes exposed polyphenol–metal network pox/plga scaffold immunosuppressive/autoimmune disorders
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description:Cytokines like IL-6, TNF-α, and IL-1β are important mediators of inflammation in many inflammatory diseases, as well as in cellular processes like cell proliferation and cell adhesion. Finding new molecules that decrease cell proliferation, adhesion (inflammatory infiltrate), and pro-inflammatory cytokine release could help in the treatment of many inflammatory diseases. The naturally derived poly(gallic acid) (PGAL), produced enzymatically from gallic acid in aqueous medium, is a non-toxic, thermostable multiradical polyanion that is antioxidant and has potential biomedical uses. Experimental evidence has demonstrated that PGAL reduces pro-inflammatory cytokines, which are the target of some inflammatory diseases. PGAL decreased IL-6, TNF-α, and IL-1β production in human monocytes exposed to PMA without affecting cell viability. Additionally, PGAL reduced cell proliferation by affecting the transition from the S phase to the G2 phase of the cell cycle. Cell adhesion experiments showed that PMA-induced cell adhesion was diminished with the presence of PGAL, particularly at a concentration of 200 μg/mL. These properties of PGAL show a potential use for treating inflammatory diseases, such as psoriasis or arthritis.
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immunoregulation
pro-inflammatory cytokines
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cell adhesion
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headline:The enzymatic poly(gallic acid) reduces pro-inflammatory cytokines in vitro, a potential application in inflammatory diseases
description:Cytokines like IL-6, TNF-α, and IL-1β are important mediators of inflammation in many inflammatory diseases, as well as in cellular processes like cell proliferation and cell adhesion. Finding new molecules that decrease cell proliferation, adhesion (inflammatory infiltrate), and pro-inflammatory cytokine release could help in the treatment of many inflammatory diseases. The naturally derived poly(gallic acid) (PGAL), produced enzymatically from gallic acid in aqueous medium, is a non-toxic, thermostable multiradical polyanion that is antioxidant and has potential biomedical uses. Experimental evidence has demonstrated that PGAL reduces pro-inflammatory cytokines, which are the target of some inflammatory diseases. PGAL decreased IL-6, TNF-α, and IL-1β production in human monocytes exposed to PMA without affecting cell viability. Additionally, PGAL reduced cell proliferation by affecting the transition from the S phase to the G2 phase of the cell cycle. Cell adhesion experiments showed that PMA-induced cell adhesion was diminished with the presence of PGAL, particularly at a concentration of 200 μg/mL. These properties of PGAL show a potential use for treating inflammatory diseases, such as psoriasis or arthritis.
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name:Instituto Nacional de Rehabilitación “Luis Guillermo Ibarra Ibarra”
address:
name:Unidad de Ingeniería de Tejidos Terapia Celular y Medicina Regenerativa, Instituto Nacional de Rehabilitación “Luis Guillermo Ibarra Ibarra”, Ciudad de México, México
type:PostalAddress
type:Organization
name:Marwin Gutiérrez
affiliation:
name:Instituto Nacional de Rehabilitación “Luis Guillermo Ibarra Ibarra”
address:
name:División de Enfermedades Musculoesqueléticas y Reumáticas, Instituto Nacional de Rehabilitación “Luis Guillermo Ibarra Ibarra”, Ciudad de México, México
type:PostalAddress
type:Organization
name:Roeb García-Arrazola
affiliation:
name:UNAM
address:
name:Departamento de Alimentos y Biotecnología, Facultad de Química, UNAM, Ciudad de México, México
type:PostalAddress
type:Organization
name:Carmen G. Hernández-Valencia
affiliation:
name:UNAM
address:
name:Departamento de Alimentos y Biotecnología, Facultad de Química, UNAM, Ciudad de México, México
type:PostalAddress
type:Organization
name:Alejandra Romero-Montero
affiliation:
name:UNAM
address:
name:Departamento de Alimentos y Biotecnología, Facultad de Química, UNAM, Ciudad de México, México
type:PostalAddress
type:Organization
name:Miguel A. Hernández-Valdepeña
affiliation:
name:UNAM
address:
name:Departamento de Alimentos y Biotecnología, Facultad de Química, UNAM, Ciudad de México, México
type:PostalAddress
type:Organization
name:Miquel Gimeno
affiliation:
name:UNAM
address:
name:Departamento de Alimentos y Biotecnología, Facultad de Química, UNAM, Ciudad de México, México
type:PostalAddress
type:Organization
email:[email protected]
name:Roberto Sánchez-Sánchez
url:http://orcid.org/0000-0003-1638-171X
affiliation:
name:Instituto Nacional de Rehabilitación “Luis Guillermo Ibarra Ibarra”
address:
name:Unidad de Ingeniería de Tejidos Terapia Celular y Medicina Regenerativa, Instituto Nacional de Rehabilitación “Luis Guillermo Ibarra Ibarra”, Ciudad de México, México
type:PostalAddress
type:Organization
email:[email protected]
PostalAddress:
name:Laboratorio de Líquido Sinovial, Instituto Nacional de Rehabilitación “Luis Guillermo Ibarra Ibarra”, Ciudad de México, México
name:Dirección de Investigación, Instituto Nacional de Cancerología, Ciudad de México, México
name:Instituto de Fisiología Celular, UNAM, Ciudad de México, México
name:Laboratorio de Biotecnología, Instituto Nacional de Rehabilitación “Luis Guillermo Ibarra Ibarra”, Ciudad de México, México
name:Laboratorio de Líquido Sinovial, Instituto Nacional de Rehabilitación “Luis Guillermo Ibarra Ibarra”, Ciudad de México, México
name:Laboratorio de Biotecnología, Instituto Nacional de Rehabilitación “Luis Guillermo Ibarra Ibarra”, Ciudad de México, México
name:Laboratorio de Biotecnología, Instituto Nacional de Rehabilitación “Luis Guillermo Ibarra Ibarra”, Ciudad de México, México
name:Laboratorio de Biotecnología, Instituto Nacional de Rehabilitación “Luis Guillermo Ibarra Ibarra”, Ciudad de México, México
name:Unidad de Ingeniería de Tejidos Terapia Celular y Medicina Regenerativa, Instituto Nacional de Rehabilitación “Luis Guillermo Ibarra Ibarra”, Ciudad de México, México
name:División de Enfermedades Musculoesqueléticas y Reumáticas, Instituto Nacional de Rehabilitación “Luis Guillermo Ibarra Ibarra”, Ciudad de México, México
name:Departamento de Alimentos y Biotecnología, Facultad de Química, UNAM, Ciudad de México, México
name:Departamento de Alimentos y Biotecnología, Facultad de Química, UNAM, Ciudad de México, México
name:Departamento de Alimentos y Biotecnología, Facultad de Química, UNAM, Ciudad de México, México
name:Departamento de Alimentos y Biotecnología, Facultad de Química, UNAM, Ciudad de México, México
name:Departamento de Alimentos y Biotecnología, Facultad de Química, UNAM, Ciudad de México, México
name:Unidad de Ingeniería de Tejidos Terapia Celular y Medicina Regenerativa, Instituto Nacional de Rehabilitación “Luis Guillermo Ibarra Ibarra”, Ciudad de México, México
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