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Topics {✒️}

peptide-based anti-pcsk9 vaccines apolipoprotein e-deficient mice yuhua liao & zhihua qiu promotes anti-inflammatory phenotype month download article/chapter low-density lipoprotein cholesterol long-term ldlc management tumor necrosis factor-α adenovirus-mediated gene delivery potential mechanisms involved pcsk9qβ-003 vaccine attenuated open cardiovasc med article cardiovascular drugs decreased total cholesterol full article pdf privacy choices/manage cookies major research plan related subjects endothelial cell dysfunction foam cell formation high density lipoproteins brown ms intestinal epithelial cells raal fj pcsk9 inhibitor therapy vaccine-based approaches therapeutic peptide vaccine cell tissue res monocyte chemoattractant protein-1 yuhua liao zhihua qiu cardiovasc drugs ther receptor-mediated pathway nanoliposomal pcsk9 vaccine macrophage cholesterol transport cholesterol efflux pathways methods mol biol phosphate-buffered saline biochim biophys acta heterozygous familial hypercholesterolaemia placebo-controlled trial curr opin lipidol common demographic parameters sterol-dependent transactivation ben djoudi ouadda tongji medical college individual participants included additional information publisher article wu conditions privacy policy

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         headline:PCSK9Qβ-003 Vaccine Attenuates Atherosclerosis in Apolipoprotein E-Deficient Mice
         description:Our group has developed a therapeutic vaccine targeting proprotein convertase subtilisin/kexin type 9 (PCSK9), named PCSK9Qβ-003. In this study, we investigated the potential effectiveness of the PCSK9Qβ-003 vaccine on atherosclerosis. Male ApoE−/− mice were randomly assigned to three groups: a phosphate-buffered saline (PBS) group, Qβ virus-like particles (VLP) group, and PCSK9Qβ-003 vaccine group. Mice in the PCSK9Qβ-003 group were injected with the PCSK9Qβ-003 vaccine four times (100 μg/time) over a period of 18 weeks. The effects of the vaccine on atherosclerotic plaque, cholesterol transport, inflammation and apoptosis were investigated. The PCSK9Qβ-003 vaccine obviously decreased total cholesterol and low-density lipoprotein cholesterol in ApoE−/− mice. Compared with the other groups, the PCSK9Qβ-003 vaccine significantly reduced the lesion area and promoted the stability of atherosclerotic plaque. The vaccine regulated cholesterol transport in the aorta of ApoE−/− mice by up-regulating the expression level of liver X receptor α and ATP binding cassette transporter A1. Additionally, macrophage infiltration and expression of monocyte chemoattractant protein-1 and tumor necrosis factor-α were significantly decreased in the mice administered the PCSK9Qβ-003 vaccine. The vaccine also markedly reduced apoptosis in the lesion area of the aorta in ApoE−/− mice. The results demonstrated that the PCSK9Qβ-003 vaccine attenuated the progression of atherosclerosis by modulating reverse cholesterol transport and inhibiting inflammation infiltration and apoptosis, which may provide a novel therapeutic approach for atherosclerosis and greatly improve treatment compliance among patients.
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            Hyperlipidemia
            Liver X receptors
            Vaccine
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            Cardiology
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      headline:PCSK9Qβ-003 Vaccine Attenuates Atherosclerosis in Apolipoprotein E-Deficient Mice
      description:Our group has developed a therapeutic vaccine targeting proprotein convertase subtilisin/kexin type 9 (PCSK9), named PCSK9Qβ-003. In this study, we investigated the potential effectiveness of the PCSK9Qβ-003 vaccine on atherosclerosis. Male ApoE−/− mice were randomly assigned to three groups: a phosphate-buffered saline (PBS) group, Qβ virus-like particles (VLP) group, and PCSK9Qβ-003 vaccine group. Mice in the PCSK9Qβ-003 group were injected with the PCSK9Qβ-003 vaccine four times (100 μg/time) over a period of 18 weeks. The effects of the vaccine on atherosclerotic plaque, cholesterol transport, inflammation and apoptosis were investigated. The PCSK9Qβ-003 vaccine obviously decreased total cholesterol and low-density lipoprotein cholesterol in ApoE−/− mice. Compared with the other groups, the PCSK9Qβ-003 vaccine significantly reduced the lesion area and promoted the stability of atherosclerotic plaque. The vaccine regulated cholesterol transport in the aorta of ApoE−/− mice by up-regulating the expression level of liver X receptor α and ATP binding cassette transporter A1. Additionally, macrophage infiltration and expression of monocyte chemoattractant protein-1 and tumor necrosis factor-α were significantly decreased in the mice administered the PCSK9Qβ-003 vaccine. The vaccine also markedly reduced apoptosis in the lesion area of the aorta in ApoE−/− mice. The results demonstrated that the PCSK9Qβ-003 vaccine attenuated the progression of atherosclerosis by modulating reverse cholesterol transport and inhibiting inflammation infiltration and apoptosis, which may provide a novel therapeutic approach for atherosclerosis and greatly improve treatment compliance among patients.
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         Atherosclerosis
         Hyperlipidemia
         Liver X receptors
         Vaccine
         PCSK9
         Cardiology
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