We are analyzing https://link.springer.com/article/10.1007/s10555-010-9223-6.

Title:
Interactions between lymphocytes and myeloid cells regulate pro- versus anti-tumor immunity | Cancer and Metastasis Reviews
Description:
Tumor-associated myeloid cells have been implicated in regulating many of the “hallmarks of cancer” and thus fostering solid tumor development and metastasis. However, the same innate leukocytes also participate in anti-tumor immunity and restraint of malignant disease. While many factors regulate the propensity of myeloid cells to promote or repress cancerous growths, polarized adaptive immune responses by B and T lymphocytes have been identified as regulators of many aspects of myeloid cell biology by specifically regulating their functional capabilities. Here, we detail the diversity of heterogeneous B and T lymphocyte populations and their impacts on solid tumor development through their abilities to regulate myeloid cell function in solid tumors.
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Keywords {🔍}

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Topics {✒️}

tumor-infiltrating foxp3-positive regulatory cell/immunoglobulin/fcγr signaling axis tumor-infiltrating foxp3-cd4+cd25+ cell-mediated antigen-specific immunotherapy regulate anti-tumor programs anti-tumor immune responses anti-tumor immune programs enhance anti-tumor responses favor pro-tumor n2 article download pdf tumor-immune surveillance programs neutralize m2-type macrophage pro-tumor myeloid cells pro-inflammatory cytokines il1 promoting cxcr-2-dependent angiogenesis regulating pro-tumor properties immune-competent mouse models t-helper-cell differentiation strong antigen-specific activation cell-dependent tumor immunity similar pro-tumor role tumor-specific th17 cells transforming growth factor il17-producing cells correlates turn activates ikk-α tumor necrosis factor lymphocyte-mediated chronic inflammation interleukin-17 promotes angiogenesis anti-tumor programs ifnγ-producing th1 cells pro-tumor pathways cd4+cd25+foxp3+ regulatory tumor-bearing state accessory-cell-dependent activation anti-tumor programming human cd4+ il-17-producing privacy choices/manage cookies anti-tumor immunity anti-tumor antibodies increased tumor size enhanced humoral responses alter adaptive immunity immune-suppressive programs [25] pro-tumor immunity myeloid suppressor cells anti-tumor activities fostered immune surveillance anti-tumor ctl multiple immunosuppressive programs prostate cancer cells

Schema {🗺️}

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         headline:Interactions between lymphocytes and myeloid cells regulate pro- versus anti-tumor immunity
         description:Tumor-associated myeloid cells have been implicated in regulating many of the “hallmarks of cancer” and thus fostering solid tumor development and metastasis. However, the same innate leukocytes also participate in anti-tumor immunity and restraint of malignant disease. While many factors regulate the propensity of myeloid cells to promote or repress cancerous growths, polarized adaptive immune responses by B and T lymphocytes have been identified as regulators of many aspects of myeloid cell biology by specifically regulating their functional capabilities. Here, we detail the diversity of heterogeneous B and T lymphocyte populations and their impacts on solid tumor development through their abilities to regulate myeloid cell function in solid tumors.
         datePublished:2010-04-21T00:00:00Z
         dateModified:2010-04-21T00:00:00Z
         pageStart:309
         pageEnd:316
         license:https://creativecommons.org/licenses/by-nc/2.0
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            Inflammation
            Lymphocyte
            Macrophage
            Metastasis
            Cancer Research
            Oncology
            Biomedicine
            general
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ScholarlyArticle:
      headline:Interactions between lymphocytes and myeloid cells regulate pro- versus anti-tumor immunity
      description:Tumor-associated myeloid cells have been implicated in regulating many of the “hallmarks of cancer” and thus fostering solid tumor development and metastasis. However, the same innate leukocytes also participate in anti-tumor immunity and restraint of malignant disease. While many factors regulate the propensity of myeloid cells to promote or repress cancerous growths, polarized adaptive immune responses by B and T lymphocytes have been identified as regulators of many aspects of myeloid cell biology by specifically regulating their functional capabilities. Here, we detail the diversity of heterogeneous B and T lymphocyte populations and their impacts on solid tumor development through their abilities to regulate myeloid cell function in solid tumors.
      datePublished:2010-04-21T00:00:00Z
      dateModified:2010-04-21T00:00:00Z
      pageStart:309
      pageEnd:316
      license:https://creativecommons.org/licenses/by-nc/2.0
      sameAs:https://doi.org/10.1007/s10555-010-9223-6
      keywords:
         Cancer
         Inflammation
         Lymphocyte
         Macrophage
         Metastasis
         Cancer Research
         Oncology
         Biomedicine
         general
      image:
         https://media.springernature.com/lw1200/springer-static/image/art%3A10.1007%2Fs10555-010-9223-6/MediaObjects/10555_2010_9223_Fig1_HTML.gif
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                  type:Organization
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            name:Pauline Andreu
            affiliation:
                  name:University of California
                  address:
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                     type:PostalAddress
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            affiliation:
                  name:University of California
                  address:
                     name:Department of Pathology, University of California, San Francisco, USA
                     type:PostalAddress
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                  name:University of California
                  address:
                     name:Helen Diller Family Comprehensive Cancer Center, University of California, San Francisco, USA
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      name:Pauline Andreu
      affiliation:
            name:University of California
            address:
               name:Department of Pathology, University of California, San Francisco, USA
               type:PostalAddress
            type:Organization
      name:Lisa M. Coussens
      affiliation:
            name:University of California
            address:
               name:Department of Pathology, University of California, San Francisco, USA
               type:PostalAddress
            type:Organization
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            address:
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      name:Department of Pathology, University of California, San Francisco, USA
      name:Department of Pathology, University of California, San Francisco, USA
      name:Helen Diller Family Comprehensive Cancer Center, University of California, San Francisco, USA

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