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cancer, google, scholar, cas, pubmed, article, colorectal, risk, antiinflammatory, drugs, nonsteroidal, cox, colon, cyclooxygenase, north, aspirin, african, study, res, carolina, health, med, university, data, americans, robert, access, expression, chemoprevention, center, usa, privacy, cookies, content, sansbury, millikan, sandler, polymorphisms, nsaid, carcinoma, prevention, human, national, chapel, hill, analysis, publish, research, search, control,

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month download article/chapter willett mj stampfer nonsteroidal anti-inflammatory drug nonsteroidal anti-inflammatory drugs steroidal anti-inflammatory drugs population-based study br mettlin nj petrelli gov/guidelines/obesity/practgde full article pdf long-term mesalamine therapy dc ainsworth colorectal cancer prevention kune lf watson population-based study post-trial folow anti-inflammatory drugs nonsteroidal antiinflammatory drugs ouellet md percival haskell wl human gastric carcinoma article cancer privacy choices/manage cookies colorectal cancer br nonsteriodal antiinflammatory drugs received grant support single nucleotide polymorphisms smalley rn dubois ferrenbach rn dubois large-bowel cancer case-control study nonaspirin nsaid users human colorectal adenomas article log colon carcinoma cells cox-2 val511ala snp national cancer institute ala cox-2 genotype regular nsaid users regular nsaid users colorectal cancer risk article cite european economic area original paper published belzile jf boivin long-term users smalley wa ray daugherty mr griffen warshauer pd stolley kawahito rl wilder radhika fm giardiello

Questions {❓}

• CC Hsieh P Maisonneuve P Boyle GJ Macfarlane C Roberston (1991) Analysis of quantitative data by quantiles in epidemiologic studies: classification according to cases, noncases, or all subjects?

Schema {🗺️}

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         description: The inducible Cyclooxygenase (COX)-2 enzyme plays an important role in inflammation and carcinogenesis. Recent reports suggest that single nucleotide polymorphisms (SNPs) in the COX-2 gene may alter enzyme function and in turn modify an individual’s risk of colon cancer. We explored the association between the COX-2 Val511Ala SNP and risk of colon cancer among 240 African American cases and 326 African American controls in a population-based, case-control study in North Carolina. We used unconditional logistic regression models to determine the odds ratios (ORs) for genotype and risk of colon cancer. We observed a non-statistically significant inverse association between any Ala COX-2 genotype and risk of colon cancer (OR = 0.62, 95% CI: 0.33, 1.16) among African Americans. The inverse association was present among non-regular NSAID users, use ≤ 3 times/week, (OR = 0.66; 95% CI: 0.32, 1.37) and regular NSAID users, use ≥3 times/week for ≥3 months, (OR = 0.41; 95% CI: 0.11, 1.54). Our results suggest that the COX-2 Val511Ala SNP does not antagonize the effect of NSAIDs on colon cancer risk and provides support that NSAID use and the COX-2 Val511Ala SNP may contribute to a reduced risk of colon cancer among African Americans.
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      headline:COX-2 Polymorphism, Use of Nonsteroidal Anti-Inflammatory Drugs, and Risk of Colon Cancer in African Americans (United States)
      description: The inducible Cyclooxygenase (COX)-2 enzyme plays an important role in inflammation and carcinogenesis. Recent reports suggest that single nucleotide polymorphisms (SNPs) in the COX-2 gene may alter enzyme function and in turn modify an individual’s risk of colon cancer. We explored the association between the COX-2 Val511Ala SNP and risk of colon cancer among 240 African American cases and 326 African American controls in a population-based, case-control study in North Carolina. We used unconditional logistic regression models to determine the odds ratios (ORs) for genotype and risk of colon cancer. We observed a non-statistically significant inverse association between any Ala COX-2 genotype and risk of colon cancer (OR = 0.62, 95% CI: 0.33, 1.16) among African Americans. The inverse association was present among non-regular NSAID users, use ≤ 3 times/week, (OR = 0.66; 95% CI: 0.32, 1.37) and regular NSAID users, use ≥3 times/week for ≥3 months, (OR = 0.41; 95% CI: 0.11, 1.54). Our results suggest that the COX-2 Val511Ala SNP does not antagonize the effect of NSAIDs on colon cancer risk and provides support that NSAID use and the COX-2 Val511Ala SNP may contribute to a reduced risk of colon cancer among African Americans.
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