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LINK . SPRINGER . COM {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Link.springer.com Make Money
  6. Keywords
  7. Topics
  8. Schema
  9. External Links
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We are analyzing https://link.springer.com/article/10.1007/s00018-016-2207-0.

Title:
Mechanisms and consequences of entosis | Cellular and Molecular Life Sciences
Description:
Multiple mechanisms have emerged where the engulfment of whole live cells, leading to the formation of what are called ‘cell-in-cell’ structures, induces cell death. Entosis is one such mechanism that drives cell-in-cell formation during carcinogenesis and development. Curiously, entotic cells participate actively in their own engulfment, by invading into their hosts, and are then killed non-cell-autonomously. Here we review the mechanisms of entosis and entotic cell death and the consequences of entosis on cell populations.
Website Age:
28 years and 1 months (reg. 1997-05-29).

Matching Content Categories {📚}

  • Telecommunications
  • Education
  • Science

Content Management System {📝}

What CMS is link.springer.com built with?

Custom-built

No common CMS systems were detected on Link.springer.com, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of link.springer.com audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
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How Does Link.springer.com Make Money? {💸}

We're unsure how the site profits.

Not all websites focus on profit; some are designed to educate, connect people, or share useful tools. People create websites for numerous reasons. And this could be one such example. Link.springer.com has a secret sauce for making money, but we can't detect it yet.

Keywords {🔍}

pubmed, article, cell, google, scholar, cas, death, central, overholtzer, wang, entosis, mol, cells, cellincell, sci, med, cancer, biol, cannibalism, usa, curr, mechanisms, formation, development, access, competition, nature, chen, sun, engulfment, entotic, huang, human, privacy, cookies, content, krishna, structures, proc, natl, acad, nat, lindsten, thompson, res, social, publish, search, life, sciences,

Topics {✒️}

e-cadherin/cd68/cd45 triple staining g-protein-coupled receptor signaling phagocytosis—plays central roles month download article/chapter cell-death process regulated full article pdf epithelial cadherin expression tumor cell progression cell-cell contact formation bim regulates apoptosis article krishna developmental photoreceptor apoptosis privacy choices/manage cookies del pozo ma mir-14 regulates autophagy human tumor samples entotic cell death programmed cell death related subjects ischemic brain injury autophagy-inducing peptides inducing cell competition +-atpase-regulated form article cellular nonapoptotic cell death entotic vacuole fission cell death subroutines cell death triggered induces cell death phagoptosis—cell death promote cell death developmental cell death mtor regulates phagosome p53-null cells primary melanoma cells dmyc transforms cells european economic area iron-dependent form targeting single membranes sporulating bacillus subtilis male-specific muscles p53-mediated activity luminal epithelial barrier winners kill losers targeting ip3-kinase 2 mailleux aa predatory slime mold par3-lgl antagonism michael overholtzer article log

Schema {🗺️}

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         headline:Mechanisms and consequences of entosis
         description:Multiple mechanisms have emerged where the engulfment of whole live cells, leading to the formation of what are called ‘cell-in-cell’ structures, induces cell death. Entosis is one such mechanism that drives cell-in-cell formation during carcinogenesis and development. Curiously, entotic cells participate actively in their own engulfment, by invading into their hosts, and are then killed non-cell-autonomously. Here we review the mechanisms of entosis and entotic cell death and the consequences of entosis on cell populations.
         datePublished:2016-04-05T00:00:00Z
         dateModified:2016-04-05T00:00:00Z
         pageStart:2379
         pageEnd:2386
         sameAs:https://doi.org/10.1007/s00018-016-2207-0
         keywords:
            Entosis
            Entotic cell death
            Cannibalism
            Cell-in-cell
            Engulfment
            Phagocytosis
            Autophagy
            LAP
            Cell competition
            Aneuploidy
            Cell Biology
            Biomedicine
            general
            Life Sciences
            Biochemistry
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            name:Cellular and Molecular Life Sciences
            issn:
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      headline:Mechanisms and consequences of entosis
      description:Multiple mechanisms have emerged where the engulfment of whole live cells, leading to the formation of what are called ‘cell-in-cell’ structures, induces cell death. Entosis is one such mechanism that drives cell-in-cell formation during carcinogenesis and development. Curiously, entotic cells participate actively in their own engulfment, by invading into their hosts, and are then killed non-cell-autonomously. Here we review the mechanisms of entosis and entotic cell death and the consequences of entosis on cell populations.
      datePublished:2016-04-05T00:00:00Z
      dateModified:2016-04-05T00:00:00Z
      pageStart:2379
      pageEnd:2386
      sameAs:https://doi.org/10.1007/s00018-016-2207-0
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         Entosis
         Entotic cell death
         Cannibalism
         Cell-in-cell
         Engulfment
         Phagocytosis
         Autophagy
         LAP
         Cell competition
         Aneuploidy
         Cell Biology
         Biomedicine
         general
         Life Sciences
         Biochemistry
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                  name:Memorial Sloan Kettering Cancer Center
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                     name:Louis V. Gerstner Jr. Graduate School of Biomedical Sciences, Memorial Sloan Kettering Cancer Center, New York, USA
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      name:Memorial Sloan Kettering Cancer Center
      address:
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               type:PostalAddress
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            name:Memorial Sloan Kettering Cancer Center
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               name:Louis V. Gerstner Jr. Graduate School of Biomedical Sciences, Memorial Sloan Kettering Cancer Center, New York, USA
               type:PostalAddress
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      name:Michael Overholtzer
      affiliation:
            name:Memorial Sloan Kettering Cancer Center
            address:
               name:Cell Biology Program, Memorial Sloan Kettering Cancer Center, New York, USA
               type:PostalAddress
            type:Organization
            name:Memorial Sloan Kettering Cancer Center
            address:
               name:Louis V. Gerstner Jr. Graduate School of Biomedical Sciences, Memorial Sloan Kettering Cancer Center, New York, USA
               type:PostalAddress
            type:Organization
      email:[email protected]
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      name:Cell Biology Program, Memorial Sloan Kettering Cancer Center, New York, USA
      name:Louis V. Gerstner Jr. Graduate School of Biomedical Sciences, Memorial Sloan Kettering Cancer Center, New York, USA
      name:Cell Biology Program, Memorial Sloan Kettering Cancer Center, New York, USA
      name:Louis V. Gerstner Jr. Graduate School of Biomedical Sciences, Memorial Sloan Kettering Cancer Center, New York, USA
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External Links {🔗}(241)

Analytics and Tracking {📊}

  • Google Tag Manager

Libraries {📚}

  • Clipboard.js
  • Prism.js

CDN Services {📦}

  • Crossref

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