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LINK . SPRINGER . COM {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Link.springer.com Make Money
  6. Keywords
  7. Topics
  8. Schema
  9. External Links
  10. Analytics And Tracking
  11. Libraries
  12. CDN Services

We are analyzing https://link.springer.com/article/10.1007/s00018-003-3228-z.

Title:
Interleukin-17: a mediator of inflammatory responses | Cellular and Molecular Life Sciences
Description:
Interleukin-17 (IL-17) is a prototype member of a new cytokine family with six species identified to date. IL-17 is secreted mainly by activated CD4+ and CD8+ T lymphocytes, while its receptor is distributed ubiquitously. IL-17 has been classified as a proinflammatory cytokine because of its ability to induce the expression of many mediators of inflammation, most strikingly those that are involved in the proliferation, maturation and chemotaxis of neutrophils. Increased levels of IL-17 have been associated with several conditions, including airway inflammation, rheumatoid arthritis, intraperitoneal abscesses and adhesions, inflammatory bowel disease, allograft rejection, psoriasis, cancer and multiple sclerosis. This review provides an overview of IL-17 activities, concentrating on those that lead to neutrophil recruitment.
Website Age:
28 years and 1 months (reg. 1997-05-29).

Matching Content Categories {📚}

  • Social Networks
  • Insurance
  • Telecommunications

Content Management System {📝}

What CMS is link.springer.com built with?

Custom-built

No common CMS systems were detected on Link.springer.com, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of link.springer.com audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,016 visitors per month in the current month.

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How Does Link.springer.com Make Money? {💸}

We can't see how the site brings in money.

Some websites aren't about earning revenue; they're built to connect communities or raise awareness. There are numerous motivations behind creating websites. This might be one of them. Link.springer.com might be plotting its profit, but the way they're doing it isn't detectable yet.

Keywords {🔍}

article, privacy, cookies, interleukin, witowski, content, information, publish, search, inflammation, data, journal, research, life, cmls, inflammatory, download, książek, jörres, cells, discover, author, optional, personal, including, parties, protection, policy, find, track, cellular, molecular, sciences, mediator, responses, review, cite, pdf, manuscript, explore, cytokine, conditions, september, related, subjects, chemokines, department, medical, received, rights,

Topics {✒️}

including airway inflammation privacy choices/manage cookies il-17-producing cells related subjects european economic area inflammatory bowel disease university medical school medical intensive care accepting optional cookies scope submit manuscript author correspondence conditions privacy policy journal finder publish data protection search search life sci article cite inflammatory responses interleukin-17 privacy policy article witowski personal data books a optional cookies manage preferences inflammation essential cookies cookies skip information journal publish usage analysis social media varying standards march 2004 volume 61 prototype member cytokine family species identified activated cd4+ distributed ubiquitously proinflammatory cytokine increased levels rheumatoid arthritis intraperitoneal abscesses allograft rejection multiple sclerosis neutrophil recruitment latest articles machine learning affiliations department universitätsklinikum charit�

Schema {🗺️}

WebPage:
      mainEntity:
         headline:Interleukin-17: a mediator of inflammatory responses
         description:Interleukin-17 (IL-17) is a prototype member of a new cytokine family with six species identified to date. IL-17 is secreted mainly by activated CD4+ and CD8+ T lymphocytes, while its receptor is distributed ubiquitously. IL-17 has been classified as a proinflammatory cytokine because of its ability to induce the expression of many mediators of inflammation, most strikingly those that are involved in the proliferation, maturation and chemotaxis of neutrophils. Increased levels of IL-17 have been associated with several conditions, including airway inflammation, rheumatoid arthritis, intraperitoneal abscesses and adhesions, inflammatory bowel disease, allograft rejection, psoriasis, cancer and multiple sclerosis. This review provides an overview of IL-17 activities, concentrating on those that lead to neutrophil recruitment.
         datePublished:
         dateModified:
         pageStart:567
         pageEnd:579
         sameAs:https://doi.org/10.1007/s00018-003-3228-z
         keywords:
            IL-17
            T cells
            inflammation
            chemokines
            granulopoiesis
            Cell Biology
            Biomedicine
            general
            Life Sciences
            Biochemistry
         image:
         isPartOf:
            name:Cellular and Molecular Life Sciences CMLS
            issn:
               1420-9071
               1420-682X
            volumeNumber:61
            type:
               Periodical
               PublicationVolume
         publisher:
            name:Birkhäuser-Verlag
            logo:
               url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
               type:ImageObject
            type:Organization
         author:
               name:J. Witowski
               affiliation:
                     name:University Medical School
                     address:
                        name:Department of Pathophysiology, University Medical School, Poznań, Poland
                        type:PostalAddress
                     type:Organization
                     name:Universitätsklinikum Charité
                     address:
                        name:Department of Nephrology and Medical Intensive Care, Universitätsklinikum Charité, Berlin, Germany
                        type:PostalAddress
                     type:Organization
               email:[email protected]
               type:Person
               name:K. Książek
               affiliation:
                     name:University Medical School
                     address:
                        name:Department of Pathophysiology, University Medical School, Poznań, Poland
                        type:PostalAddress
                     type:Organization
               type:Person
               name:A. Jörres
               affiliation:
                     name:Universitätsklinikum Charité
                     address:
                        name:Department of Nephrology and Medical Intensive Care, Universitätsklinikum Charité, Berlin, Germany
                        type:PostalAddress
                     type:Organization
               type:Person
         isAccessibleForFree:1
         type:ScholarlyArticle
      context:https://schema.org
ScholarlyArticle:
      headline:Interleukin-17: a mediator of inflammatory responses
      description:Interleukin-17 (IL-17) is a prototype member of a new cytokine family with six species identified to date. IL-17 is secreted mainly by activated CD4+ and CD8+ T lymphocytes, while its receptor is distributed ubiquitously. IL-17 has been classified as a proinflammatory cytokine because of its ability to induce the expression of many mediators of inflammation, most strikingly those that are involved in the proliferation, maturation and chemotaxis of neutrophils. Increased levels of IL-17 have been associated with several conditions, including airway inflammation, rheumatoid arthritis, intraperitoneal abscesses and adhesions, inflammatory bowel disease, allograft rejection, psoriasis, cancer and multiple sclerosis. This review provides an overview of IL-17 activities, concentrating on those that lead to neutrophil recruitment.
      datePublished:
      dateModified:
      pageStart:567
      pageEnd:579
      sameAs:https://doi.org/10.1007/s00018-003-3228-z
      keywords:
         IL-17
         T cells
         inflammation
         chemokines
         granulopoiesis
         Cell Biology
         Biomedicine
         general
         Life Sciences
         Biochemistry
      image:
      isPartOf:
         name:Cellular and Molecular Life Sciences CMLS
         issn:
            1420-9071
            1420-682X
         volumeNumber:61
         type:
            Periodical
            PublicationVolume
      publisher:
         name:Birkhäuser-Verlag
         logo:
            url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
            type:ImageObject
         type:Organization
      author:
            name:J. Witowski
            affiliation:
                  name:University Medical School
                  address:
                     name:Department of Pathophysiology, University Medical School, Poznań, Poland
                     type:PostalAddress
                  type:Organization
                  name:Universitätsklinikum Charité
                  address:
                     name:Department of Nephrology and Medical Intensive Care, Universitätsklinikum Charité, Berlin, Germany
                     type:PostalAddress
                  type:Organization
            email:[email protected]
            type:Person
            name:K. Książek
            affiliation:
                  name:University Medical School
                  address:
                     name:Department of Pathophysiology, University Medical School, Poznań, Poland
                     type:PostalAddress
                  type:Organization
            type:Person
            name:A. Jörres
            affiliation:
                  name:Universitätsklinikum Charité
                  address:
                     name:Department of Nephrology and Medical Intensive Care, Universitätsklinikum Charité, Berlin, Germany
                     type:PostalAddress
                  type:Organization
            type:Person
      isAccessibleForFree:1
["Periodical","PublicationVolume"]:
      name:Cellular and Molecular Life Sciences CMLS
      issn:
         1420-9071
         1420-682X
      volumeNumber:61
Organization:
      name:Birkhäuser-Verlag
      logo:
         url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
         type:ImageObject
      name:University Medical School
      address:
         name:Department of Pathophysiology, University Medical School, Poznań, Poland
         type:PostalAddress
      name:Universitätsklinikum Charité
      address:
         name:Department of Nephrology and Medical Intensive Care, Universitätsklinikum Charité, Berlin, Germany
         type:PostalAddress
      name:University Medical School
      address:
         name:Department of Pathophysiology, University Medical School, Poznań, Poland
         type:PostalAddress
      name:Universitätsklinikum Charité
      address:
         name:Department of Nephrology and Medical Intensive Care, Universitätsklinikum Charité, Berlin, Germany
         type:PostalAddress
ImageObject:
      url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
Person:
      name:J. Witowski
      affiliation:
            name:University Medical School
            address:
               name:Department of Pathophysiology, University Medical School, Poznań, Poland
               type:PostalAddress
            type:Organization
            name:Universitätsklinikum Charité
            address:
               name:Department of Nephrology and Medical Intensive Care, Universitätsklinikum Charité, Berlin, Germany
               type:PostalAddress
            type:Organization
      email:[email protected]
      name:K. Książek
      affiliation:
            name:University Medical School
            address:
               name:Department of Pathophysiology, University Medical School, Poznań, Poland
               type:PostalAddress
            type:Organization
      name:A. Jörres
      affiliation:
            name:Universitätsklinikum Charité
            address:
               name:Department of Nephrology and Medical Intensive Care, Universitätsklinikum Charité, Berlin, Germany
               type:PostalAddress
            type:Organization
PostalAddress:
      name:Department of Pathophysiology, University Medical School, Poznań, Poland
      name:Department of Nephrology and Medical Intensive Care, Universitätsklinikum Charité, Berlin, Germany
      name:Department of Pathophysiology, University Medical School, Poznań, Poland
      name:Department of Nephrology and Medical Intensive Care, Universitätsklinikum Charité, Berlin, Germany

External Links {🔗}(29)

Analytics and Tracking {📊}

  • Google Tag Manager

Libraries {📚}

  • Clipboard.js
  • Prism.js

CDN Services {📦}

  • Crossref

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