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LINK . SPRINGER . COM {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Link.springer.com Make Money
  6. Keywords
  7. Topics
  8. Schema
  9. External Links
  10. Analytics And Tracking
  11. Libraries
  12. CDN Services

We are analyzing https://link.springer.com/article/10.1007/bf00987321.

Title:
Invasion promoter versus invasion suppressor molecules: the paradigm of E-cadherin | Molecular Biology Reports
Description:
Metastasis determines cancer malignancy. Neoplastic cells metastasize through a multistep process of invasion. At each step, these cells create a dynamic micro-ecosystem in which the elements of the host are considered to paricipate actively invasion. Within such micro-ecosystems, invasion is believed to be governed by a balance between the activation of promoter (i-minus) and suppressor (i-plus) genes. Products of such genes regulate the expression of the invasive (I-plus) and the nonivasive (I-minus) phenotypes. Experimentsin vitro andin vivo, as well as observations on human cancers have put forward the cell-cell adhesion molecule E-cadherin (L-CAM; uvomorulin) as a regulator of epithelial organization and an invasion suppressor.
Website Age:
28 years and 1 months (reg. 1997-05-29).

Matching Content Categories {πŸ“š}

  • Education
  • Science
  • Telecommunications

Content Management System {πŸ“}

What CMS is link.springer.com built with?

Custom-built

No common CMS systems were detected on Link.springer.com, and no known web development framework was identified.

Traffic Estimate {πŸ“ˆ}

What is the average monthly size of link.springer.com audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
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How Does Link.springer.com Make Money? {πŸ’Έ}

We can't tell how the site generates income.

Earning money isn't the goal of every website; some are designed to offer support or promote social causes. People have different reasons for creating websites. This might be one such reason. Link.springer.com might have a hidden revenue stream, but it's not something we can detect.

Keywords {πŸ”}

google, scholar, pubmed, cell, cancer, van, biol, mareel, res, roy, takeichi, metastasis, invasion, article, bracke, vleminckx, vakaet, kemler, pathol, biology, access, behrens, birchmeier, nature, usa, development, press, rev, int, hirano, sci, privacy, cookies, content, research, molecular, gao, gumbiner, proc, natl, acad, hirohashi, publish, search, suppressor, ecadherin, marc, cells, open, eds,

Topics {βœ’οΈ}

e-cadherin special issue month download article/chapter acta oto-rhino-laryngol molecular cell biology invasion suppressor l-cam paricipate actively invasion e-cadherin related subjects full article pdf molecular pathology published privacy choices/manage cookies epithelial organization extracellular vesicles mareel mm article mareel van putten wlj van moorselaar rja cancer metastasis rev european economic area cell biology 2 cell biology 3 van larebeke na de bruyne gk conditions privacy policy dynamic micro-ecosystem nelson wj basic research developmental biology neoplastic cells metastasize nasim mm accepting optional cookies article log suppressor kao l check access instant access molecular genetics journal finder publish promoter uvomorulin carter bs article cite van roy van roy invasion dunn rs thompson ad thiery jp trends cell biol

Schema {πŸ—ΊοΈ}

WebPage:
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         headline:Invasion promoter versus invasion suppressor molecules: the paradigm of E-cadherin
         description:Metastasis determines cancer malignancy. Neoplastic cells metastasize through a multistep process of invasion. At each step, these cells create a dynamic micro-ecosystem in which the elements of the host are considered to paricipate actively invasion. Within such micro-ecosystems, invasion is believed to be governed by a balance between the activation of promoter (i-minus) and suppressor (i-plus) genes. Products of such genes regulate the expression of the invasive (I-plus) and the nonivasive (I-minus) phenotypes. Experimentsin vitro andin vivo, as well as observations on human cancers have put forward the cell-cell adhesion molecule E-cadherin (L-CAM; uvomorulin) as a regulator of epithelial organization and an invasion suppressor.
         datePublished:
         dateModified:
         pageStart:45
         pageEnd:67
         sameAs:https://doi.org/10.1007/BF00987321
         keywords:
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            metastasis
            cell adhesion
            E-cadherin
            L-CAM
            uvomorulin
            IGF-I
            Animal Biochemistry
            Animal Anatomy / Morphology / Histology
         image:
         isPartOf:
            name:Molecular Biology Reports
            issn:
               1573-4978
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            volumeNumber:19
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         author:
               name:Marc Mareel
               affiliation:
                     name:University Hospital
                     address:
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      headline:Invasion promoter versus invasion suppressor molecules: the paradigm of E-cadherin
      description:Metastasis determines cancer malignancy. Neoplastic cells metastasize through a multistep process of invasion. At each step, these cells create a dynamic micro-ecosystem in which the elements of the host are considered to paricipate actively invasion. Within such micro-ecosystems, invasion is believed to be governed by a balance between the activation of promoter (i-minus) and suppressor (i-plus) genes. Products of such genes regulate the expression of the invasive (I-plus) and the nonivasive (I-minus) phenotypes. Experimentsin vitro andin vivo, as well as observations on human cancers have put forward the cell-cell adhesion molecule E-cadherin (L-CAM; uvomorulin) as a regulator of epithelial organization and an invasion suppressor.
      datePublished:
      dateModified:
      pageStart:45
      pageEnd:67
      sameAs:https://doi.org/10.1007/BF00987321
      keywords:
         invasion
         metastasis
         cell adhesion
         E-cadherin
         L-CAM
         uvomorulin
         IGF-I
         Animal Biochemistry
         Animal Anatomy / Morphology / Histology
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                     type:PostalAddress
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                  name:University of Ghent, K.L.
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            name:University of Ghent, K.L.
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               name:Laboratory of Molecular Cell Biology, Department of Molecular Genetics, University of Ghent, K.L., Ghent, Belgium
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      name:Department of Radiotherapy, Nuclear Medicine and Experimental Cancerology, University Hospital, Ghent, Belgium
      name:Department of Radiotherapy, Nuclear Medicine and Experimental Cancerology, University Hospital, Ghent, Belgium
      name:Laboratory of Molecular Cell Biology, Department of Molecular Genetics, University of Ghent, K.L., Ghent, Belgium
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External Links {πŸ”—}(262)

Analytics and Tracking {πŸ“Š}

  • Google Tag Manager

Libraries {πŸ“š}

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CDN Services {πŸ“¦}

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