We are analyzing https://link.springer.com/article/10.1007/bf00155046.

Title:
Tributary vein obstruction pathogenesis and treatment of sequelae | Documenta Ophthalmologica
Description:
An acute branch retinal vein obstruction or occlusion is followed by a series of haemodynamic events designed to overcome the obstruction and restore equilibrium within the retinal vasculature. Experimental studies would indicate that the immediate post-obstructive retinal oedema is probably the result of capillary and venular distension due to the sudden increase in intraluminar hydrostatic pressure. The widespread extravasation of blood which occurs some time later is probably the result of degenerative changes in the capillary walls secondary to prolonged stasis and hypoxia. In many tributary vein obstructions, readjustments within the involved retinal microvasculature are effective in overcoming the effects of the obstruction. The involved retinal capillaries gradually return to normal or nearly normal, with preservation of their physiological functions. Good vision is often maintained and no therapy is required. In other cases, often where associated arterial insufficiency exists, significant venous stasis persists and the retinal capillaries remain incompetent with the formation of intraretinal oedema. Evidence available at this time would indicate that selective photocoagulation is of some benefit in preventing the chronic accumulation of macular fluid in certain cases and avoiding the unwanted sequelae of cystic macular degenerative changes and pigmentation abnormalities at the fovea. Photocoagulation appears to be less justified in cases of long-established macular oedema with degenerative changes already in evidence. In certain varieties of tributary vein occlusion characterized by marked arterial insufficiency, the involved capillary bed fails to perfuse fully. Neovascularization is frequently associated with extensive areas of non-perfused retina. Neovascularization, although it may appear 3 – 6 months following the acute obstructive episode, rarely demands treatment within the year. The neovascularization associated with a branch vein occlusion is generally subtle, slow-growing, and responds well to photocoagulation. Papillary neovascularization indicates widespread ischaemia, and often suggests implication of further quadrants of the retina in the ischaemic process. Papillary neovascularization can be indirectly influenced by extensive photocoagulation of the ischaemic retina resulting from the vein occlusion.
Website Age:
28 years and 1 months (reg. 1997-05-29).

Matching Content Categories {📚}

Education
Health & Fitness
Science

Content Management System {📝}

What CMS is link.springer.com built with?

Custom-built

No common CMS systems were detected on Link.springer.com, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of link.springer.com audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month

Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

check SE Ranking
check Ahrefs
check Similarweb
check Ubersuggest
check Semrush

How Does Link.springer.com Make Money? {💸}

We don't see any clear sign of profit-making.

Many websites are intended to earn money, but some serve to share ideas or build connections. Websites exist for all kinds of purposes. This might be one of them. Link.springer.com might be earning cash quietly, but we haven't detected the monetization method.

Keywords {🔍}

google, scholar, retinal, ophthal, vein, occlusion, branch, obstruction, photocoagulation, retina, article, venous, archer, macular, neovascularization, trans, disease, arch, tributary, treatment, soc, vascular, privacy, cookies, content, studies, oedema, ocular, thrombosis, acta, information, publish, search, experimental, therapy, arterial, insufficiency, access, chapter, ophthalmology, wise, klin, mbl, data, log, journal, research, ophthalmologica, pathogenesis, sequelae,

Topics {✒️}

long-established macular oedema month download article/chapter post-obstructive retinal oedema retinal circulatory disorders arterial insufficiency exists marked arterial insufficiency retinal vein obstruction retinal vein occlusion tributary vein obstructions privacy choices/manage cookies retinal vascular occlusion retinal vasculature involved retinal microvasculature branch vein occlusion central retinal vein retinal vein thrombosis retinal venous occlusion acute obstructive episode full article pdf branch vein obstructions retinal vessel obstruction retinal vascular disease induced ocular hypertension ocular hypertension induced factors influencing retinal arterial insufficiency exaggerated macular response macular dysfunction secondary retinal venous occlusions occlusive vascular disease branch venous thrombosis retinal blood vessels european economic area haemodynamic events designed venular distension due intraluminar hydrostatic pressure swept source oct low molecular weight scieral suction cup intraretinal oedema cystic macular degenerative fluorescein fundus angiography capillary walls secondary ischaemic retina resulting accepting optional cookies related subjects den netzhautvenen check access instant access conditions privacy policy

Schema {🗺️}

WebPage:
      mainEntity:
         headline:Tributary vein obstruction pathogenesis and treatment of sequelae
         description:An acute branch retinal vein obstruction or occlusion is followed by a series of haemodynamic events designed to overcome the obstruction and restore equilibrium within the retinal vasculature. Experimental studies would indicate that the immediate post-obstructive retinal oedema is probably the result of capillary and venular distension due to the sudden increase in intraluminar hydrostatic pressure. The widespread extravasation of blood which occurs some time later is probably the result of degenerative changes in the capillary walls secondary to prolonged stasis and hypoxia. In many tributary vein obstructions, readjustments within the involved retinal microvasculature are effective in overcoming the effects of the obstruction. The involved retinal capillaries gradually return to normal or nearly normal, with preservation of their physiological functions. Good vision is often maintained and no therapy is required. In other cases, often where associated arterial insufficiency exists, significant venous stasis persists and the retinal capillaries remain incompetent with the formation of intraretinal oedema. Evidence available at this time would indicate that selective photocoagulation is of some benefit in preventing the chronic accumulation of macular fluid in certain cases and avoiding the unwanted sequelae of cystic macular degenerative changes and pigmentation abnormalities at the fovea. Photocoagulation appears to be less justified in cases of long-established macular oedema with degenerative changes already in evidence. In certain varieties of tributary vein occlusion characterized by marked arterial insufficiency, the involved capillary bed fails to perfuse fully. Neovascularization is frequently associated with extensive areas of non-perfused retina. Neovascularization, although it may appear 3 – 6 months following the acute obstructive episode, rarely demands treatment within the year. The neovascularization associated with a branch vein occlusion is generally subtle, slow-growing, and responds well to photocoagulation. Papillary neovascularization indicates widespread ischaemia, and often suggests implication of further quadrants of the retina in the ischaemic process. Papillary neovascularization can be indirectly influenced by extensive photocoagulation of the ischaemic retina resulting from the vein occlusion.
         datePublished:
         dateModified:
         pageStart:339
         pageEnd:360
         sameAs:https://doi.org/10.1007/BF00155046
         keywords:
            Retina
            Macular Oedema
            Retinal Vasculature
            Retinal Oedema
            Arterial Insufficiency
            Ophthalmology
         image:
         isPartOf:
            name:Documenta Ophthalmologica
            issn:
               1573-2622
               0012-4486
            volumeNumber:40
            type:
               Periodical
               PublicationVolume
         publisher:
            name:Kluwer Academic Publishers
            logo:
               url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
               type:ImageObject
            type:Organization
         author:
               name:Desmond B. Archer
               affiliation:
                     name:
                     address:
                        name:Belfast
                        type:PostalAddress
                     type:Organization
               type:Person
         isAccessibleForFree:
         hasPart:
            isAccessibleForFree:
            cssSelector:.main-content
            type:WebPageElement
         type:ScholarlyArticle
      context:https://schema.org
ScholarlyArticle:
      headline:Tributary vein obstruction pathogenesis and treatment of sequelae
      description:An acute branch retinal vein obstruction or occlusion is followed by a series of haemodynamic events designed to overcome the obstruction and restore equilibrium within the retinal vasculature. Experimental studies would indicate that the immediate post-obstructive retinal oedema is probably the result of capillary and venular distension due to the sudden increase in intraluminar hydrostatic pressure. The widespread extravasation of blood which occurs some time later is probably the result of degenerative changes in the capillary walls secondary to prolonged stasis and hypoxia. In many tributary vein obstructions, readjustments within the involved retinal microvasculature are effective in overcoming the effects of the obstruction. The involved retinal capillaries gradually return to normal or nearly normal, with preservation of their physiological functions. Good vision is often maintained and no therapy is required. In other cases, often where associated arterial insufficiency exists, significant venous stasis persists and the retinal capillaries remain incompetent with the formation of intraretinal oedema. Evidence available at this time would indicate that selective photocoagulation is of some benefit in preventing the chronic accumulation of macular fluid in certain cases and avoiding the unwanted sequelae of cystic macular degenerative changes and pigmentation abnormalities at the fovea. Photocoagulation appears to be less justified in cases of long-established macular oedema with degenerative changes already in evidence. In certain varieties of tributary vein occlusion characterized by marked arterial insufficiency, the involved capillary bed fails to perfuse fully. Neovascularization is frequently associated with extensive areas of non-perfused retina. Neovascularization, although it may appear 3 – 6 months following the acute obstructive episode, rarely demands treatment within the year. The neovascularization associated with a branch vein occlusion is generally subtle, slow-growing, and responds well to photocoagulation. Papillary neovascularization indicates widespread ischaemia, and often suggests implication of further quadrants of the retina in the ischaemic process. Papillary neovascularization can be indirectly influenced by extensive photocoagulation of the ischaemic retina resulting from the vein occlusion.
      datePublished:
      dateModified:
      pageStart:339
      pageEnd:360
      sameAs:https://doi.org/10.1007/BF00155046
      keywords:
         Retina
         Macular Oedema
         Retinal Vasculature
         Retinal Oedema
         Arterial Insufficiency
         Ophthalmology
      image:
      isPartOf:
         name:Documenta Ophthalmologica
         issn:
            1573-2622
            0012-4486
         volumeNumber:40
         type:
            Periodical
            PublicationVolume
      publisher:
         name:Kluwer Academic Publishers
         logo:
            url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
            type:ImageObject
         type:Organization
      author:
            name:Desmond B. Archer
            affiliation:
                  name:
                  address:
                     name:Belfast
                     type:PostalAddress
                  type:Organization
            type:Person
      isAccessibleForFree:
      hasPart:
         isAccessibleForFree:
         cssSelector:.main-content
         type:WebPageElement
["Periodical","PublicationVolume"]:
      name:Documenta Ophthalmologica
      issn:
         1573-2622
         0012-4486
      volumeNumber:40
Organization:
      name:Kluwer Academic Publishers
      logo:
         url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
         type:ImageObject
      name:
      address:
         name:Belfast
         type:PostalAddress
ImageObject:
      url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
Person:
      name:Desmond B. Archer
      affiliation:
            name:
            address:
               name:Belfast
               type:PostalAddress
            type:Organization
PostalAddress:
      name:Belfast
WebPageElement:
      isAccessibleForFree:
      cssSelector:.main-content

External Links {🔗}(71)

Analytics and Tracking {📊}

Google Tag Manager

Libraries {📚}

Clipboard.js
Prism.js

CDN Services {📦}

Crossref

4.49s.

LINK . SPRINGER . COM {}