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Structural Biology of the Membrane Attack Complex | SpringerLink
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The complement system is an intricate network of serum proteins that mediates humoral innate immunity through an amplification cascade that ultimately leads to recruitment of inflammatory cells or opsonisation or killing of pathogens. One effector arm of this network...
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Questions {โ}
- Bamford DH, Grimes JM, Stuart DI (2005) What does structure tell us about virus evolution?
- Markiewski MM, Nilsson B, Ekdahl KN, Mollnes TE, Lambris JD (2007) Complement and coagulation: strangers or partners in crime?
- Nabholz M, Tschopp J (1989) CTL-mediated cytolysis: perforin and alternative pathways?
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ScholarlyArticle:
headline:Structural Biology of the Membrane Attack Complex
pageEnd:116
pageStart:83
image:https://media.springernature.com/w153/springer-static/cover/book/978-94-017-8881-6.jpg
genre:
Biomedical and Life Sciences
Biomedical and Life Sciences (R0)
isPartOf:
name:MACPF/CDC Proteins - Agents of Defence, Attack and Invasion
isbn:
978-94-017-8881-6
978-94-017-8880-9
type:Book
publisher:
name:Springer Netherlands
logo:
url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
type:ImageObject
type:Organization
author:
name:Andreas F.-P. Sonnen
affiliation:
name:University Medical Center Freiburg
address:
name:Center for Chronic Immunodeficiency, University Medical Center Freiburg, Freiburg, Germany
type:PostalAddress
type:Organization
email:[email protected]
type:Person
name:Philipp Henneke
affiliation:
name:University Medical Center Freiburg
address:
name:Center for Chronic Immunodeficiency, University Medical Center Freiburg, Freiburg, Germany
type:PostalAddress
type:Organization
type:Person
keywords:Complememt system, Lipid membrane pore, Membrane attack complex, Pore-forming proteins, Structural biology
description:The complement system is an intricate network of serum proteins that mediates humoral innate immunity through an amplification cascade that ultimately leads to recruitment of inflammatory cells or opsonisation or killing of pathogens. One effector arm of this network is the terminal pathway of complement, which leads to the formation of the membrane attack complex (MAC) composed of complement components C5b, C6, C7, C8 and C9. Upon formation of C5
convertases via the classical or alternative pathways of complement activation, C5b is generated from C5
by proteolytic cleavage, nucleating a series of association and polymerisation reactions of the MAC-constituting complement components that culminate in pore formation of pathogenic membranes. Recent structures of MAC components and homologous proteins significantly increased our understanding of oligomerisation, membrane association and integration, shedding light onto the molecular mechanism of this important branch of the innate immune system.
datePublished:2014
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isbn:
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978-94-017-8880-9
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name:Springer Netherlands
logo:
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name:University Medical Center Freiburg
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affiliation:
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name:Center for Chronic Immunodeficiency, University Medical Center Freiburg, Freiburg, Germany
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name:University Medical Center Freiburg
address:
name:Center for Chronic Immunodeficiency, University Medical Center Freiburg, Freiburg, Germany
type:PostalAddress
type:Organization
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