We are analyzing https://link.springer.com/chapter/10.1007/978-3-030-84965-8_7.

Title:
Redox Signalling, Autophagy and Ageing | SpringerLink
Description:
Reactive oxygen species (ROS) transform cell responses through miscellaneous processes. These act as signalling molecules when present in low concentrations or damage cell machinery when present in high levels. ROS-mediated mitochondrial damage leads to lesser...
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28 years and 1 months (reg. 1997-05-29).

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Keywords {🔍}

pubmed, google, scholar, article, cas, aging, autophagy, central, cell, ageing, redox, biol, ros, mitochondrial, signalling, stress, httpsdoiorg, oxidative, med, oxygen, role, signaling, rev, chapter, longevity, reactive, species, mol, diseases, cellular, sci, httpsdoiorgs, nat, signal, free, wang, mechanisms, res, cuervo, healthy, homeostasis, biochemistry, human, agerelated, exp, zhang, antioxidants, mitochondria, privacy, cookies,

Topics {✒️}

month download article/chapter long-lived mouse models redox-sensitive signalling cascades cysteine-mediated redox signaling ageing fatma hussain brand md stress-triggered redox signalling de la fuente lapierre lr ros-dependent signal transduction age-related autophagy alterations aging-related cardiovascular diseases device instant download privacy choices/manage cookies age-related human diseases pham-huy jones dp ros-mediated cellular signaling mitochondrial transition pore transform cell responses reactive oxygen species aging-related neurodegenerative diseases anomalous mitochondrial function longest lived animal senescence article 17 damage cell machinery free radical theory european economic area numerous pathological conditions de almeida dsg vitro comparative evaluation resveratrol modulator capacity autophagosome-lysosome fusion iron-sulfur clusters averill-bates da superior proteome stability mitochondrial redox signaling enhanced cell death age-related decline mitochondrial respiratory chain fernández-ayala dj chapter redox signaling ubiquitin-proteasome system cell proliferation restriction stem cell protection conditions privacy policy rubinsztein dc lesser oxidative phosphorylation age-related diseases endo-lysosomal systems

Questions {❓}

Foyer CH, Noctor G (2016) Stress-triggered redox signalling: what’s in pROSpect?
Luo J, Mills K, le Cessie S, Noordam R, van Heemst D (2020a) Ageing, age-related diseases and oxidative stress: what to do next?
Madeo F, Tavernarakis N, Kroemer G (2010) Can autophagy promote longevity?
Massey AC, Kiffin R, Cuervo AM (2006) Autophagic defects in aging: looking for an “emergency exit”?
Panel M, Ghaleh B, Morin D (2018) Mitochondria and aging: a role for the mitochondrial transition pore?

Schema {🗺️}

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      headline:Redox Signalling, Autophagy and Ageing
      pageEnd:145
      pageStart:117
      image:https://media.springernature.com/w153/springer-static/cover/book/978-3-030-84965-8.jpg
      genre:
         Biomedical and Life Sciences
         Biomedical and Life Sciences (R0)
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      author:
            name:Fatma Hussain
            affiliation:
                  name:University of Agriculture
                  address:
                     name:Clinico-Medical Biochemistry Laboratory, Department of Biochemistry, Faculty of Sciences, University of Agriculture, Faisalabad, Pakistan
                     type:PostalAddress
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            type:Person
            name:Umm-E-Ammara Warraich
            affiliation:
                  name:University of Agriculture
                  address:
                     name:Clinico-Medical Biochemistry Laboratory, Department of Biochemistry, Faculty of Sciences, University of Agriculture, Faisalabad, Pakistan
                     type:PostalAddress
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            name:Amer Jamil
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                     name:Molecular Biochemistry Laboratory, Department of Biochemistry, Faculty of Sciences, University of Agriculture, Faisalabad, Pakistan
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      keywords:Reactive oxygen species, Autophagy, Redox signalling, Ageing
      description:Reactive oxygen species (ROS) transform cell responses through miscellaneous processes. These act as signalling molecules when present in low concentrations or damage cell machinery when present in high levels. ROS-mediated mitochondrial damage leads to lesser oxidative phosphorylation and enhanced cell death. Aberrant cell death and redox signalling are implicated in numerous pathological conditions such as cardiac, neurodegenerative, and metabolic diseases. Cells have adopted an auto-degradation process as a cytoprotective strategy by inhibition of aberrant cell death and oxidation stress along with activation of autophagy. Autophagy, a pervasive degradation or turnover homeostatic mechanism of cell organelles and components, involves differentiation and ageing. Any dysfunction of autophagy can lead to anomalous mitochondrial function and ROS. Redox signalling cascade is related to the etiology of ageing, and the cellular machinery that regulates redox, autophagy and ageing has been elucidated. Redox signalling orchestrates autophagy and ageing processes leading to altered transduction of redox homeostasis. This chapter discusses ageing process, generation of ROS, redox signalling, autophagy types and mechanisms, the interplay between redox signalling, autophagy and ageing.
      datePublished:2022
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      affiliation:
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            address:
               name:Clinico-Medical Biochemistry Laboratory, Department of Biochemistry, Faculty of Sciences, University of Agriculture, Faisalabad, Pakistan
               type:PostalAddress
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      name:Amer Jamil
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