Here's how LINK.SPRINGER.COM makes money* and how much!

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LINK . SPRINGER . COM {}

Detected CMS Systems:

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Link.springer.com Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Schema
  10. External Links
  11. Analytics And Tracking
  12. Libraries

We are analyzing https://link.springer.com/chapter/10.1007/978-1-4939-2895-8_14.

Title:
Glucocorticoids and Cancer | SpringerLink
Description:
Unlike other steroid hormone receptors, the glucocorticoid receptor (GR) is not considered an oncogene. In breast cancer, the estrogen receptor (ER) drives cell growth, proliferation, and metastasis, and the androgen receptor (AR) plays a similar role in prostate...
Website Age:
28 years and 1 months (reg. 1997-05-29).

Matching Content Categories {📚}

  • Education
  • Health & Fitness
  • Science

Content Management System {📝}

What CMS is link.springer.com built with?


Link.springer.com utilizes PLONE.

Traffic Estimate {📈}

What is the average monthly size of link.springer.com audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
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How Does Link.springer.com Make Money? {💸}

We don’t know how the website earns money.

Websites don't always need to be profitable; some serve as platforms for education or personal expression. Websites can serve multiple purposes. And this might be one of them. Link.springer.com might have a hidden revenue stream, but it's not something we can detect.

Keywords {🔍}

pubmed, google, scholar, article, cas, cancer, leukemia, glucocorticoid, lymphoblastic, acute, receptor, central, oncol, clin, treatment, blood, cell, glucocorticoids, chapter, patients, resistance, doijco, med, cells, glucocorticoidinduced, phase, prostate, apoptosis, doiblood, study, signaling, res, childhood, breast, chemotherapy, dexamethasone, cisplatin, privacy, cookies, content, information, publish, research, medicine, hormone, role, lymphoid, mechanisms, effect, endocrinology,

Topics {✒️}

month download article/chapter b-cell receptor signaling pi3-kinase/akt pathways small-cell lung cancer small-cell lung cancer glucocorticoid-induced leucine zipper cancer anorexia-cachexia syndrome advanced aids-related kaposi castration-resistant prostate cancer glucocorticoid-induced leukemia apoptosis ligand-induced nuclear translocation glucocorticoid-induced cell death leukaemia stem-cell development prostate cancer progression acute lymphoblastic leukemia kendall ec acute lymphoblastic leukaemia chronic lymphocytic leukemia privacy choices/manage cookies previously untreated advanced device instant download gene-expression patterns glucocorticoid-induced apoptosis glucocorticoid‐induced apoptosis glucocorticoid receptor signaling editor information editors early gene expression nuclear receptor signaling lymphoid leukemia cells mol cell biol ligand determine sensitivity rajkumar sv tumor suppressor activity hox gene promoters cell death differ cell death dis annu rev biochem transitional cell cancer steroid hormone receptors pui c reduced glucose metabolism overcome glucocorticoid resistance breast cancer ligand-independent phosphorylation prednisolone-responsive genes curr probl cancer pituitary adrenotropic hormone advanced prostate cancer phase iii study european economic area

Questions {❓}

  • Glucocorticoids and prostate cancer treatment: friend or foe?
  • Selective glucocorticoid receptor modulators: future of glucocorticoid immunosuppressive therapy?

Schema {🗺️}

ScholarlyArticle:
      headline:Glucocorticoids and Cancer
      pageEnd:333
      pageStart:315
      image:https://media.springernature.com/w153/springer-static/cover/book/978-1-4939-2895-8.jpg
      genre:
         Biomedical and Life Sciences
         Biomedical and Life Sciences (R0)
      isPartOf:
         name:Glucocorticoid Signaling
         isbn:
            978-1-4939-2895-8
            978-1-4939-2894-1
         type:Book
      publisher:
         name:Springer New York
         logo:
            url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
            type:ImageObject
         type:Organization
      author:
            name:Miles A. Pufall
            affiliation:
                  name:Carver College of Medicine, Holden Comprehensive Cancer Center
                  address:
                     name:Department of Biochemistry, Carver College of Medicine, Holden Comprehensive Cancer Center, Iowa City, USA
                     type:PostalAddress
                  type:Organization
            email:[email protected]
            type:Person
      keywords:Leukemia, Apoptosis, Lymphoma, Chemotherapy, Palliative
      description:Unlike other steroid hormone receptors, the glucocorticoid receptor (GR) is not considered an oncogene. In breast cancer, the estrogen receptor (ER) drives cell growth, proliferation, and metastasis, and the androgen receptor (AR) plays a similar role in prostate cancer. Accordingly, treatment of these diseases has focused on blocking steroid hormone receptor function. In contrast, glucocorticoids (GCs) work through GR to arrest growth and induce apoptosis in lymphoid tissue. Glucocorticoids are amazingly effective in this role, and have been deployed as the cornerstone of lymphoid cancer treatment for decades. Unfortunately, not all patients respond to GCs and dosage is restricted by immediate and long term side effects. In this chapter we review the treatment protocols that employ glucocorticoids as a curative agent, elaborate on what is known about their mechanism of action in these cancers, and also summarize the palliative uses of glucocorticoids for other cancers.
      datePublished:2015
      isAccessibleForFree:
      hasPart:
         isAccessibleForFree:
         cssSelector:.main-content
         type:WebPageElement
      context:https://schema.org
Book:
      name:Glucocorticoid Signaling
      isbn:
         978-1-4939-2895-8
         978-1-4939-2894-1
Organization:
      name:Springer New York
      logo:
         url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
         type:ImageObject
      name:Carver College of Medicine, Holden Comprehensive Cancer Center
      address:
         name:Department of Biochemistry, Carver College of Medicine, Holden Comprehensive Cancer Center, Iowa City, USA
         type:PostalAddress
ImageObject:
      url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
Person:
      name:Miles A. Pufall
      affiliation:
            name:Carver College of Medicine, Holden Comprehensive Cancer Center
            address:
               name:Department of Biochemistry, Carver College of Medicine, Holden Comprehensive Cancer Center, Iowa City, USA
               type:PostalAddress
            type:Organization
      email:[email protected]
PostalAddress:
      name:Department of Biochemistry, Carver College of Medicine, Holden Comprehensive Cancer Center, Iowa City, USA
WebPageElement:
      isAccessibleForFree:
      cssSelector:.main-content

External Links {🔗}(362)

Analytics and Tracking {📊}

  • Google Tag Manager

Libraries {📚}

  • Clipboard.js

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