LINK . SPRINGER . COM {}

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Apache Lenya (1 occurrences)

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We are analyzing https://link.springer.com/chapter/10.1007/978-1-4419-6612-4_24.

Title:
Lymphotoxin’s Link to Carcinogenesis: Friend or Foe? From Lymphoid Neogenesis to Hepatocellular Carcinoma and Prostate Cancer | SpringerLink
Description:
Tumor necrosis factor alpha (TNFα) was initially discovered due to its tumor necrotizing activity in mice, leading to the death of vascular endothelial cells. Today, pro- and anti-tumorigenic effects of TNF are discussed and TNF as well as other members of the...
Website Age:
28 years and 1 months (reg. 1997-05-29).

Matching Content Categories {📚}

Education
Health & Fitness
Science

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What CMS is link.springer.com built with?

Link.springer.com employs APACHE LENYA.

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What is the average monthly size of link.springer.com audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month

Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
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How Does Link.springer.com Make Money? {💸}

We can't figure out the monetization strategy.

Websites don't always need to be profitable; some serve as platforms for education or personal expression. Websites can serve multiple purposes. And this might be one of them. Link.springer.com might be earning cash quietly, but we haven't detected the monetization method.

Keywords {🔍}

google, scholar, pubmed, cas, article, lymphotoxin, cancer, immunol, lymphoid, cell, mice, receptor, tumor, tnf, necrosis, factor, expression, nature, med, ruddle, development, browning, inflammation, cells, beta, lymphotoxinbeta, signaling, research, neogenesis, role, liver, exp, alpha, immune, biol, hepatitis, pathway, science, carcinoma, heikenwalder, chapter, wang, transgenic, lymphocyte, karin, privacy, cookies, function, content, information,

Topics {✒️}

cell-mediated ifn-gamma response lymphotoxin-alpha double-deficient mice t-cell-mediated liver injury ikk/nf-kappab activation pathway lymphotoxin-beta receptor signaling lymphotoxin-alpha-expressing th1 regulate aire-dependent transcripts month download article/chapter cytokine-driven compensatory proliferation lymphotoxin-beta receptor expression murine collagen-induced arthritis tuberculin-sensitive lymphoid cells blocking light/hvem signaling lymphotoxin beta-deficient mice enhances tnf-induced apoptosis nf-kappab transcription factors virus-induced systemic shock normal t-cell development lymphotoxin-alpha-deficient mice tnf-beta transgenic mice noncanonical nf-kappab pathway tnf receptor-2-deficient mice tumor necrosis factor lymphotoxin-dependent lymphoid neogenesis lt betar-ig protein tnf alpha-deficient mice lymphotoxin-beta receptor lymphotoxin beta receptor tnf family research promotes chemical hepatocarcinogenesis lymphotoxin receptor signaling lymphotoxin alpha/beta lymphotoxin-driven pathway tumour-necrosis factor tumour necrosis factor privacy choices/manage cookies lymphotoxin beta expression de sauvage fj aggarwal bb tumor necrotizing activity roche research foundation device instant download human lymphotoxin derived de fougerolles ar humoral immune response secondary lymphoid tissues chemokine tca4/slc chronic liver injury lymphoid organ development conference paper advances

Questions {❓}

Balkwill F, Mantovani A (2001) Inflammation and cancer: back to Virchow?
Lymphotoxin’s Link to Carcinogenesis: Friend or Foe?
Lymphotoxin’s Link to Carcinogenesis: Friend or Foe?

Schema {🗺️}

ScholarlyArticle:
      headline:Lymphotoxin’s Link to Carcinogenesis: Friend or Foe? From Lymphoid Neogenesis to Hepatocellular Carcinoma and Prostate Cancer
      pageEnd:249
      pageStart:231
      image:https://media.springernature.com/w153/springer-static/cover/book/978-1-4419-6612-4.jpg
      genre:
         Biomedical and Life Sciences
         Biomedical and Life Sciences (R0)
      isPartOf:
         name:Advances in TNF Family Research
         isbn:
            978-1-4419-6612-4
            978-1-4419-6611-7
         type:Book
      publisher:
         name:Springer New York
         logo:
            url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
            type:ImageObject
         type:Organization
      author:
            name:Monika Julia Wolf
            affiliation:
                  name:Institute of Neuropathology, University Hospital Zurich
                  address:
                     name:Department of Pathology, Institute of Neuropathology, University Hospital Zurich, Zurich, Switzerland
                     type:PostalAddress
                  type:Organization
            type:Person
            name:Gitta Maria Seleznik
            affiliation:
                  name:Institute of Neuropathology, University Hospital Zurich
                  address:
                     name:Department of Pathology, Institute of Neuropathology, University Hospital Zurich, Zurich, Switzerland
                     type:PostalAddress
                  type:Organization
            type:Person
            name:Mathias Heikenwalder
            affiliation:
                  name:University Hospital Zurich
                  address:
                     name:Department of Pathology, University Hospital Zurich, Zurich, Switzerland
                     type:PostalAddress
                  type:Organization
            email:[email protected]
            type:Person
      keywords:Experimental Autoimmune Encephalomyelitis, Herpes Virus Entry Mediator, Nasopharyngeal Epithelial Cell, Lymphoid Tissue Inducer Cell, Lymphoid Neogenesis
      description:Tumor necrosis factor alpha (TNFα) was initially discovered due to its tumor necrotizing activity in mice, leading to the death of vascular endothelial cells. Today, pro- and anti-tumorigenic effects of TNF are discussed and TNF as well as other members of the TNF superfamily (TNFSF) were described to play an important role in the development and maintenance of lymphoid tissue and inflammatory reactions. Notably, recent reports indicate a pivotal role of lymphotoxin (LT), a closely related cytokine, in controlling the development of liver and prostate cancer or nasopharyngeal carcinoma – however by distinct mechanisms. Here, we review the pleiotropic functions attributed to LT, the effects of its deregulation and discuss recent literature on LT’s link to cancer.
      datePublished:2011
      isAccessibleForFree:
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Book:
      name:Advances in TNF Family Research
      isbn:
         978-1-4419-6612-4
         978-1-4419-6611-7
Organization:
      name:Springer New York
      logo:
         url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
         type:ImageObject
      name:Institute of Neuropathology, University Hospital Zurich
      address:
         name:Department of Pathology, Institute of Neuropathology, University Hospital Zurich, Zurich, Switzerland
         type:PostalAddress
      name:Institute of Neuropathology, University Hospital Zurich
      address:
         name:Department of Pathology, Institute of Neuropathology, University Hospital Zurich, Zurich, Switzerland
         type:PostalAddress
      name:University Hospital Zurich
      address:
         name:Department of Pathology, University Hospital Zurich, Zurich, Switzerland
         type:PostalAddress
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Person:
      name:Monika Julia Wolf
      affiliation:
            name:Institute of Neuropathology, University Hospital Zurich
            address:
               name:Department of Pathology, Institute of Neuropathology, University Hospital Zurich, Zurich, Switzerland
               type:PostalAddress
            type:Organization
      name:Gitta Maria Seleznik
      affiliation:
            name:Institute of Neuropathology, University Hospital Zurich
            address:
               name:Department of Pathology, Institute of Neuropathology, University Hospital Zurich, Zurich, Switzerland
               type:PostalAddress
            type:Organization
      name:Mathias Heikenwalder
      affiliation:
            name:University Hospital Zurich
            address:
               name:Department of Pathology, University Hospital Zurich, Zurich, Switzerland
               type:PostalAddress
            type:Organization
      email:[email protected]
PostalAddress:
      name:Department of Pathology, Institute of Neuropathology, University Hospital Zurich, Zurich, Switzerland
      name:Department of Pathology, Institute of Neuropathology, University Hospital Zurich, Zurich, Switzerland
      name:Department of Pathology, University Hospital Zurich, Zurich, Switzerland
WebPageElement:
      isAccessibleForFree:
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External Links {🔗}(298)

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