Here's how LINK.SPRINGER.COM makes money* and how much!

*Please read our disclaimer before using our estimates.
Loading...

LINK . SPRINGER . COM {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Link.springer.com Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Schema
  10. External Links
  11. Analytics And Tracking
  12. Libraries

We are analyzing https://link.springer.com/chapter/10.1007/978-0-387-09794-7_8.

Title:
Mitochondrial Oscillations in Physiology and Pathophysiology | SpringerLink
Description:
Oscillations in chemical reactions and metabolic pathways have historiacally served as prototypes for understanding the dynamics of complex nonlinear systems. This chapter reviews the oscillatory behavior of mitochondria, with a focus on the mitochondrial oscillator...
Website Age:
28 years and 1 months (reg. 1997-05-29).

Matching Content Categories {📚}

  • Education
  • Science
  • Telecommunications

Content Management System {📝}

What CMS is link.springer.com built with?

Custom-built

No common CMS systems were detected on Link.springer.com, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of link.springer.com audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

check SE Ranking
check Ahrefs
check Similarweb
check Ubersuggest
check Semrush

How Does Link.springer.com Make Money? {💸}

We can't figure out the monetization strategy.

Some websites aren't about earning revenue; they're built to connect communities or raise awareness. There are numerous motivations behind creating websites. This might be one of them. Link.springer.com could be getting rich in stealth mode, or the way it's monetizing isn't detectable.

Keywords {🔍}

google, scholar, cas, pubmed, article, mitochondrial, oscillations, biophys, mitochondria, biochem, aon, cell, cortassa, orourke, heart, oscillatory, membrane, chapter, biol, cells, control, reduced, nucleotide, calcium, physiol, cellular, metabolic, cardiac, release, chem, biochim, acta, mechanisms, ros, network, potential, usa, arch, permeability, transition, privacy, cookies, content, analysis, information, publish, oscillators, access, download, spontaneous,

Topics {✒️}

month download article/chapter continuous sr2+-induced oscillations digitonin-treated tetrahymena pyriformis time-dependent membrane potentials high-amplitude oscillatory mode induced ros release reduced nicotinamide-adenine dinucleotide ultradian clock-driven oscillations springer science+business media ischemia/reperfusion scenario induced mitochondrial oscillations privacy choices/manage cookies sr ca2+ release mitochondrial membrane potential reduced diphosphopyridine nucleotide van der mark mitochondrial permeability transition cellular oscillatory mechanisms permeability pore transition permeability transition pore evtodienko yv spontaneous mitochondrial depolarizations device instant download mitochondrial calcium spiking reduced phosphopyridine nucleotide mitochondrial metabolism triggered editor information editors small-amplitude oscillations mitochondrial oscillator dependent cell-free extract cytosolic calcium oscillations main low-frequency mitochondrial energy conservation mitochondrial ion channels cardiac impulse propagation membrane anion uniporter membrane anion channel single rat cardiomyocytes mitochondrial redox waves mitochondrial fluorescence patterns download preview pdf cell calcium signalling european economic area reactive oxygen species long term recordings hydrogen peroxide diffusion rapp pe european biochemical societies 2h+/me2+ exchange robb-gaspers ld

Questions {❓}

  • Why homeodynamics, not homeostasis?

Schema {🗺️}

ScholarlyArticle:
      headline:Mitochondrial Oscillations in Physiology and Pathophysiology
      pageEnd:117
      pageStart:98
      image:https://media.springernature.com/w153/springer-static/cover/book/978-0-387-09794-7.jpg
      genre:
         Biomedical and Life Sciences
         Biomedical and Life Sciences (R0)
      isPartOf:
         name:Cellular Oscillatory Mechanisms
         isbn:
            978-0-387-09794-7
            978-0-387-09793-0
         type:Book
      publisher:
         name:Springer New York
         logo:
            url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
            type:ImageObject
         type:Organization
      author:
            name:Miguel A. Aon
            affiliation:
                  name:The Johns Hopkins University
                  address:
                     name:Division of Cardiology Institute of Molecular Cardiobiology, The Johns Hopkins University, Baltimore, USA
                     type:PostalAddress
                  type:Organization
            type:Person
            name:Sonia Cortassa
            affiliation:
                  name:The Johns Hopkins University
                  address:
                     name:Division of Cardiology Institute of Molecular Cardiobiology, The Johns Hopkins University, Baltimore, USA
                     type:PostalAddress
                  type:Organization
            type:Person
            name:Brian O’Rourke
            affiliation:
                  name:The Johns Hopkins University
                  address:
                     name:Division of Cardiology Institute of Molecular Cardiobiology, The Johns Hopkins University, Baltimore, USA
                     type:PostalAddress
                  type:Organization
            email:[email protected]
            type:Person
      keywords:Reactive Oxygen Species Production, Mitochondrial Membrane Potential, Permeability Transition Pore, Mitochondrial Reactive Oxygen Species, Reactive Oxygen Species Accumulation
      description:Oscillations in chemical reactions and metabolic pathways have historiacally served as prototypes for understanding the dynamics of complex nonlinear systems. This chapter reviews the oscillatory behavior of mitochondria, with a focus on the mitochondrial oscillator dependent on reactive oxygen species (ROS), as first described in heart cells. Experimental and theoretical evidence now indicates that mitochondrial energetic variables oscillate autonomously as part of a network of coupled oscillators under both physiological and pathological conditions. The physiological domain is characterized by small-amplitude oscillations in mitochondrial membrane potential (ΔΨm) showing correlated behavior over a wide range of frequencies, as determined using Power Spectral Analysis and Relative Dispersion Analysis of long term recordings of ΔΨm. Under metabolic stress, when the balance between ROS generation and ROS scavenging is perturbed, the mitochondrial network throughout the cell locks to one main low-frequency, high-amplitude oscillatory mode. This behavior has major pathological implications because the energy dissipation and cellular redox changes that occur during ΔΨm depolarization result in suppression of electrical excitability and Ca2+ handling, the two main functions of the cardiac cell. In an ischemia/reperfusion scenario these alterations scale up to the level of the whole organ, giving rise to fatal arrhythmias.
      datePublished:2008
      isAccessibleForFree:
      hasPart:
         isAccessibleForFree:
         cssSelector:.main-content
         type:WebPageElement
      context:https://schema.org
Book:
      name:Cellular Oscillatory Mechanisms
      isbn:
         978-0-387-09794-7
         978-0-387-09793-0
Organization:
      name:Springer New York
      logo:
         url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
         type:ImageObject
      name:The Johns Hopkins University
      address:
         name:Division of Cardiology Institute of Molecular Cardiobiology, The Johns Hopkins University, Baltimore, USA
         type:PostalAddress
      name:The Johns Hopkins University
      address:
         name:Division of Cardiology Institute of Molecular Cardiobiology, The Johns Hopkins University, Baltimore, USA
         type:PostalAddress
      name:The Johns Hopkins University
      address:
         name:Division of Cardiology Institute of Molecular Cardiobiology, The Johns Hopkins University, Baltimore, USA
         type:PostalAddress
ImageObject:
      url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
Person:
      name:Miguel A. Aon
      affiliation:
            name:The Johns Hopkins University
            address:
               name:Division of Cardiology Institute of Molecular Cardiobiology, The Johns Hopkins University, Baltimore, USA
               type:PostalAddress
            type:Organization
      name:Sonia Cortassa
      affiliation:
            name:The Johns Hopkins University
            address:
               name:Division of Cardiology Institute of Molecular Cardiobiology, The Johns Hopkins University, Baltimore, USA
               type:PostalAddress
            type:Organization
      name:Brian O’Rourke
      affiliation:
            name:The Johns Hopkins University
            address:
               name:Division of Cardiology Institute of Molecular Cardiobiology, The Johns Hopkins University, Baltimore, USA
               type:PostalAddress
            type:Organization
      email:[email protected]
PostalAddress:
      name:Division of Cardiology Institute of Molecular Cardiobiology, The Johns Hopkins University, Baltimore, USA
      name:Division of Cardiology Institute of Molecular Cardiobiology, The Johns Hopkins University, Baltimore, USA
      name:Division of Cardiology Institute of Molecular Cardiobiology, The Johns Hopkins University, Baltimore, USA
WebPageElement:
      isAccessibleForFree:
      cssSelector:.main-content

External Links {🔗}(221)

Analytics and Tracking {📊}

  • Google Tag Manager

Libraries {📚}

  • Clipboard.js

4.44s.