
LINK . SPRINGER . COM {
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Title:
The Basic Biology of Metastasis | SpringerLink
Description:
It is the ability to metastasize that makes cancer a fatal disease. Of the 555,500 cancer-related deaths expected this year in the United States, the vast majority are due to the development of metastatic disease rather than the primary tumor (Jemal et al., 2002)....
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Topics {βοΈ}
stress-activated protein kinase-2/p38 parathyroid hormone-related protein nm23-transfected melanoma cells endothelium-attached tumor cells activated ras regulates human colon cancer human bladder tumour cell adhesion proteins squamous cell carcinoma privacy choices/manage cookies clinically localized carcinoma human melanoma cells vascular channel formation specific secondary sites circulating prostate cells download preview pdf micrometastatic tumour cells prostatic cancer located metastasis suppressor gene human melanoma metastasis tumor cell proliferation e-cadherin expression human glandular kallikrein prostate cancer metastases experimental cell research breast cancer metastasis kinase substrate prostate cancer progression metastasis suppressor pathways rhogdi2 gene expression solitary cancer cells european economic area complete remissions tend 125 i-5-iodo-2β-deoxyuridine anti-angiogenic therapy biochimica biophysica acta neoadjuvant androgen deprivation organspecific metastatic environment late metastatic relapse winship cancer institute prostate cancer metastasis conditions privacy policy national cancer institute matrix metalloproteinase activity reduced tumor incidence anti-metastasis therapy metastasis suppressor genes tumor-bone interactions van der vliet mammary gland biology
Questions {β}
- (2001) Solitary cancer cells as a possible source of tumour dormancy?
Schema {πΊοΈ}
ScholarlyArticle:
headline:The Basic Biology of Metastasis
pageEnd:21
pageStart:1
image:https://media.springernature.com/w153/springer-static/cover/book/978-1-4419-9129-4.jpg
genre:
Medicine
Medicine (R0)
isPartOf:
name:The Biology of Skeletal Metastases
isbn:
978-1-4419-9129-4
978-1-4613-4803-0
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publisher:
name:Springer US
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author:
name:Victoria L. Robinson
affiliation:
name:The University of Chicago
address:
name:Section of Urology, Department of Surgery, The University of Chicago, Chicago, USA
type:PostalAddress
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type:Person
name:Eric C. Kauffman
affiliation:
name:The University of Chicago
address:
name:Section of Urology, Department of Surgery, The University of Chicago, Chicago, USA
type:PostalAddress
type:Organization
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name:Mitchell H. Sokoloff
affiliation:
name:The University of Chicago
address:
name:Section of Urology, Department of Surgery, The University of Chicago, Chicago, USA
type:PostalAddress
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name:Carrie W. Rinker-Schaeffer
affiliation:
name:The University of Chicago
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name:Section of Urology, Department of Surgery, The University of Chicago, Chicago, USA
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name:The University of Chicago
address:
name:Section of Hematotogy/Oncology, Department of Medicine, The University of Chicago, Chicago, USA
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name:The University of Chicago Comprehensive Cancer Research Center
address:
name:The Genitourinary Oncology Research Program, The University of Chicago Comprehensive Cancer Research Center, Chicago, USA
type:PostalAddress
type:Organization
type:Person
keywords:Prostate Cancer, Skeletal Metastasis, Vasculogenic Mimicry, Secondary Site, Metastatic Cascade
description:It is the ability to metastasize that makes cancer a fatal disease. Of the 555,500 cancer-related deaths expected this year in the United States, the vast majority are due to the development of metastatic disease rather than the primary tumor (Jemal et al., 2002). Although treatment is available for patients with metastatic cancer, options are limited and responses can be quite variable. Furthermore, complete remissions tend to be limited in both scope and duration.
datePublished:2004
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