
FEBS . ONLINELIBRARY . WILEY . COM {
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Title:
Honokiol inhibits epithelial—mesenchymal transition in breast cancer cells by targeting signal transducer and activator of transcription 3/Zeb1/E‐cadherin axis - Avtanski - 2014 - Molecular Oncology - Wiley Online Library
Description:
Shows that honokiol inhibits EMT in breast cancer cells. Reveals that Stat3 inhibition is integral for honokiol-mediated EMT inhibition. Mechanistic insights show the involvement of Stat3/Zeb1/E-cad...
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30 years and 8 months (reg. 1994-10-12).
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pubmatic.com, appnexus.com, rubiconproject.com, indexexchange.com, openx.com, smaato.com, ad-generation.jp, districtm.io, yieldmo.com, rhythmone.com, yahoo.com, gumgum.com, admanmedia.com, sharethrough.com, emxdgt.com, contextweb.com, smartadserver.com, themediagrid.com, sovrn.com, lijit.com, beachfront.com, improvedigital.com, mintegral.com, sonobi.com, onetag.com, media.net, risecodes.com, uis.mobfox.com, mediago.io, adyoulike.com, minutemedia.com, visiblemeasures.com, undertone.com, imds.tv, kargo.com, start.io, connectad.io, onlinemediasolutions.com, supply.colossusssp.com, opera.comHow Much Does Febs.onlinelibrary.wiley.com Make? {💰}
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Keywords {🔍}
cells, hnk, stat, cancer, breast, expression, emt, zeb, honokiol, figure, ecadherin, inhibition, treated, analysis, treatment, promoter, mcf, epithelial, mdamb, inhibits, tumor, hnkmediated, vehicle, mesenchymal, stattic, markers, cell, subjected, tumors, total, invasion, migration, isolated, activation, potential, mcfa, vimentin, control, active, increased, evidence, recruitment, role, occludin, assay, rna, constitutively, molecular, metastatic, results,
Topics {✒️}
block tgfβ/tnfα-induced emt article information search tgfβ + tnfα + hnk-treated cells stat3/zeb1/e-cadherin axis tgfβ/tnfα treatment induced dna-binding transcription factor inducing mesenchymal–epithelial transition real-time quantitative pcr epithelium-derived tumors switching previously unrecognized cross-talk cross blood–brain barrier potentiated hnk-mediated release mda-mb-231 cell line tgfβ + tnfα-treated cells putative stat3-binding sites potentiated hnk-mediated inhibition putative zeb1-binding sites hnk-mediated increased expression potentiates hnk-mediated inhibition emt-related select mirnas increased e-cadherin expression e-cadherin expression levels stat3-dependent manner leading spindle-shaped cells mda-mb-231 cells treated diminished hnk-mediated activation epithelial–mesenchymal transition mammary epithelial cells anti-stat3 antibodies revealed specific anti-adhesive antibodies hnk-mediated zeb1 inhibition wild-type stat3 hnk-mediated emt inhibition natural phenolic compound constitutively active form small molecule inhibitor photographed 48 h-post treatment tgfβ/tnfα treatment constitutively active stat3 tgfβ + tnfα +hnk cancer cell lines cell line authentication epithelial marker profiles effectively inhibits phosphorylation hnk-mediated molecular increased e-cadherin hnk triggers release zeb1-mediated repressive effect stat3-mediated release underlying molecular mechanisms
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