FACULTYOPINIONS . COM {}

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We began analyzing https://archive.connect.h1.co/article/1121852/, but it redirected us to https://archive.connect.h1.co/article/1121852/. The analysis below is for the second page.

Title[redir]:
Bifurcation dynamics in lineage-co ... | Article | H1 Connect
Description:
Lineage specification of multipotent progenitor cells is governed by a balance of lineage-affiliated transcription factors, such as GATA1 and PU.1, which r

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Keywords {🔍}

latest, recommendation, finding, cell, cells, biology, lineage, developmental, interesting, hypothesis, decision, commitment, confirmation, faculty, progenitor, sep, fate, control, gene, expression, molecular, progenitors, jan, bifurcation, dynamics, bipotent, huang, alfonso, martinez, arias, precursor, myelomonocytic, lineages, gata, transcription, factors, manuscript, dynamical, transition, mechanisms, morphogenesis, apr, controls, jul, oct, liver, good, teaching, singlecell, human,

Topics {✒️}

developmental molecular mechanisms t-cell lineage commitment bipotent progenitor cells single-cell roadmap mammalian progenitor cells cell fate decision 2023 confirmation good human esc models developmental biology lineage-committed cells gene expression dynamical system makes embryonic brain development megakaryocyte/erythrocyte lineages archived bifurcation dynamics liver regeneration cell development faculty opinions finding reprogramming finding dedifferentiation cell progenitors lineage bifurcation lineage-commitment lineage commitment dendritic cells h1 company decision depends 22 latest recommendation erythroid/megakaryocyte myelomonocytic lineages dynamical systems commitment spectrum finding finding 1 doctors search difficult paper 1 transcription factors similar pieces work exist strong anchor experimental results metastable state multilineage priming attractors representing intuitive predictions transcriptome chang hh haematopoietic progenitors malcolm alison 26 ng kk

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ScholarlyArticle:
      context:https://schema.org
      headline:Bifurcation dynamics in lineage-commitment in bipotent progenitor cells.
      abstract:Lineage specification of multipotent progenitor cells is governed by a balance of lineage-affiliated transcription factors, such as GATA1 and PU.1, which regulate the choice between erythroid and myelomonocytic fates. But how ratios of lineage-determining transcription factors stabilize progenitor cells and resolve their indeterminacy to commit them to discrete, mutually exclusive fates remains unexplained. We used a simple model and experimental measurements to analyze the dynamics of a binary fate decision governed by a gene-circuit containing auto-stimulation and cross-inhibition, as embodied by the GATA1-PU.1 paradigm. This circuit generates stable attractors corresponding to erythroid and myelomonocytic fates, as well as an uncommitted metastable state characterized by coexpression of both regulators, explaining the phenomenon of "multilineage priming". GATA1 and PU.1 mRNA and transcriptome dynamics of differentiating progenitor cells confirm that commitment occurs in two stages, as suggested by the model: first, the progenitor state is destabilized in an almost symmetrical bifurcation event, resulting in a poised state at the boundary between the two lineage-specific attractors; second, the cell is driven to the respective, now accessible attractors. This minimal model captures fundamental features of binary cell fate decisions, uniting the concepts of stochastic (selective) and deterministic (instructive) regulation, and hence, may apply to a wider range of binary fate decision points.
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WebPage:
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