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DOI . ORG {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Doi.org Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Schema
  10. Social Networks
  11. External Links
  12. Analytics And Tracking
  13. Libraries
  14. Hosting Providers
  15. CDN Services

We began analyzing https://elifesciences.org/articles/01202, but it redirected us to https://elifesciences.org/articles/01202. The analysis below is for the second page.

Title[redir]:
Expansion of intestinal Prevotella copri correlates with enhanced susceptibility to arthritis | eLife
Description:
We share our bodies with a diverse set of microorganisms, known collectively as the human microbiome. Indeed, estimates suggest that our bodies contain 10 times as many microbial cells as human cells. Our stomach and intestines alone are home to many hundreds and possibly thousands of microbial species that break down indigestible compounds and help to prevent the growth of harmful bacteria. The immune system must therefore learn to tolerate these microorganisms, while retaining the ability to launch attacks against microorganisms that cause harm. Failure of this process may increase the risk of autoimmune diseases in which the body mistakenly attacks its own cells and tissues. Rheumatoid arthritis is a chronic autoimmune disease marked by inflammation of the joints. Although the causes of rheumatoid arthritis are unknown, mice with mutations that increase the risk of the disease remain healthy if they are kept under sterile conditions. However, if these mice are exposed to certain species of bacteria sometimes found in the gut, they begin to show signs of joint inflammation. Here, Scher et al. used genome sequencing to compare gut bacteria from patients with rheumatoid arthritis and healthy controls. A bacterial species called Prevotella copri was more abundant in patients suffering from untreated rheumatoid arthritis than in healthy individuals. Moreover, the presence of P. copri corresponded to a reduction in the abundance of other bacterial groups—including a number of beneficial microbes. In a mouse model of gut inflammation, animals colonized with P. copri had more severe disease than controls, consistent with a pro-inflammatory function of this organism. Current treatments for rheumatoid arthritis target symptoms. However, by highlighting the role played by gut bacteria, the work of Scher et al. suggests that novel treatment options focused on curbing the spread of P. copri in the gut could delay or prevent the onset of this disease.

Matching Content Categories {📚}

  • Science
  • Health & Fitness
  • Education

Content Management System {📝}

What CMS is doi.org built with?

Custom-built

No common CMS systems were detected on Doi.org, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of doi.org audience?

🌟 Strong Traffic: 100k - 200k visitors per month


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How Does Doi.org Make Money? {💸}

We can't see how the site brings in money.

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Keywords {🔍}

figure, copri, nora, data, arthritis, google, scholar, prevotella, subjects, healthy, microbiota, disease, patients, gut, reads, abundance, mice, analysis, rheumatoid, human, httpsdoiorgelife, genome, supplement, orfs, download, microbiome, medicine, competing, interests, york, university, united, protocol, otu, asset, article, states, genes, present, source, samples, controls, presence, cells, request, detailed, intestinal, bacteroides, metagenomic, inflammation,

Topics {✒️}

005 download elife-01202-fig1-data1-v1 006 download elife-01202-fig1-data2-v1 010 download elife-01202-fig2-data1-v1 011 download elife-01202-fig2-data2-v1 015 download elife-01202-fig3-data1-v1 016 download elife-01202-fig3-data2-v1 020 download elife-01202-fig4-data1-v1 022 download elife-01202-fig5-data1-v1 024 download elife-01202-fig6-data1-v1 disease-modifying anti-rheumatic drugs prevotella-dominated metagenome reveals anti-citrullinated peptide antibodies vancomycin-resistant enterococcus domination disease-modifying antirheumatic drugs segmented filamentous bacteria bray-curtis distances demonstrated anti–citrullinated protein antibodies sequence-based typing methodology patient-derived strains showed hla-class ii genotype metagenome-wide association study obtain draft patient-derived foxp3+ cd4+ t-cells circulating pro-inflammatory cytokines paired-end reads aligning residing microbial communities dss-induced colitis request class ii locus abdollahi-roodsaz 2 weeks post-gavage shared-epitope risk alleles gut microbiota richness tissue-specific autoimmune disease chemically induced colitis phosphoadenosine phosphosulfate reductase hla-drb1 alleles supports otu-based microbial diversity zanin-zhorov c-reactive protein terminal electron acceptors elife sciences publications local immune response intestinal lamina propria united states contribution antibiotic-treated c57bl/6 mice inter-individual strain variability confer higher risk genetically susceptible host thioredoxin-thioredoxin reductase system dhf reductase-high microbiota

Questions {❓}

  • Gov/bioproject/?

Schema {🗺️}

ScholarlyArticle:
      context:https://schema.org
      mainEntityOfPage:
         type:WebPage
         id:https://elifesciences.org/articles/01202
      headline:Expansion of intestinal Prevotella copri correlates with enhanced susceptibility to arthritis
      datePublished:2013-11-05
      author:
            type:Person
            name:Jose U Scher
            type:Person
            name:Andrew Sczesnak
            type:Person
            name:Randy S Longman
            type:Person
            name:Nicola Segata
            type:Person
            name:Carles Ubeda
            type:Person
            name:Craig Bielski
            type:Person
            name:Tim Rostron
            type:Person
            name:Vincenzo Cerundolo
            type:Person
            name:Eric G Pamer
            type:Person
            name:Steven B Abramson
            type:Person
            name:Curtis Huttenhower
            type:Person
            name:Dan R Littman
      publisher:
         type:Organization
         name:eLife Sciences Publications, Ltd
         logo:
            type:ImageObject
            url:https://elifesciences.org/assets/patterns/img/patterns/organisms/[email protected]
      keywords:
         microbiome
         inflammation
         autoimmunity
         metagenomics
         rheumatoid
         arthritis
      about:
         Immunology and Inflammation
      description:The sequencing of microbial genomes reveals that the presence of a particular microbial species in the gut may increase the risk of the autoimmune disease rheumatoid arthritis.
      isPartOf:
         type:Periodical
         name:eLife
         issn:2050-084X
WebPage:
      id:https://elifesciences.org/articles/01202
Person:
      name:Jose U Scher
      name:Andrew Sczesnak
      name:Randy S Longman
      name:Nicola Segata
      name:Carles Ubeda
      name:Craig Bielski
      name:Tim Rostron
      name:Vincenzo Cerundolo
      name:Eric G Pamer
      name:Steven B Abramson
      name:Curtis Huttenhower
      name:Dan R Littman
Organization:
      name:eLife Sciences Publications, Ltd
      logo:
         type:ImageObject
         url:https://elifesciences.org/assets/patterns/img/patterns/organisms/[email protected]
ImageObject:
      url:https://elifesciences.org/assets/patterns/img/patterns/organisms/[email protected]
Periodical:
      name:eLife
      issn:2050-084X

Social Networks {👍}(10)

External Links {🔗}(531)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Tag Manager

Libraries {📚}

  • Highcharts
  • PhotoSwipe

Emails and Hosting {✉️}

Mail Servers:

  • mx.zoho.eu
  • mx2.zoho.eu
  • mx3.zoho.eu

Name Servers:

  • josh.ns.cloudflare.com
  • zita.ns.cloudflare.com

CDN Services {📦}

  • Cloudflare
  • Hypothes

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