DOI . ORG {}

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We began analyzing https://www.tandfonline.com/doi/abs/10.4161/cc.8.20.9761, but it redirected us to https://www.tandfonline.com/doi/abs/10.4161/cc.8.20.9761. The analysis below is for the second page.

Title[redir]:
Retinoid signaling regulates breast cancer stem cell differentiation: Cell Cycle: Vol 8, No 20
Description:
The cancer stem cell (CSC) hypothesis implicates the development of new therapeutic approaches to target the CSC population. Characterization of the pathways that regulate CSCs activity will facili...

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Education
Science
Non-Profit & Charity

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What CMS is doi.org built with?

Custom-built

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🌠 Phenomenal Traffic: 5M - 10M visitors per month

Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
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How Does Doi.org Make Money? {💸}

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Keywords {🔍}

journals, open, sciences, permissions, access, search, taylor, francis, page, browse, journal, studies, articles, find, environment, science, breast, article, group, publish, health, social, information, cell, technology, development, register, read, retinoid, signaling, reprints, cscs, corporate, content, close, menu, select, calls, papers, suggester, publishing, guidance, author, services, bioscience, earth, engineering, humanities, medicine, physical,

Topics {✒️}

social care medicine cancer stem cell francis group retinoid signaling twitter page taylor request academic permissions date register journal search calls target breast cscs find guidance journals books regulate cscs activity identified gene sets receive personalised research journals browse breast cscs citing articles based obtain permissions instantly corporate permissions access rights cart search cell lines read lists articles provide access reprints differentiation journals a open view content information crossref citations obtain permissions register log issues volume 8 selecting permissions permissions form publish page published online enzymatic activity taylor search crossref icon find leisure tourism scheduled work julien wicinski nathalie cervera florence monville pascal finetti

Questions {❓}

Please note: Selecting permissions does not provide access to the full text of the article, please see our help page How do I view content?
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Schema {🗺️}

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            name:Retinoid signaling regulates breast canc ....
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      name:Retinoid signaling regulates breast canc ....
PublicationIssue:
      id:#issue
      issueNumber:20
      datePublished:2009-10-15
      isPartOf:
         id:#periodical
         type:
            PublicationVolume
            Periodical
         name:Cell Cycle
         issn:
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["PublicationVolume","Periodical"]:
      id:#periodical
      name:Cell Cycle
      issn:
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         1551-4005
      volumeNumber:8
      publisher:Taylor & Francis Group
ScholarlyArticle:
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      isPartOf:#periodical
      sameAs:https://doi.org/10.4161/cc.8.20.9761
      identifier:10.4161/cc.8.20.9761
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      articleSection:Extra Views
      name:Retinoid signaling regulates breast cancer stem cell differentiation
      headline:Retinoid signaling regulates breast cancer stem cell differentiation
      abstract:The cancer stem cell (CSC) hypothesis implicates the development of new therapeutic approaches to target the CSC population. Characterization of the pathways that regulate CSCs activity will facilitate the development of targeted therapies. We recently reported that the enzymatic activity of ALDH1, as measured by the ALDELFUOR assay, can be utilized to isolate normal and malignant breast stem cells in both primary tumors and cell lines. In this study, utilizing a tumorsphere assay, we have demonstrated the role of retinoid signaling in the regulation of breast CSCs self-renewal and differentiation. Utilizing the gene set enrichment analysis (GSEA) algorithm we identified gene sets and pathways associated with retinoid signaling. These pathways regulate breast CSCs biology and their inhibition may provide novel therapeutic approaches to target breast CSCs.
      description:The cancer stem cell (CSC) hypothesis implicates the development of new therapeutic approaches to target the CSC population. Characterization of the pathways that regulate CSCs activity will facilitate the development of targeted therapies. We recently reported that the enzymatic activity of ALDH1, as measured by the ALDELFUOR assay, can be utilized to isolate normal and malignant breast stem cells in both primary tumors and cell lines. In this study, utilizing a tumorsphere assay, we have demonstrated the role of retinoid signaling in the regulation of breast CSCs self-renewal and differentiation. Utilizing the gene set enrichment analysis (GSEA) algorithm we identified gene sets and pathways associated with retinoid signaling. These pathways regulate breast CSCs biology and their inhibition may provide novel therapeutic approaches to target breast CSCs.
      author:
            type:Person
            name:Christophe Ginestier
            type:Person
            name:Nathalie Cervera
            type:Person
            name:Florence Monville
            type:Person
            name:Christophe Ginestier
            type:Person
            name:Daniel Birnbaum
            type:Person
            name:Julien Wicinski
            type:Person
            name:Pascal Finetti
            type:Person
            name:Max S. Wicha
            type:Person
            name:FranÃ§ois Bertucci
            type:Person
            name:Emmanuelle Charafe-Jauffret
      pageStart:3297
      pageEnd:3302
      datePublished:2009-10-15
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         type:Organization
         name:Taylor & Francis
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Person:
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      name:Nathalie Cervera
      name:Florence Monville
      name:Christophe Ginestier
      name:Daniel Birnbaum
      name:Julien Wicinski
      name:Pascal Finetti
      name:Max S. Wicha
      name:FranÃ§ois Bertucci
      name:Emmanuelle Charafe-Jauffret
Organization:
      name:Taylor & Francis
      logo:
         type:ImageObject
         url:https://www.tandfonline.com/pb-assets/Images/Taylor_and_Francis_Group_Logo-1742461082.png
ImageObject:
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External Links {🔗}(104)

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