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We began analyzing https://journals.aai.org/jimmunol/article/183/1/66/84370/Aryl-Hydrocarbon-Receptor-Activation-Inhibits-In, but it redirected us to https://journals.aai.org/jimmunol/article/183/1/66/84370/Aryl-Hydrocarbon-Receptor-Activation-Inhibits-In. The analysis below is for the second page.

Title[redir]:
Aryl Hydrocarbon Receptor Activation Inhibits In Vitro Differentiation of Human Monocytes and Langerhans Dendritic Cells1 | The Journal of Immunology | American Association of Immunologists
Description:
Abstract. The transcription factor aryl hydrocarbon receptor (AhR) represents a promising therapeutic target in allergy and autoimmunity. AhR signaling ind

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Keywords {🔍}

cells, ahr, vaf, cell, differentiation, fig, expression, cultures, lcs, activation, monocyte, monocytes, progenitor, blood, human, generation, presence, cda, receptor, generated, granulocytes, monocytic, analyzed, signaling, levels, induction, absence, hydrocarbon, dcs, shown, maturation, aryl, effect, ngml, vitro, analysis, ligands, tgfβ, dendritic, inhibition, cultured, experiments, gfp, open, gene, myeloid, expansion, control, days, inhibited,

Topics {✒️}

real-time rt-pcr analysis tnf-α/nf-κb signaling impairment tnf-α/nf-κb signaling real-time rt-pcr tnf-α-induced nfκb activation basic helix-loop-helix/ transforming growth factor-β1 g-csf-dependent granulocyte differentiation privacy policy transforming growth factor-β block nf-κb p65 healthy full-term deliveries tgf-β1-dependent lc cultures empty vector pbmn-ires-gfp share functional characteristics granulocyte colony-stimulating factor fitc-conjugated mabs specific serum-free conditions allophycocyanin-conjugated mabs specific langerhans-type dendritic cells potent anti-inflammatory activity pe-conjugated mabs specific tgf-β1 costimulation induces anti-cd40-induced activation open biosystems mimic tgf-β1 signaling lc-type dendritic cells cd14+cd11bdim/neg cells boosting il-17-producing helper lentiviral gipz-short-hairpin human monocyte-derived dcs retroviral construct pbmn-pu lineage-determining transcriptional processes cis-regulatory dna elements tgf-β1-dependent manner specific anti-proliferative effect m-csf/il-6-supplemented cultures monocyte-specific differentiation conditions human cord blood cell-dependent allergic asthma ah-receptor cdna reveals monocyte-derived dc generation monocyte/dc subset differentiation monocyte/dc lineage commitment 8 tetrachlorodibenzo-p-dioxin main modulatory effect generate monocyte-derived dcs m-csf-dependent monocyte ahr nuclear translocator mediating agonist-induced activation

Schema {🗺️}

ScholarlyArticle:
      context:https://schema.org
      id:https://journals.aai.org/jimmunol/article/183/1/66/84370/Aryl-Hydrocarbon-Receptor-Activation-Inhibits-In
      name:Aryl Hydrocarbon Receptor Activation Inhibits In Vitro Differentiation of Human Monocytes and Langerhans Dendritic Cells1
      datePublished:2009-07-01
      isPartOf:
         id:https://journals.aai.org/jimmunol/issue/183/1
         type:PublicationIssue
         issueNumber:1
         datePublished:2009-07-01
         isPartOf:
            id:https://journals.aai.org/jimmunol
            type:Periodical
            name:The Journal of Immunology
            issn:
               1550-6606
      url:https://dx.doi.org/10.4049/jimmunol.0802997
      inLanguage:en
      copyrightHolder:
      copyrightYear:2025
      publisher:
      author:
            name:Platzer, Barbara
            affiliation:* Division of Gastroenterology and Nutrition, Children’s Hospital Boston, Harvard Medical School, Boston, MA 02115;
            type:Person
            name:Richter, Susanne
            affiliation:† Institute of Immunology, Center of Physiology, Pathophysiology and Immunology, Medical University Vienna, Vienna, Austria; and
            type:Person
            name:Kneidinger, Doris
            affiliation:† Institute of Immunology, Center of Physiology, Pathophysiology and Immunology, Medical University Vienna, Vienna, Austria; and
            type:Person
            name:Waltenberger, Darina
            affiliation:† Institute of Immunology, Center of Physiology, Pathophysiology and Immunology, Medical University Vienna, Vienna, Austria; and
            type:Person
            name:Woisetschläger, Maximilian
            affiliation:‡ Department of Autoimmunity and Transplantation, Novartis Institutes for Biomedical Research (NIBR), Vienna, Austria
            type:Person
            name:Strobl, Herbert
            affiliation:† Institute of Immunology, Center of Physiology, Pathophysiology and Immunology, Medical University Vienna, Vienna, Austria; and
            type:Person
      description:Abstract. The transcription factor aryl hydrocarbon receptor (AhR) represents a promising therapeutic target in allergy and autoimmunity. AhR signaling ind
      pageStart:66
      pageEnd:74
      siteName:American Association of Immunologists
      thumbnailURL:https://aai.silverchair-cdn.com/aai/content_public/journal/jimmunol/issue/183/1/2/m_jimmunol_183_1_cover.png?Expires=1814123631&Signature=AKHMrOs-lqB1dtiM8siJw3uo5tM8xQVAWkCvil8EGjCkQkKR~mViZkDHHaW8Oex~W6w5BL7Dooe58fVr8zDwwvqMqcVw4a5linww1ty9jZ-lWJIdKJkt8rsOml~D5WbJGlO~FJ5wnDYnvz4S4GJtaYRhSs3NhzjrX2bGHf34TyPjm1rLT9GBv-LvQjXuPjSztMRHAbWLUisxqK~6IGa3GdY-RrNqBstzpKB05-ccEA4cc9H9AeVzD6A0i6p8K~1c7jC9rxHGrTnKnIwI-fjP8-h1kVUajApMjXbe76jKmlbjy8pAScof2H8Cd1nMGy6slBhxR7LjNsSAeBHnxF60AA__&Key-Pair-Id=APKAIE5G5CRDK6RD3PGA
      headline:Aryl Hydrocarbon Receptor Activation Inhibits In Vitro Differentiation of Human Monocytes and Langerhans Dendritic Cells1
      image:https://aai.silverchair-cdn.com/aai/content_public/journal/jimmunol/issue/183/1/2/m_jimmunol_183_1_cover.png?Expires=1814123631&Signature=AKHMrOs-lqB1dtiM8siJw3uo5tM8xQVAWkCvil8EGjCkQkKR~mViZkDHHaW8Oex~W6w5BL7Dooe58fVr8zDwwvqMqcVw4a5linww1ty9jZ-lWJIdKJkt8rsOml~D5WbJGlO~FJ5wnDYnvz4S4GJtaYRhSs3NhzjrX2bGHf34TyPjm1rLT9GBv-LvQjXuPjSztMRHAbWLUisxqK~6IGa3GdY-RrNqBstzpKB05-ccEA4cc9H9AeVzD6A0i6p8K~1c7jC9rxHGrTnKnIwI-fjP8-h1kVUajApMjXbe76jKmlbjy8pAScof2H8Cd1nMGy6slBhxR7LjNsSAeBHnxF60AA__&Key-Pair-Id=APKAIE5G5CRDK6RD3PGA
      image:alt:Issue Cover
PublicationIssue:
      id:https://journals.aai.org/jimmunol/issue/183/1
      issueNumber:1
      datePublished:2009-07-01
      isPartOf:
         id:https://journals.aai.org/jimmunol
         type:Periodical
         name:The Journal of Immunology
         issn:
            1550-6606
Periodical:
      id:https://journals.aai.org/jimmunol
      name:The Journal of Immunology
      issn:
         1550-6606
Person:
      name:Platzer, Barbara
      affiliation:* Division of Gastroenterology and Nutrition, Children’s Hospital Boston, Harvard Medical School, Boston, MA 02115;
      name:Richter, Susanne
      affiliation:† Institute of Immunology, Center of Physiology, Pathophysiology and Immunology, Medical University Vienna, Vienna, Austria; and
      name:Kneidinger, Doris
      affiliation:† Institute of Immunology, Center of Physiology, Pathophysiology and Immunology, Medical University Vienna, Vienna, Austria; and
      name:Waltenberger, Darina
      affiliation:† Institute of Immunology, Center of Physiology, Pathophysiology and Immunology, Medical University Vienna, Vienna, Austria; and
      name:Woisetschläger, Maximilian
      affiliation:‡ Department of Autoimmunity and Transplantation, Novartis Institutes for Biomedical Research (NIBR), Vienna, Austria
      name:Strobl, Herbert
      affiliation:† Institute of Immunology, Center of Physiology, Pathophysiology and Immunology, Medical University Vienna, Vienna, Austria; and

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