Here's how DOI.ORG makes money* and how much!

*Please read our disclaimer before using our estimates.
Loading...

DOI . ORG {}

Detected CMS Systems:

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Doi.org Make Money
  6. Keywords
  7. Topics
  8. Schema
  9. Social Networks
  10. External Links
  11. Libraries
  12. Hosting Providers
  13. CDN Services

We began analyzing https://www.frontiersin.org/journals/cell-and-developmental-biology/articles/10.3389/fcell.2023.1252942/full, but it redirected us to https://www.frontiersin.org/journals/cell-and-developmental-biology/articles/10.3389/fcell.2023.1252942/full. The analysis below is for the second page.

Title[redir]:
Frontiers | Tanshinone IIA inhibits cardiomyocyte pyroptosis through TLR4/NF-κB p65 pathway after acute myocardial infarction
Description:
Background: Tanshinone IIA, derived from Radix Salviae Miltiorrhizae(Salvia miltiorrhiza Bunge), constitutes a significant component of this traditional Chin...

Matching Content Categories {📚}

  • Education
  • Science
  • Insurance

Content Management System {📝}

What CMS is doi.org built with?


Doi.org is powered by TYPESETTER CMS.

Traffic Estimate {📈}

What is the average monthly size of doi.org audience?

🏙️ Massive Traffic: 50M - 100M visitors per month


Based on our best estimate, this website will receive around 76,079,999 visitors per month in the current month.

check SE Ranking
check Ahrefs
check Similarweb
check Ubersuggest
check Semrush

How Does Doi.org Make Money? {💸}

We're unsure how the site profits.

Some websites aren't about earning revenue; they're built to connect communities or raise awareness. There are numerous motivations behind creating websites. This might be one of them. Doi.org has a secret sauce for making money, but we can't detect it yet.

Keywords {🔍}

tanshinone, iia, text, crossref, full, google, scholar, pyroptosis, abstract, pubmed, cell, group, cardiac, pathway, myocardial, heart, cells, figure, model, function, liu, signaling, wang, zhang, cardiomyocyte, effects, levels, apoptosis, ilβ, injury, potential, expression, tlr, tlrnfκb, china, rats, staining, nfκb, inflammatory, treatment, nlrp, caspase, activation, captopril, cardiomyocytes, med, left, analysis, therapeutic, research,

Topics {✒️}

tgf-β/pi3k/akt/enos pathway tlr4/tak1/nf-κb signaling pathway tlr4/nf-κb/pfkfb3 signal transduction tlr4/myd88/nf-κb signal pathway ampks/mtor-dependent autophagy pathway tlr4/nf-κb/pfkfb3 signaling tlr4/nf-κb pathway assumes tlr4/nf-κb p65 pathway suppressing trl4/nf-κb signaling c-type lectin receptors tlr4/nf-κb/nlrp3 pathway tlr4-nf-κb-nlrp3 pathway data availability statement tlr4/nf-κb pathway phosphorylated nf-κb p65 address hypoxic/ischemic encephalopathy mkk4/jnk2 signaling pathway activating pi3k/akt signaling nf-κb p65 activity nlrp3/caspase-1/gsdmd signaling pathway anti-nf-κb p65 tlr4/nf-κbp65 pathway daxx/mek/erk1/2 pathway impede tlr4/nlrp3-activated pyroptosis lipopolysaccharide-induced cardiac dysfunction radiation-induced cardiomyocyte injury ethics statement acute myocardial infarction /r-induced cell damage enzyme-linked immunosorbent assay myocardial ischemia/reperfusion injury myocardial ischemia-reperfusion injury mimic ischemia-reperfusion injury hepatic ischemia/reperfusion injury lps-induced cardiac dysfunction open-access article distributed pnf-κb p65 expression doxorubicin-triggered cardiac toxicity cardiac ischemia-reperfusion injury nf-κb signaling shigella flexneri-induced inflammation tukey post-hoc test nf-κb p65 promoting nlrp3-mediated pyroptosis article/supplementary material left ventricular systolic /r-induced cellular apoptosis mitigate inflammasome-induced pyroptosis nlrp3 inflammasome-induced pyroptosis 8-week post-surgery period

Schema {🗺️}

ScholarlyArticle:
      context:https://schema.org
      headline:Tanshinone IIA inhibits cardiomyocyte pyroptosis through TLR4/NF-κB p65 pathway after acute myocardial infarction
      author:
            type:Person
            name:Ruoning Chai
            affiliation:
               Department of Cardiology, China
            type:Person
            name:Zelin Ye
            affiliation:
               Department of Cardiology, China
            type:Person
            name:Wenjing Xue
            affiliation:
               Department of Cardiology, China
            type:Person
            name:Shuqing Shi
            affiliation:
               Department of Cardiology, China
            type:Person
            name:Yi Wei
            affiliation:
               Department of Cardiology, China
            type:Person
            name:Yuanhui Hu
            affiliation:
               Department of Cardiology, China
            type:Person
            name:Huaqin Wu
            affiliation:
               Department of Cardiology, China
      datePublished:2023-09-12
      dateModified:2025-07-02
      publisher:
         type:Organization
         name:Frontiers
      isPartOf:
         type:PublicationIssue
         datePublished:2023
         isPartOf:
            type:PublicationVolume
            volumeNumber:11
            isPartOf:
               type:Periodical
               name:Frontiers in Cell and Developmental Biology
      citation:
         https://doi.org/10.3389/fcell.2023.1252942
      inLanguage:en
Person:
      name:Ruoning Chai
      affiliation:
         Department of Cardiology, China
      name:Zelin Ye
      affiliation:
         Department of Cardiology, China
      name:Wenjing Xue
      affiliation:
         Department of Cardiology, China
      name:Shuqing Shi
      affiliation:
         Department of Cardiology, China
      name:Yi Wei
      affiliation:
         Department of Cardiology, China
      name:Yuanhui Hu
      affiliation:
         Department of Cardiology, China
      name:Huaqin Wu
      affiliation:
         Department of Cardiology, China
Organization:
      name:Frontiers
PublicationIssue:
      datePublished:2023
      isPartOf:
         type:PublicationVolume
         volumeNumber:11
         isPartOf:
            type:Periodical
            name:Frontiers in Cell and Developmental Biology
PublicationVolume:
      volumeNumber:11
      isPartOf:
         type:Periodical
         name:Frontiers in Cell and Developmental Biology
Periodical:
      name:Frontiers in Cell and Developmental Biology

Social Networks {👍}(7)

External Links {🔗}(301)

Libraries {📚}

  • Vue.js

Emails and Hosting {✉️}

Mail Servers:

  • mx.zoho.eu
  • mx2.zoho.eu
  • mx3.zoho.eu

Name Servers:

  • josh.ns.cloudflare.com
  • zita.ns.cloudflare.com

CDN Services {📦}

  • Cloudflare
  • Crossref

6.49s.