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DOI . ORG {}

Detected CMS Systems:

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Doi.org Make Money
  6. Keywords
  7. Topics
  8. Schema
  9. Social Networks
  10. External Links
  11. Libraries
  12. Hosting Providers
  13. CDN Services

We began analyzing https://www.frontiersin.org/journals/immunology/articles/10.3389/fimmu.2015.00639/full, but it redirected us to https://www.frontiersin.org/journals/immunology/articles/10.3389/fimmu.2015.00639/full. The analysis below is for the second page.

Title[redir]:
Frontiers | The Treg/Th17 Axis: A Dynamic Balance Regulated by the Gut Microbiome
Description:
T-helper 17 (Th17) and T-regulatory (Treg) cells are frequently found at barrier surfaces, particularly within the intestinal mucosa, where they function to ...

Matching Content Categories {πŸ“š}

  • Science
  • Telecommunications
  • Education

Content Management System {πŸ“}

What CMS is doi.org built with?


Doi.org is powered by TYPESETTER CMS.

Traffic Estimate {πŸ“ˆ}

What is the average monthly size of doi.org audience?

🌍 Impressive Traffic: 500k - 1M visitors per month


Based on our best estimate, this website will receive around 600,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

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How Does Doi.org Make Money? {πŸ’Έ}

We find it hard to spot revenue streams.

While many websites aim to make money, others are created to share knowledge or showcase creativity. People build websites for various reasons. This could be one of them. Doi.org might be plotting its profit, but the way they're doing it isn't detectable yet.

Keywords {πŸ”}

cells, text, crossref, full, google, scholar, abstract, pubmed, cell, tregs, regulatory, intestinal, treg, foxp, differentiation, gut, immunity, immunol, responses, bacteria, commensal, inflammation, receptor, function, immune, disease, tcells, induction, microbiome, tgfΞ², inflammatory, human, colitis, mice, ibd, med, tregth, tcell, generation, nature, sfb, doijimmuni, bowel, plasticity, regulation, microbial, expression, fragilis, doinature, host,

Topics {βœ’οΈ}

short-chain fatty acid tgf-beta-induced foxp3 inhibits short-chain fatty acids il-17-producing foxp3+cd4+ lymphocytes il-17-producing rorΞ³t+tΞ±Ξ² cells cells induce cd4+cd25-foxp3 il-17-producing t-helper cells open-access article distributed rorΞ³t+tΞ±Ξ² cells derived naΓ―ve cd4+ t-cells rorΞ³t+foxp3+ treg/th17 cells drive il-23-dependent inflammation upregulating g-protein receptor hypoxia-inducible factor 1Ξ± peripheral cd4+cd25- naive bacteria-producing ahr ligands relieve foxp3-mediated inhibition exhibits treg-inducing activity antigen-naΓ―ve t-cells t-bet-dependent manner antigen-specific peripheral shaping wild-type c57bl/6 mice adenosine monophosphate-activated kinase il-12p35-deficient mice t-betβˆ’/βˆ’rag2βˆ’/βˆ’ mice treg-specific myd88 deficiency preventing th17-mediated colitis de novo generation treg t-cell receptor foxp3 transcription-factor-dependent interest statement directing t-helper cells il-17-producing t-cells blocking il-6r expression attenuates bacteria-induced colitis microbial-activated dendritic cells aryl hydrocarbon receptor promotes t-cell differentiation cell contact-dependent immunosuppression proinflammatory t-cell responses lineage-specific transcription factors activated effector t-cells il-6-stimulated t-cells research search frontiers il-23r/il-22 pathway differentiated treg/effector cells colonic helios-negative tregs tgfΞ² represses il-23r segmented filamentous bacteria il-10-producing foxp3+ tregs

Schema {πŸ—ΊοΈ}

ScholarlyArticle:
      context:https://schema.org
      headline:The Treg/Th17 Axis: A Dynamic Balance Regulated by the Gut Microbiome
      author:
            type:Person
            name:Sara Omenetti
            affiliation:
               Department of Pathology, Case Western Reserve University School of Medicine, USA
            type:Person
            name:Theresa T. Pizarro
            affiliation:
               Department of Pathology, Case Western Reserve University School of Medicine, USA
      datePublished:2015-12-17
      dateModified:2025-06-30
      publisher:
         type:Organization
         name:Frontiers
      isPartOf:
         type:PublicationIssue
         datePublished:2015
         isPartOf:
            type:PublicationVolume
            volumeNumber:6
            isPartOf:
               type:Periodical
               name:Frontiers in Immunology
      citation:
         https://doi.org/10.3389/fimmu.2015.00639
      inLanguage:en
Person:
      name:Sara Omenetti
      affiliation:
         Department of Pathology, Case Western Reserve University School of Medicine, USA
      name:Theresa T. Pizarro
      affiliation:
         Department of Pathology, Case Western Reserve University School of Medicine, USA
Organization:
      name:Frontiers
PublicationIssue:
      datePublished:2015
      isPartOf:
         type:PublicationVolume
         volumeNumber:6
         isPartOf:
            type:Periodical
            name:Frontiers in Immunology
PublicationVolume:
      volumeNumber:6
      isPartOf:
         type:Periodical
         name:Frontiers in Immunology
Periodical:
      name:Frontiers in Immunology

External Links {πŸ”—}(404)

Libraries {πŸ“š}

  • Vue.js

Emails and Hosting {βœ‰οΈ}

Mail Servers:

  • mx.zoho.eu
  • mx2.zoho.eu
  • mx3.zoho.eu

Name Servers:

  • josh.ns.cloudflare.com
  • zita.ns.cloudflare.com

CDN Services {πŸ“¦}

  • Cloudflare
  • Crossref

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