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DOI . ORG {}

Detected CMS Systems:

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Doi.org Make Money
  6. Keywords
  7. Topics
  8. Schema
  9. Social Networks
  10. External Links
  11. Libraries
  12. Hosting Providers
  13. CDN Services

We began analyzing https://www.frontiersin.org/journals/cell-and-developmental-biology/articles/10.3389/fcell.2021.637162/full, but it redirected us to https://www.frontiersin.org/journals/cell-and-developmental-biology/articles/10.3389/fcell.2021.637162/full. The analysis below is for the second page.

Title[redir]:
Frontiers | Characteristics and Biomarkers of Ferroptosis
Description:
The induction and consequences of regulated cell death (RCD) are accompanied by changes in gene and protein expression, biochemical pathways, as well as cell...

Matching Content Categories {πŸ“š}

  • Telecommunications
  • Science
  • Education

Content Management System {πŸ“}

What CMS is doi.org built with?


Doi.org employs TYPESETTER CMS.

Traffic Estimate {πŸ“ˆ}

What is the average monthly size of doi.org audience?

πŸ™οΈ Massive Traffic: 50M - 100M visitors per month


Based on our best estimate, this website will receive around 76,079,999 visitors per month in the current month.

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How Does Doi.org Make Money? {πŸ’Έ}

We can't see how the site brings in money.

While many websites aim to make money, others are created to share knowledge or showcase creativity. People build websites for various reasons. This could be one of them. Doi.org might be making money, but it's not detectable how they're doing it.

Keywords {πŸ”}

ferroptosis, doi, text, crossref, full, google, scholar, pubmed, abstract, cell, death, ferroptotic, lipid, protein, kang, cells, tang, liu, peroxidation, cancer, chen, biol, iron, yang, degradation, sun, damage, dixon, gpx, autophagy, wang, res, gene, molecular, pathway, rcd, oxidative, ptgs, nat, response, erastin, dai, zhang, commun, expression, diseases, membrane, chac, glutathione, sensitivity,

Topics {βœ’οΈ}

apoptosis-inducing factor-homologous mitochondrion p62-keap1-nrf2 pathway protects ros-induced caspase-independent apoptosis dependent chaperone-mediated autophagy induces caspase-independent apoptosis high-iron diet triggers p53-independent tumor suppression p53-mediated tumour suppression autophagy-dependent cell death gfp-lc3-rfp-lc3Ξ΄g atf4-atf3-chop cascade Ξ³-glutamyl cyclotransferase activity circadian clock protects glutathione-independent ferroptosis suppressor er stress-independent ferroptosis enzyme-linked immunosorbent assay open-access article distributed tissue ischemia-reperfusion injury promote anti-tumor immunity lipid-soluble fluorescent probes atf4-chop-chac1 pathway iron-dependent enzymes present gpx4 inhibitor-induced ferroptosis ferroptosis-resistant cell state ferroptosis-induced sterile inflammation iron-triggered ros production anti-ferroptotic protein appears cystine/glutamate antiporter chaperone-mediated autophagy suppress erastin-induced ferroptosis stimulating iron export molecular signaling achieved cell–cell communication results stony brook medicine research search frontiers promoting ferritin export iron metabolism-related proteins cell type-dependent role erastin-induced ferritin degradation iron-dependent form iron overload-related diseases mir-214-mediated tfr1 dependent lipid uptake renal cell carcinoma context-dependent manner polyunsaturated fatty acids molecular regulators related oxidative damage-related rcd autophagy-dependent ferroptosis traumatic brain injury

Schema {πŸ—ΊοΈ}

ScholarlyArticle:
      context:https://schema.org
      headline:Characteristics and Biomarkers of Ferroptosis
      author:
            type:Person
            name:Xin Chen
            affiliation:
               Guangzhou Municipal and Guangdong Provincial Key Laboratory of Protein Modification and Degradation, The Third Affiliated Hospital, School of Basic Medical Sciences, Guangzhou Medical University, China
               Affiliated Cancer Hospital & Institute of Guangzhou Medical University, China
               Department of Surgery, UT Southwestern Medical Center, Dallas, United States
            type:Person
            name:Paul B. Comish
            affiliation:
               Department of Surgery, UT Southwestern Medical Center, Dallas, United States
            type:Person
            name:Daolin Tang
            affiliation:
               Guangzhou Municipal and Guangdong Provincial Key Laboratory of Protein Modification and Degradation, The Third Affiliated Hospital, School of Basic Medical Sciences, Guangzhou Medical University, China
               Department of Surgery, UT Southwestern Medical Center, Dallas, United States
            type:Person
            name:Rui Kang
            affiliation:
               Department of Surgery, UT Southwestern Medical Center, Dallas, United States
      datePublished:2021-01-21
      dateModified:2025-07-03
      publisher:
         type:Organization
         name:Frontiers
      isPartOf:
         type:PublicationIssue
         datePublished:2021
         isPartOf:
            type:PublicationVolume
            volumeNumber:9
            isPartOf:
               type:Periodical
               name:Frontiers in Cell and Developmental Biology
      citation:
         https://doi.org/10.3389/fcell.2021.637162
      inLanguage:en
Person:
      name:Xin Chen
      affiliation:
         Guangzhou Municipal and Guangdong Provincial Key Laboratory of Protein Modification and Degradation, The Third Affiliated Hospital, School of Basic Medical Sciences, Guangzhou Medical University, China
         Affiliated Cancer Hospital & Institute of Guangzhou Medical University, China
         Department of Surgery, UT Southwestern Medical Center, Dallas, United States
      name:Paul B. Comish
      affiliation:
         Department of Surgery, UT Southwestern Medical Center, Dallas, United States
      name:Daolin Tang
      affiliation:
         Guangzhou Municipal and Guangdong Provincial Key Laboratory of Protein Modification and Degradation, The Third Affiliated Hospital, School of Basic Medical Sciences, Guangzhou Medical University, China
         Department of Surgery, UT Southwestern Medical Center, Dallas, United States
      name:Rui Kang
      affiliation:
         Department of Surgery, UT Southwestern Medical Center, Dallas, United States
Organization:
      name:Frontiers
PublicationIssue:
      datePublished:2021
      isPartOf:
         type:PublicationVolume
         volumeNumber:9
         isPartOf:
            type:Periodical
            name:Frontiers in Cell and Developmental Biology
PublicationVolume:
      volumeNumber:9
      isPartOf:
         type:Periodical
         name:Frontiers in Cell and Developmental Biology
Periodical:
      name:Frontiers in Cell and Developmental Biology

Social Networks {πŸ‘}(7)

External Links {πŸ”—}(389)

Libraries {πŸ“š}

  • Vue.js

Emails and Hosting {βœ‰οΈ}

Mail Servers:

  • mx.zoho.eu
  • mx2.zoho.eu
  • mx3.zoho.eu

Name Servers:

  • josh.ns.cloudflare.com
  • zita.ns.cloudflare.com

CDN Services {πŸ“¦}

  • Cloudflare
  • Crossref

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