
DOI . ORG {
}
Title[redir]:
USP5 and Uterine Corpus Endometrial Carcinoma Cell Growth | CMAR
Description:
USP5 functioned as an oncogene in UCEC, which provided new insights into pathogenesis of UCEC and a promising molecular target for UCEC diagnosis and therapy
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Keywords {π}
usp, cells, cell, cancer, ucec, hecb, figure, kle, assay, proliferation, overexpression, migration, promotes, cycle, apoptosis, shown, analysis, western, level, control, endometrial, assessed, protein, mtor, carcinoma, knockdown, blot, results, progression, growth, deubiquitinating, sirnas, expression, patients, downregulation, pathway, compared, press, medical, found, tcga, data, ubiquitin, sirna, analyzed, dove, mtorebp, activation, performed, wound,
Topics {βοΈ}
pi3k/akt signaling pathway wnt/beta-catenin signaling including fast-track processing alleviating p14arf-p53 signaling mouse anti-rabbit igg rabbit anti-mouse igg regulating jmjd3/clu axis dove medical press activating mtor/4ebp1 pathway mediate mtor/4ebp1 activation wnt/Ξ²-catenin pathway specific author g0/g1-phase transfected 2-tailed unpaired t-test ubiquitin-specific protease 5 g0/g1-phase showed scientific research project manuscript login favored authors promotes hcc progression significantly impeded accompanied cell cycle transition cell growth curves promote cell division ucec cell survival cell proliferation inhibition promote ucec proliferation jmjd3/clu axis observe cell migration g0/g1-phase usp5 promotes tumorigenesis impair migration ability cell proliferation curves cell cycle arrest cell cycle distributions cell cycle stages ucec cell proliferation beta-catenin protein mtor/4ebp1 pathway facilitate tumor growth enhanced cell viabilities offer real benefits epithelial neoplasm originating deubiquitinating enzyme usp5 free ubiquitin pool usp5 downregulation suppressed promote tumor progression ubiquitin proteasome pathway copy number variations mol med rep
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