DOI . ORG {
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Detected CMS Systems:
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- Hubspot (1 occurrences)
We began analyzing https://journals.plos.org/plospathogens/article?id=10.1371/journal.ppat.1007796, but it redirected us to https://journals.plos.org/plospathogens/article?id=10.1371/journal.ppat.1007796. The analysis below is for the second page.
Title[redir]:
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Description:
Author summary Epstein–Barr virus (EBV) is a ubiquitous oncogenic virus that contributes to approximately 2% of all cancers by modulating a myriad of host cell activities. M6A is the most abundant RNA modification which is important for infection with HIV-1, HCV, Zika virus, KSHV and SV40 virus. The role of m6A modification and the associated enzymes in EBV infection and related oncogenic activities has not been explored. We investigated the role of m6A modification on EBV-infection and examined its effects on the proliferative activities of the virus which contributes to cancer. We showed that the m6A methyltransferase METTL14 is an important factor in EBV-induced oncogenesis. METTL14 was dramatically increased in EBV latently infected cells. Knock-down of METTL14 led to decreased latent viral gene expression. The essential EBV latent antigen EBNA3C was up-regulated by METTL14-mediated m6A modification, and its expression led to a feedback loop in up-regulation of METTL14 transcription, and the stability of its protein. We now provide a more comprehensive understanding of EBV and the host cell RNA processing machinery critical for regulation of EBV activities to maintain latent infection and its oncogenic properties.
Title[redir]:
No title found...
Description:
Author summary Epstein–Barr virus (EBV) is a ubiquitous oncogenic virus that contributes to approximately 2% of all cancers by modulating a myriad of host cell activities. M6A is the most abundant RNA modification which is important for infection with HIV-1, HCV, Zika virus, KSHV and SV40 virus. The role of m6A modification and the associated enzymes in EBV infection and related oncogenic activities has not been explored. We investigated the role of m6A modification on EBV-infection and examined its effects on the proliferative activities of the virus which contributes to cancer. We showed that the m6A methyltransferase METTL14 is an important factor in EBV-induced oncogenesis. METTL14 was dramatically increased in EBV latently infected cells. Knock-down of METTL14 led to decreased latent viral gene expression. The essential EBV latent antigen EBNA3C was up-regulated by METTL14-mediated m6A modification, and its expression led to a feedback loop in up-regulation of METTL14 transcription, and the stability of its protein. We now provide a more comprehensive understanding of EBV and the host cell RNA processing machinery critical for regulation of EBV activities to maintain latent infection and its oncogenic properties.
Matching Content Categories {📚}
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- Video & Online Content
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What CMS is doi.org built with?
Doi.org is built with WORDPRESS.
But there are also traces of other content systems on the page (hubspot).
Traffic Estimate {📈}
What is the average monthly size of doi.org audience?
🏙️ Massive Traffic: 50M - 100M visitors per month
Based on our best estimate, this website will receive around 91,705,781 visitors per month in the current month.
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We can't see how the site brings in money.
Earning money isn't the goal of every website; some are designed to offer support or promote social causes. People have different reasons for creating websites. This might be one such reason. Doi.org might be cashing in, but we can't detect the method they're using.
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Keywords {🔍}
mettl, cells, ebnac, fig, expression, ebv, modification, latent, cell, lytic, results, rna, lcls, transcripts, article, viral, view, google, scholar, levels, infection, pmid, pubmedncbi, lcl, gene, virus, knockdown, stability, protein, bjab, ythdf, antibodies, genes, mrna, showed, western, role, growth, transfected, performed, transcription, akata, blot, control, ebvpositive, alkbh, detected, experiments, million, lysed,
Topics {✒️}
rna biology 11 plos pathog 15 plos pathog 14 plos pathog 9 cookies plos diseases linked health dual-luciferase reporter assay epstein-barr virus mettl3-mettl14-wtap-mediated m6a modification ebv-mediated cell transformation elute-frag-prime stage single-nucleotide-resolution mapping quantitative real-time pcr tumor virus-mediated oncogenesis transcription repressor rbp-jk 293t-bac-ebv cells uninduced original author human b-cell lines ribo-rna deleted mrna real-time pcr assays ebv epitranscriptome reprogramming hek293t-bac-ebv cells histone acetyltransferase activity ebv-positive tissues compared cfar immunology core myc-tagged full length 293t-bac-ebv cells ebv-positive b-cells final rna-seq library drive ebv-mediated tumorigenesis supporting information files analyze cfse-labeled cells full-blown lytic reactivation viral-mediated transformation methyl-specific binding proteins rna n6-methyladenosine methylation methyltransferase complex composed xeno-transplant model proto-oncogene c-myc full-length ebna3c protein mettl14-mediated m6a modification wild-type mettl14 promoter myc-tagged specific regions scfskp2 complex targeted viral-encoded oncoprotein lymphoblastoid cell lines methyltransferase complex required end repair process promoting cell growth
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