DOI . ORG {
}
Detected CMS Systems:
- Wordpress (1 occurrences)
- Apache Lenya (1 occurrences)
- Hubspot (1 occurrences)
We began analyzing https://journals.plos.org/plospathogens/article?id=10.1371/journal.ppat.1004077, but it redirected us to https://journals.plos.org/plospathogens/article?id=10.1371/journal.ppat.1004077. The analysis below is for the second page.
Title[redir]:
No title found...
Description:
Author Summary Several highly pathogenic viruses encode small transmembrane proteins with ion-conduction properties named viroporins. Viroporins are generally involved in virus production and maturation processes, which many times are achieved by altering the ion homeostasis of cell organelles. Cells have evolved mechanisms to sense these imbalances in ion concentrations as a danger signal, and consequently trigger the innate immune system. Recently, it has been demonstrated that viroporins are inducers of cytosolic macromolecular complexes named inflammasomes that trigger the activation of key inflammatory cytokines such as IL-1Ξ². The repercussions of this system in viral pathogenesis or disease outcome are currently being explored. SARS-CoV infection induces an uncontrolled inflammatory response leading to pulmonary damage, edema accumulation, severe hypoxemia and eventually death. In this study, we report that SARS-CoV E protein ion channel activity is a determinant of virulence, as the elimination of this function attenuated the virus, reducing the harmful inflammatory cytokine burst produced after infection, in which inflammasome activation plays a critical role. This led to less pulmonary damage and to disease resolution. These novel findings may be of relevance for other viral infections and can possibly be translated in order to find therapies for their associated diseases.
Title[redir]:
No title found...
Description:
Author Summary Several highly pathogenic viruses encode small transmembrane proteins with ion-conduction properties named viroporins. Viroporins are generally involved in virus production and maturation processes, which many times are achieved by altering the ion homeostasis of cell organelles. Cells have evolved mechanisms to sense these imbalances in ion concentrations as a danger signal, and consequently trigger the innate immune system. Recently, it has been demonstrated that viroporins are inducers of cytosolic macromolecular complexes named inflammasomes that trigger the activation of key inflammatory cytokines such as IL-1Ξ². The repercussions of this system in viral pathogenesis or disease outcome are currently being explored. SARS-CoV infection induces an uncontrolled inflammatory response leading to pulmonary damage, edema accumulation, severe hypoxemia and eventually death. In this study, we report that SARS-CoV E protein ion channel activity is a determinant of virulence, as the elimination of this function attenuated the virus, reducing the harmful inflammatory cytokine burst produced after infection, in which inflammasome activation plays a critical role. This led to less pulmonary damage and to disease resolution. These novel findings may be of relevance for other viral infections and can possibly be translated in order to find therapies for their associated diseases.
Matching Content Categories {π}
- Science
- Health & Fitness
- Sports
Content Management System {π}
What CMS is doi.org built with?
Doi.org operates using WORDPRESS.
But there are also traces of other content systems on the page (Apache Lenya, 1hubspot).
Traffic Estimate {π}
What is the average monthly size of doi.org audience?
ποΈ Massive Traffic: 50M - 100M visitors per month
Based on our best estimate, this website will receive around 76,504,851 visitors per month in the current month.
check SE Ranking
check Ahrefs
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How Does Doi.org Make Money? {πΈ}
We see no obvious way the site makes money.
Some websites aren't about earning revenue; they're built to connect communities or raise awareness. There are numerous motivations behind creating websites. This might be one of them. Doi.org might be plotting its profit, but the way they're doing it isn't detectable yet.
Wordpress Themes and Plugins {π¨}
What WordPress theme does this site use?
It is strange but we were not able to detect any theme on the page.
What WordPress plugins does this website use?
It is strange but we were not able to detect any plugins on the page.
Keywords {π}
protein, activity, virus, mice, view, article, viruses, google, scholar, sarscov, ion, infected, mutant, cells, infection, viral, coronavirus, lung, fig, lacking, channel, gene, mutations, respiratory, cell, lungs, edema, transmembrane, production, dpi, severe, plos, acute, ilΞ², domain, virol, image, virulence, original, levels, syndrome, disease, animals, mouse, parental, conductivity, growth, displaying, accumulation, rna,
Topics {βοΈ}
public health measures open-access article distributed animal health research reverse genetics ratio pro-il-1Ξ²/beta-actin relative contributed reagents/materials/analysis tools long-alkyl-chain iminosugar derivatives systems plos pathog 10 plos pathog 7 plos pathog 5 plos pathog 8 plos pathog 6 plos pathog 9 plos pathog 3 plos pathog 4 cookies plos increasing pro-il-1Ξ² transcription jlnt le mld plos engineered rsars-cov-e-n15d health rsars-cov-e-v25f t30i protein ion channels rsars-cov-e-n15a viruses recombinant mouse-adapted sars-covs mouse-adapted sars-coronavirus pro-il-1Ξ² mrna found rsars-cov-e-v25f l37r nf-kappab mediated inflammation rsars-cov-e-v25f l27s inflammatory cytokines il-1Ξ² sars-cov-ma15 strain highly pathogenic sars-cov rsars-cov-eicβ evolved variants epithelial sodium channels highly pathogenic mers-cov severe respiratory disease fundacion caixa castello-bancaixa larger side chain vertebrate pathogens rabbit anti goat small envelope protein pro-il-1Ξ² mrna transmissible gastroenteritis coronavirus mouse-adapted virus lacking host-specific effect mediated mouse adapted sars-cov mouse-adapted sars-cov sars-cov infection induces
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