DOI . ORG {
}
Detected CMS Systems:
- Wordpress (1 occurrences)
- Hubspot (1 occurrences)
We began analyzing https://journals.plos.org/plosbiology/article?id=10.1371/journal.pbio.0050172, but it redirected us to https://journals.plos.org/plosbiology/article?id=10.1371/journal.pbio.0050172. The analysis below is for the second page.
Title[redir]:
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Description:
Mutations in the gene that codes for PINK1 cause a common form of Parkinson disease. Here the authors show that PINK1 phosphorylates TRAP1, which suppresses apoptotic release of cytochrome c from mitochondria.
Title[redir]:
No title found...
Description:
Mutations in the gene that codes for PINK1 cause a common form of Parkinson disease. Here the authors show that PINK1 phosphorylates TRAP1, which suppresses apoptotic release of cytochrome c from mitochondria.
Matching Content Categories {📚}
- Science
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Content Management System {📝}
What CMS is doi.org built with?
Doi.org employs WORDPRESS.
But there are also traces of other content systems on the page (hubspot).
Traffic Estimate {📈}
What is the average monthly size of doi.org audience?
🌍 Impressive Traffic: 500k - 1M visitors per month
Based on our best estimate, this website will receive around 600,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.
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How Does Doi.org Make Money? {💸}
We can't tell how the site generates income.
Many websites are intended to earn money, but some serve to share ideas or build connections. Websites exist for all kinds of purposes. This might be one of them. Doi.org has a secret sauce for making money, but we can't detect it yet.
Wordpress Themes and Plugins {🎨}
What WordPress theme does this site use?
It is strange but we were not able to detect any theme on the page.
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Keywords {🔍}
pink, trap, cells, cytochrome, figure, cell, mitochondrial, mutations, article, wildtype, view, kinase, google, scholar, protein, phosphorylation, mitochondria, release, sirna, mutant, transfected, oxidativestressinduced, apoptosis, control, data, activity, immunoblotting, mutation, effect, vivo, antibody, disease, death, pdlinked, plos, endogenous, survival, experiments, analysis, oxidative, function, antibodies, proteins, molecular, vitro, significant, sem, image, significantly, independent,
Topics {✒️}
small interfering rna health grants ns050650 c-terminally flag-tagged wild-type plos biol 5 cookies plos affinity-purified flag-tagged wild-type open-access article distributed flag-tagged pink1-protein complexes prevent oxidative-stress-induced apoptosis medicine genetics oxidative-stress-induced cell death flag-tagged wild-type oxidative-stress-induced trap1 phosphorylation parkinson disease-related mutations wild-type pink1-transfected cells inhibit oxidative-stress-induced cytochrome flag-tagged pink1 protein alzheimer diseases pink1-linked early-onset pd oxidative-stress-induced apoptosis immunoprecipitation experiments failed mitochondrial serine/threonine kinase oxidative-stress-induced phosphorylation exogenous wild-type pink1 h2o2-treated vector-transfected controls snap-25-interacting protein implicated c-terminal truncation hereditary early-onset parkinson' post-transfection dopaminergic cells sh-sy5y mitochondrial complex diseases original author sirna transfection altered alpha-synuclein metabolism overexpressing wild-type pink1 wild-type pink1 resulted wild-type pink1 caused hsp90-related protein trap1 oxidative-stress-induced levels h2o2-induced cell death oxidative-stress-induced cytochrome identify pink1-interacting proteins endogenous wild-type pink1 vector-transfected pc12 cells polyclonal anti-pink1 antibody anti-apoptotic signaling pathway accelerated h2o2-induced cytochrome recombinant wild-type
Questions {❓}
- (2006) Clinical spectrum of homozygous and heterozygous PINK1 mutations in a large German family with Parkinson disease: Role of a single hit?
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