DOI . ORG {
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We began analyzing https://journals.plos.org/plosgenetics/article?id=10.1371/journal.pgen.1007802, but it redirected us to https://journals.plos.org/plosgenetics/article?id=10.1371/journal.pgen.1007802. The analysis below is for the second page.
Title[redir]:
No title found...
Description:
Author summary Breast cancer is the most common cancer in women worldwide. The molecular mechanisms underlying the disease have been extensively studied, leading to dramatic improvements in diagnostic and prognostic approaches. Despite the overall improvements in survival rate, numerous cases of death by breast cancer are still reported per year, alerting us about the potential gap of knowledge in cancer molecular biology era. The emerging advances in new generation sequencing techniques have revealed that the majority of genome is transcribed into non-protein coding RNAs or ncRNAs, including thousands of long ncRNAs (lncRNAs) of unknown function. Natural antisense RNAs (NATs) constitute a group of lncRNAs that are transcribed in the opposite direction to a sense protein-coding or non-coding gene with partial or complete complementarity. In this manuscript, we investigate the role of NATs in breast cancer progression, focusing on the role of PDCD4-AS1, a NAT expressed from the established tumor suppressor PDCD4 gene locus. We observe that both PDCD4-AS1 and PDCD4 display concordant expression in breast cancer cell lines and patients. In mammary epithelial cells, PDCD4-AS1 promotes the stability of PDCD4 mRNA. PDCD4-AS1 by forming RNA duplex with PDCD4 RNA prevents the interaction between PDCD4 RNA and RNA decay factors in the nucleus.
Title[redir]:
No title found...
Description:
Author summary Breast cancer is the most common cancer in women worldwide. The molecular mechanisms underlying the disease have been extensively studied, leading to dramatic improvements in diagnostic and prognostic approaches. Despite the overall improvements in survival rate, numerous cases of death by breast cancer are still reported per year, alerting us about the potential gap of knowledge in cancer molecular biology era. The emerging advances in new generation sequencing techniques have revealed that the majority of genome is transcribed into non-protein coding RNAs or ncRNAs, including thousands of long ncRNAs (lncRNAs) of unknown function. Natural antisense RNAs (NATs) constitute a group of lncRNAs that are transcribed in the opposite direction to a sense protein-coding or non-coding gene with partial or complete complementarity. In this manuscript, we investigate the role of NATs in breast cancer progression, focusing on the role of PDCD4-AS1, a NAT expressed from the established tumor suppressor PDCD4 gene locus. We observe that both PDCD4-AS1 and PDCD4 display concordant expression in breast cancer cell lines and patients. In mammary epithelial cells, PDCD4-AS1 promotes the stability of PDCD4 mRNA. PDCD4-AS1 by forming RNA duplex with PDCD4 RNA prevents the interaction between PDCD4 RNA and RNA decay factors in the nucleus.
Matching Content Categories {📚}
- Science
- Education
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Content Management System {📝}
What CMS is doi.org built with?
Doi.org utilizes WORDPRESS.
But there are also traces of other content systems on the page (hubspot).
Traffic Estimate {📈}
What is the average monthly size of doi.org audience?
🏙️ Massive Traffic: 50M - 100M visitors per month
Based on our best estimate, this website will receive around 76,079,999 visitors per month in the current month.
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How Does Doi.org Make Money? {💸}
We can't figure out the monetization strategy.
Websites don't always need to be profitable; some serve as platforms for education or personal expression. Websites can serve multiple purposes. And this might be one of them. Doi.org has a secret sauce for making money, but we can't detect it yet.
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Keywords {🔍}
pdcd, cells, pdcdas, rna, mrna, expression, article, view, google, scholar, fig, cancer, cell, pmid, pubmedncbi, breast, gene, levels, control, stability, genes, rtqpcr, progression, hur, lncrnas, sense, protein, tumor, pdcdasdepleted, nats, antisense, human, suppressor, data, nat, patients, rnas, significant, regulates, observed, depletion, transcripts, showed, migration, assay, long, coding, natural, lncrna, noncoding,
Topics {✒️}
health [gm088252 plos genet 14 cookies plos plos decay-promoting rna-binding proteins diseases medicine small duplex pdcd4/pdcd4-as1 gene locus pdcd4-as1/pdcd4 gene locus modulating post-transcriptional processing double-stranded/duplex rnas prevent rnase-mediated degradation selected pdcd4-as1/pdcd4 pair pdcd4-as1 shrna-treated cells pdcd4-as1-depleted m1 cells rna-editing enzymes adar1 rna-binding proteins pdcd4-as1-depleted tnbc cells shrna lentivirus-mediated transduction nat/protein-coding gene pairs streptavidin-mediated rna pull breast cancer biology incubated biotin-labeled pdcd4-as1 long-term cell proliferation au-rich sequence element hypoxia-inducible factor 1alpha pdcd4-as1 negatively regulates protein-coding rna genes pdcd4-overexpressing m1 cells tumor biology pdcd4-as1-mediated regulation stabilizing rna-binding protein tumor suppressor gene showed increased population mutant pdcd4-as1 constructs t-cell intracellular antigen-1 tumor suppressor genes patient rna-seq data pdcd4-as1-depleted cells pdcd4-as1 depleted cells sense protein-coding genes pdcd4-as1 acts upstream pdcd4-as1 acted upstream single pdcd4-as1 rna pdcd4-as1 rna level differentiated low-grade carcinomas robust transcriptome-wide discovery pdcd4-depleted m1 cells pdcd4-as1 positively regulates
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