Here's how DOI.ORG makes money* and how much!

*Please read our disclaimer before using our estimates.
Loading...

DOI . ORG {}

Detected CMS Systems:

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Doi.org Make Money
  6. Wordpress Themes And Plugins
  7. Keywords
  8. Topics
  9. Questions
  10. Social Networks
  11. External Links
  12. Analytics And Tracking
  13. Libraries
  14. Hosting Providers
  15. CDN Services

We began analyzing https://journals.plos.org/plosgenetics/article?id=10.1371/journal.pgen.1007037, but it redirected us to https://journals.plos.org/plosgenetics/article?id=10.1371/journal.pgen.1007037. The analysis below is for the second page.

Title[redir]:
No title found...
Description:
Author summary Programmed cell death (PCD) has crucial roles in development and immunity in multicellular organisms. In plants, rapid PCD induction, so-called hypersensitive response (HR) cell death, can be triggered as a part of immune system, and plays an important role in restricting pathogen growth. Despite its importance, cell death induction can backfire on plants because of the diversified infection strategies of plant pathogens. It is therefore assumed that plants have mechanisms by which they are able to minimize PCD induction during plant-pathogen interactions. However, their existence and biological significance are not clear yet. Here, we demonstrate that PSIG1, which has the GYF domain that is highly conserved among diverse eukaryotic species, restricts cell death induction during pathogen invasions. Importantly, psig1 mutants do not display autoimmune phenotypes, and are more susceptible to the virulent bacterial pathogen. Our findings suggest that the restriction of cell death can have benefits for plants to defend themselves against hemi-biotrophic bacterial pathogen infections. We further provide evidence suggesting a mechanism by which PSIG1 may contain cell death by regulating the RNA metabolism machinery.

Matching Content Categories {πŸ“š}

  • Science
  • Education
  • Telecommunications

Content Management System {πŸ“}

What CMS is doi.org built with?


Doi.org uses WORDPRESS. But there are also traces of other content systems on the page (hubspot).

Traffic Estimate {πŸ“ˆ}

What is the average monthly size of doi.org audience?

🌍 Impressive Traffic: 500k - 1M visitors per month


Based on our best estimate, this website will receive around 600,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

check SE Ranking
check Ahrefs
check Similarweb
check Ubersuggest
check Semrush

How Does Doi.org Make Money? {πŸ’Έ}

We don’t know how the website earns money.

While profit motivates many websites, others exist to inspire, entertain, or provide valuable resources. Websites have a variety of goals. And this might be one of them. Doi.org might be earning cash quietly, but we haven't detected the monetization method.

Wordpress Themes and Plugins {🎨}

What WordPress theme does this site use?

It is strange but we were not able to detect any theme on the page.

What WordPress plugins does this website use?

It is strange but we were not able to detect any plugins on the page.

Keywords {πŸ”}

psig, cell, death, fig, plants, article, view, google, scholar, plant, mutants, pmid, pubmedncbi, pto, gyf, mutant, smg, blue, domain, induction, arabidopsis, cells, leaves, trypan, inoculation, inoculated, data, pathogen, determined, infection, phenotype, proteins, stained, dead, bacterial, test, days, pathogens, avrrps, hpa, shown, growth, enhanced, gene, spray, cfu, bak, expression, statistical, differences,

Topics {βœ’οΈ}

proline-rich sequence proline-proline-glycine-phenylalanine tir-nb-lrr-type receptor proteins plos genet 13 plos genet cookies plos plos flagellin-induced complex alternative nb-lrr-type receptors brassinosteroid-independent cell-death pathways nonsense-mediated rna decay hemi-biotrophic bacterial pathogens rps4-triggered cell death recognizes proline-rich sequences hemi-biotrophic bacterial pathogen temperature-dependent cell death rrs1/rps4-triggered eti [49] pathogen-triggered cell death hemi-biotrophic pathogens switch eti-triggered cell death t-dna insertion sites proline-rich sequence recognition full-length psig1 interacted pseudomonas syringae pv avrrpm1-triggered cell death agrobacterium carrying pjl3-p19 avrrps4-triggered cell death early pamp-responsive phosphoprotein plant cell-death control c-terminally truncated psig1 t-dna insertion site 8-mm-diameter cork borer psig1-dependent cell death early pamp-responsive protein original author cell death-triggering mechanisms avirulent bacterial pathogens stop codon-mutated psig1pro pseudomonas syringae effector lactophenol-trypan blue solution reactive oxygen intermediates downy mildew fungus semi-quantitative rt-pcr hemi-biotrophic pathogens hemi-biotrophic pathogens [2] ile-type gyf domains agonist-dependent gain control pentr4 dual-selection vector label-free quantitative proteomics conserved Ξ²-hairpin recognition flg22-induced stomatal closure

Questions {❓}

  • To die or not to die?

External Links {πŸ”—}(382)

Analytics and Tracking {πŸ“Š}

  • Facebook Pixel
  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager
  • Google Universal Analytics
  • HubSpot

Libraries {πŸ“š}

  • Foundation
  • jQuery
  • Leaflet
  • Lightbox
  • Modernizr
  • Moment.js
  • Underscore.js
  • Video.js
  • Vue.js
  • Zoom.js

Emails and Hosting {βœ‰οΈ}

Mail Servers:

  • mx.zoho.eu
  • mx2.zoho.eu
  • mx3.zoho.eu

Name Servers:

  • josh.ns.cloudflare.com
  • zita.ns.cloudflare.com

CDN Services {πŸ“¦}

  • Cloudflare
  • Crossref

7.61s.