Here's how DOI.ORG makes money* and how much!

*Please read our disclaimer before using our estimates.
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DOI . ORG {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Doi.org Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. External Links
  10. Analytics And Tracking
  11. Libraries
  12. Hosting Providers

We began analyzing https://www.jci.org/articles/view/6870, but it redirected us to https://www.jci.org/articles/view/6870. The analysis below is for the second page.

Title[redir]:
JCI - Matrix metalloproteinases in angiogenesis: a moving target for therapeutic intervention
Description:
No description found...

Matching Content Categories {📚}

  • Education
  • Science
  • Telecommunications

Content Management System {📝}

What CMS is doi.org built with?

Custom-built

No common CMS systems were detected on Doi.org, but we identified it was custom coded using Ruby on Rails (Ruby).

Traffic Estimate {📈}

What is the average monthly size of doi.org audience?

🌟 Strong Traffic: 100k - 200k visitors per month


Based on our best estimate, this website will receive around 100,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

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How Does Doi.org Make Money? {💸}

We're unsure how the site profits.

Not all websites focus on profit; some are designed to educate, connect people, or share useful tools. People create websites for numerous reasons. And this could be one such example. Doi.org might be cashing in, but we can't detect the method they're using.

Keywords {🔍}

mmp, endothelial, cell, matrix, angiogenesis, binding, cells, αvβ, timp, activity, pex, view, mtmmp, mmps, extracellular, angiogenic, activation, article, pubmed, google, scholar, response, prommp, domain, crossref, role, inhibitors, formation, growth, type, mechanism, collagen, protease, specific, surface, inhibitor, involved, membrane, interaction, metalloproteinases, endogenous, studies, invasion, production, expression, receptor, angiostatin, metalloproteinase, biol, proliferation,

Topics {✒️}

pro-mmp-2-timp-2-mt-1-mmp ternary complex timp-2–free mt-1-mmp molecule mt-1-mmp/timp-2 complex localizes clinical investigation issn degrade endothelial-derived perlecan nf-κb–dependent mechanism cell-matrix adhesive interactions videos conversations endothelial cell-matrix interactions local mt-1-mmp concentration exogenous pro-mmp-2 induces gelatinase a-deficient mice 35-kda cooh-terminal fragment cell-extracellular matrix interactions fibrin-rich provisional matrix matrix metalloproteinases mt1-mmp αvβ3-bound mmp-2 interact alters signal transduction c-terminal pex domain conferred fibrin-invasive potential growth plate vascularization microvascular endothelial cells pro-mmp-2 involves timp-2 facilitate pro-mmp-2 activation endothelial cell survival mmp-2–mediated matrix proteolysis bladed propeller composed initiating signaling pathways mt-1-mmp–dependent binding mt-1-mmp–mediated activation endothelial cell functions angiostatin-converting enzyme activities enhanced cellular invasion modulate cell growth interstitial matrix forms endothelial cell invasion review series αvβ3-bound mmp-2 inhibited human matrix metalloproteinase-2 enhanced cellular production cell surface–bound mmp-2 cell-surface localization endothelial sprout invasion lewis lung carcinoma free mt-1-mmp unidentified angiogenic factor endothelial cell responses endothelial cell responding growth factors sequestered influence mmp production

Questions {❓}

  • , PEX inhibitors) present a safer strategy for targeting angiogenesis?
  • Are other mechanisms of MMP activation operative in endothelial cells in contact with basement membrane and/or provisional matrix?
  • But how do endothelial cells activate pro-MMP-2?
  • Can we make highly selective synthetic MMP inhibitors that eliminate potential side effects?
  • Does MMP activity, known to be active in generating angiogenesis inhibitors such as angiostatin, mediate this shift in endothelial cell phenotype?
  • Does TIMP-2 interaction with the PEX domain of MMP-2 compete for binding to the αvβ3 receptor?
  • Is MMP-2 binding to the αvβ3 receptor the mechanism that alters signal transduction and/or endothelial cell survival and proliferation?
  • Need help?
  • The question remains, how does the MMP-2 that binds to αvβ3 become activated?
  • What is the mechanism for generation of endogenous PEX, and how is this process initiated or controlled?
  • What is the role of MMPs in generating angiogenesis inhibitors during endothelial sprout formation?

External Links {🔗}(161)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Tag Manager
  • Google Universal Analytics

Libraries {📚}

  • Foundation
  • Modernizr
  • Video.js

Emails and Hosting {✉️}

Mail Servers:

  • mx.zoho.eu
  • mx2.zoho.eu
  • mx3.zoho.eu

Name Servers:

  • josh.ns.cloudflare.com
  • zita.ns.cloudflare.com
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