Here's how DOI.ORG makes money* and how much!

*Please read our disclaimer before using our estimates.
Loading...

DOI . ORG {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Doi.org Make Money
  6. Keywords
  7. Topics
  8. Social Networks
  9. External Links
  10. Analytics And Tracking
  11. Libraries
  12. Hosting Providers
  13. CDN Services

We began analyzing https://journals.physiology.org/doi/10.1152/physrev.00040.2007, but it redirected us to https://journals.physiology.org/doi/10.1152/physrev.00040.2007. The analysis below is for the second page.

Title[redir]:
Interpreting Neonatal Lethal Phenotypes in Mouse Mutants: Insights Into Gene Function and Human Diseases | Physiological Reviews | American Physiological Society
Description:
The mouse represents the model of choice to study the biological function of mammalian genes through mutation of its genome. However, the biggest challenge of mouse geneticists remains the phenotypic analysis of mouse mutants. A survey of mouse mutant databases reveals a surprisingly high number of gene mutations leading to neonatal death. These genetically modified mouse mutants have been instrumental in elucidating gene function and have become important models of congenital human diseases. The main complication when phenotyping mutant mice dying during the neonatal period is the large spectrum of physiological systems whose defects can challenge neonatal survival. Here, we present a comprehensive review of gene mutations leading to neonatal lethality and discuss the impact of these mutations on the major physiological processes critical to mouse newborn survival: parturition, breathing, suckling, and homeostasis. Selected examples of mouse mutants are highlighted to illustrate how the precise identification of the timing and cause of death associated with these physiological processes allows for a more profound understanding of the underlying cellular and molecular defects. This review provides a guide for the analysis of neonatal lethal phenotypes in mutant mice that will be helpful for dissecting out the function of specific genes during mouse development.

Matching Content Categories {๐Ÿ“š}

  • Science
  • Games
  • Family & Parenting

Content Management System {๐Ÿ“}

What CMS is doi.org built with?

Custom-built

No common CMS systems were detected on Doi.org, and no known web development framework was identified.

Traffic Estimate {๐Ÿ“ˆ}

What is the average monthly size of doi.org audience?

๐ŸŒŸ Strong Traffic: 100k - 200k visitors per month


Based on our best estimate, this website will receive around 100,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

check SE Ranking
check Ahrefs
check Similarweb
check Ubersuggest
check Semrush

How Does Doi.org Make Money? {๐Ÿ’ธ}

We can't figure out the monetization strategy.

Not all websites are made for profit; some exist to inform or educate users. Or any other reason why people make websites. And this might be the case. Doi.org could be secretly minting cash, but we can't detect the process.

Keywords {๐Ÿ”}

mice, neonatal, mutant, defects, birth, mutants, gene, death, respiratory, mouse, survival, fig, development, abnormal, function, lung, genes, analysis, normal, modified, suckling, lethal, lungs, newborns, encodes, physiological, newborn, copyright, breathing, absence, revealed, panel, failure, leads, phenotypes, mutations, due, permission, severe, lethality, examples, results, kidney, important, knockout, essential, involved, deletion, distress, skeletal,

Topics {โœ’๏ธ}

kinase-deficient pdgf ฮฒ-receptor parathyroid hormone-related peptide engineered zinc-finger nucleases marc saba-el-leil inspiration-related activity recorded odorant-evoked excitatory response platelet-derived growth factor phosphatidylinositol-4-phosphate 5-kinase essential aps website aps member massive intra-abdominal hemorrhage nmda receptor ฮต-subunit cell-autonomous muscular defects journals quick search high-throughput phenotyping strategies brain-derived neurotrophic factor represent life-threatening challenges fasting conditions applied physiology journal protein c-deficient pups rockefeller university press oxford university press mouse mutant presenting universitรฉ de montrรฉal gene encoding pdgf-bb physiological genomics journal metabolism american journal decreased volume american physiological society normal fluid-electrolyte homeostasis human newborns presenting epithelial-mesenchymal interactions homeobox transcription factor cell-autonomous factors transcription factor krox-20 neural tube defects fetal-type acetylcholine receptor download figuredownload powerpoint neural tube closure cntfr-deficient mice die neonatal lethal phenotypes proven valuable models ureter give rise energy-related functions tbx18-deficient newborn mice ciliary neurotrophic factor atg5 homozygous mutants proper gas exchange inadequate lung clearance affecting kidney ontology

Social Networks {๐Ÿ‘}(4)

External Links {๐Ÿ”—}(1274)

Analytics and Tracking {๐Ÿ“Š}

  • Crazy Egg
  • Google Analytics
  • Google Tag Manager
  • Google Universal Analytics
  • Hotjar
  • Snowplow

Libraries {๐Ÿ“š}

  • Dropzone.js
  • Knockout.js
  • Vue.js
  • Zoom.js

Emails and Hosting {โœ‰๏ธ}

Mail Servers:

  • mx.zoho.eu
  • mx2.zoho.eu
  • mx3.zoho.eu

Name Servers:

  • josh.ns.cloudflare.com
  • zita.ns.cloudflare.com

CDN Services {๐Ÿ“ฆ}

  • Feathr
  • Informz
  • Pbgrd
  • Scite

4.12s.