DOI . ORG {}

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We began analyzing https://journals.physiology.org/doi/10.1152/physrev.00016.2017, but it redirected us to https://journals.physiology.org/doi/10.1152/physrev.00016.2017. The analysis below is for the second page.

Title[redir]:
PTX3, a Humoral Pattern Recognition Molecule, in Innate Immunity, Tissue Repair, and Cancer | Physiological Reviews | American Physiological Society
Description:
Innate immunity includes a cellular and a humoral arm. PTX3 is a fluid-phase pattern recognition molecule conserved in evolution which acts as a key component of humoral innate immunity in infections of fungal, bacterial, and viral origin. PTX3 binds conserved microbial structures and self-components under conditions of inflammation and activates effector functions (complement, phagocytosis). Moreover, it has a complex regulatory role in inflammation, such as ischemia/reperfusion injury and cancer-related inflammation, as well as in extracellular matrix organization and remodeling, with profound implications in physiology and pathology. Finally, PTX3 acts as an extrinsic oncosuppressor gene by taming tumor-promoting inflammation in murine and selected human tumors. Thus evidence suggests that PTX3 is a key homeostatic component at the crossroad of innate immunity, inflammation, tissue repair, and cancer. Dissecting the complexity of PTX3 pathophysiology and human genetics paves the way to diagnostic and therapeutic exploitation.

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Science
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🌌 Gigantic Traffic: 2M - 5M visitors per month

Based on our best estimate, this website will receive around 4,235,289 visitors per month in the current month.

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Keywords {🔍}

ptx, vol, pentraxin, cells, journal, inflammation, complement, role, innate, interaction, tissue, response, immunity, levels, patients, cancer, human, inflammatory, long, mice, research, matrix, disease, activation, immunology, humoral, protein, frontiers, clinical, gene, cell, milan, crp, molecular, increased, domain, recognition, april, university, immune, infection, italy, infections, injury, regulation, diseases, repair, medicine, expression, lung,

Topics {✒️}

small-angle x-ray scattering inter-α-inhibitor regulate crosslinking open life sciences acute graft-versus-host disease akt/nf-κb signaling elderly nursing home coiled-coil secondary structures β-jelly roll topology aps members proinflammatory program induced media/public single-cell transcriptional roadmap downstream fus/chop translocation age-related macular degeneration microtiter plate-based assay promoting pericellular fibrinolysis ventilator-induced lung injury cross-linking hyaluronic acid oocyte-derived soluble factors tlr4-dependent nf-κb exaggerated allergen-induced inflammation limiting complement-mediated inflammation tumor necrosis factor-α tnf-α-induced protein 6 single n-glycosylation site ischemia/reperfusion-induced kidney damage pro-inflammatory cytokine production appl physiol press volume 105 taming tumor-promoting inflammation physiological genomics journal enhanced complement-mediated clearance ovoalbumin-induced experimental asthma abnormal th2-skewed response chemically induced mesenchymal complement-mediated immune response long nh2-terminal domain american physiological society postischemic renal injury reflect cancer-related inflammation ptx3 activates dc-sign tailor anti-gvhd therapy long-lasting inflammatory response nh2-terminal domain acting myeloid dendritic cells ptx3-deficient mice led aps website ptx3 nh2-terminal domain congenital heart surgery fgf-dependent neo-angiogenesis

Questions {❓}

Immune response in COVID-19: what is next?

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