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DOI . ORG {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
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  6. Keywords
  7. Topics
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We began analyzing https://www.annualreviews.org/content/journals/10.1146/annurev.neuro.24.1.1121, but it redirected us to https://www.annualreviews.org/content/journals/10.1146/annurev.neuro.24.1.1121. The analysis below is for the second page.

Title[redir]:
Neurodegenerative Tauopathies | Annual Reviews
Description:
β–ͺ Abstract  The defining neuropathological characteristics of Alzheimer's disease are abundant filamentous tau lesions and deposits of fibrillar amyloid Ξ² peptides. Prominent filamentous tau inclusions and brain degeneration in the absence of Ξ²-amyloid deposits are also hallmarks of neurodegenerative tauopathies exemplified by sporadic corticobasal degeneration, progressive supranuclear palsy, and Pick's disease, as well as by hereditary frontotemporal dementia and parkinsonism linked to chromosome 17 (FTDP-17). Because multiple tau gene mutations are pathogenic for FTDP-17 and tau polymorphisms appear to be genetic risk factors for sporadic progressive supranuclear palsy and corticobasal degeneration, tau abnormalities are linked directly to the etiology and pathogenesis of neurodegenerative disease. Indeed, emerging data support the hypothesis that different tau gene mutations are pathogenic because they impair tau functions, promote tau fibrillization, or perturb tau gene splicing, thereby leading to formation of biochemically and structurally distinct aggregates of tau. Nonetheless, different members of the same kindred often exhibit diverse FTDP-17 syndromes, which suggests that additional genetic or epigenetic factors influence the phenotypic manifestations of neurodegenerative tauopathies. Although these and other hypothetical mechanisms of neurodegenerative tauopathies remain to be tested and validated, transgenic models are increasingly available for this purpose, and they will accelerate discovery of more effective therapies for neurodegenerative tauopathies and related disorders, including Alzheimer's disease.

Matching Content Categories {πŸ“š}

  • Science
  • Education
  • Non-Profit & Charity

Content Management System {πŸ“}

What CMS is doi.org built with?

Custom-built

No common CMS systems were detected on Doi.org, and no known web development framework was identified.

Traffic Estimate {πŸ“ˆ}

What is the average monthly size of doi.org audience?

πŸ™οΈ Massive Traffic: 50M - 100M visitors per month


Based on our best estimate, this website will receive around 91,205,781 visitors per month in the current month.

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How Does Doi.org Make Money? {πŸ’Έ}

We're unsure if the website is profiting.

Not every website is profit-driven; some are created to spread information or serve as an online presence. Websites can be made for many reasons. This could be one of them. Doi.org could be secretly minting cash, but we can't detect the process.

Keywords {πŸ”}

tau, neurodegenerative, annual, vol, disease, article, tauopathies, reviews, institutional, review, volume, alzheimers, filamentous, deposits, brain, degeneration, ftdp, gene, contact, usage, content, login, publications, journal, subscribe, neuroscience, john, publication, info, share, sporadic, corticobasal, progressive, supranuclear, palsy, frontotemporal, dementia, linked, mutations, pathogenic, genetic, factors, mechanisms, related, cited, cortex, circuits, attention, knowable, magazine,

Topics {βœ’οΈ}

annual reviews read annual reviews progressive supranuclear palsy tau gene mutations news careers contact neurodegenerative tauopathies exemplified neurodegenerative tauopathies remain track usage defining neuropathological characteristics Ξ²-amyloid deposits emerging data support structurally distinct aggregates epigenetic factors influence submission editorial principles impair tau functions promote tau fibrillization genetic risk factors sporadic corticobasal degeneration human brain michael hereditary frontotemporal dementia cited rss feed article preparation john duncan vol preferences neurodegenerative tauopathies 357–381 emotion circuits discover content attention system filamentous deposits policies contact publications a journal information frontotemporal dementia tau polymorphisms tau abnormalities tau protein corticobasal degeneration brain degeneration neuroscience volume 24 journals a additional genetic cohen vol strick vol ledoux vol petersen vol grain libraries brain joseph neurodegenerative disease understand skip michel goedert

Social Networks {πŸ‘}(5)

External Links {πŸ”—}(172)

Analytics and Tracking {πŸ“Š}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {πŸ“š}

  • Fancybox
  • jQuery
  • Prism.js

Emails and Hosting {βœ‰οΈ}

Mail Servers:

  • mx.zoho.eu
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Name Servers:

  • josh.ns.cloudflare.com
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CDN Services {πŸ“¦}

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