Here's how DOI.ORG makes money* and how much!

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DOI . ORG {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Doi.org Make Money
  6. Keywords
  7. Topics
  8. Social Networks
  9. External Links
  10. Analytics And Tracking
  11. Libraries
  12. Hosting Providers
  13. CDN Services

We began analyzing https://www.annualreviews.org/content/journals/10.1146/annurev-pathol-020712-163930, but it redirected us to https://www.annualreviews.org/content/journals/10.1146/annurev-pathol-020712-163930. The analysis below is for the second page.

Title[redir]:
Host Responses in Tissue Repair and Fibrosis | Annual Reviews
Description:
Myofibroblasts accumulate in the spaces between organ structures and produce extracellular matrix (ECM) proteins, including collagen I. They are the primary “effector” cells in tissue remodeling and fibrosis. Previously, leukocyte progenitors termed fibrocytes and myofibroblasts generated from epithelial cells through epithelial-to-mesenchymal transition (EMT) were considered the primary sources of ECM-producing myofibroblasts in injured tissues. However, genetic fate mapping experiments suggest that mesenchyme-derived cells, known as resident fibroblasts, and pericytes are the primary precursors of scar-forming myofibroblasts, whereas epithelial cells, endothelial cells, and myeloid leukocytes contribute to fibrogenesis predominantly by producing key fibrogenic cytokines and by promoting cell-to-cell communication. Numerous cytokines derived from T cells, macrophages, and other myeloid cell populations are important drivers of myofibroblast differentiation. Monocyte-derived cell populations are key regulators of the fibrotic process: They act as a brake on the processes driving fibrogenesis, and they dismantle and degrade established fibrosis. We discuss the origins, modes of activation, and fate of myofibroblasts in various important fibrotic diseases and describe how manipulation of macrophage activation could help ameliorate fibrosis.

Matching Content Categories {📚}

  • Science
  • Telecommunications
  • Health & Fitness

Content Management System {📝}

What CMS is doi.org built with?

Custom-built

No common CMS systems were detected on Doi.org, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of doi.org audience?

🏙️ Massive Traffic: 50M - 100M visitors per month


Based on our best estimate, this website will receive around 93,526,998 visitors per month in the current month.

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How Does Doi.org Make Money? {💸}

We find it hard to spot revenue streams.

While profit motivates many websites, others exist to inspire, entertain, or provide valuable resources. Websites have a variety of goals. And this might be one of them. Doi.org might be making money, but it's not detectable how they're doing it.

Keywords {🔍}

annual, vol, cells, article, fibrosis, myofibroblasts, review, disease, reviews, institutional, mechanisms, volume, primary, fibrotic, contact, usage, content, login, publications, journal, subscribe, pathology, tissue, publication, info, share, epithelial, fate, myeloid, fibrogenesis, key, cytokines, cell, populations, important, myofibroblast, activation, cited, cancer, impact, knowable, magazine, news, charleston, advisor, grain, copyright, cookies, track, preferencesi,

Topics {✒️}

annual reviews read monocyte-derived cell populations annual reviews numerous cytokines derived news careers contact clinical impact myeloid cell populations scar-forming myofibroblasts track usage produce extracellular matrix myeloid leukocytes contribute pierre-yves desprez resistance sreya bagchi submission editorial principles ecm-producing myofibroblasts degrade established fibrosis mesenchyme-derived cells processes driving fibrogenesis judith campisi vol disease relevance marcia cited rss feed important fibrotic diseases primary “effector” cells article preparation preferences discover content policies contact myofibroblasts generated publications a journal information key regulators disease volume 8 promoting cell cell communication journals a fibrosis jeremy ameliorate fibrosis fibrotic disease fibrogenesis predominantly important drivers sinclair vol engleman vol friedman vol croce vol grain libraries primary sources primary precursors fibrotic process endothelial cells understand skip

Social Networks {👍}(5)

External Links {🔗}(173)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {📚}

  • Fancybox
  • jQuery
  • Prism.js

Emails and Hosting {✉️}

Mail Servers:

  • mx.zoho.eu
  • mx2.zoho.eu
  • mx3.zoho.eu

Name Servers:

  • josh.ns.cloudflare.com
  • zita.ns.cloudflare.com

CDN Services {📦}

  • Cloudflare
  • Shareaholic

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