Here's how DOI.ORG makes money* and how much!

*Please read our disclaimer before using our estimates.
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DOI . ORG {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Doi.org Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. External Links
  10. Analytics And Tracking
  11. Libraries
  12. Hosting Providers

We began analyzing https://genesdev.cshlp.org/content/13/13/1742, but it redirected us to https://genesdev.cshlp.org/content/13/13/1742. The analysis below is for the second page.

Title[redir]:
p27Kip1 induction and inhibition of proliferation by the intracellular Ah receptor in developing thymus and hepatoma cells
Description:
A biweekly scientific journal publishing high-quality research in molecular biology and genetics, cancer biology, biochemistry, and related fields

Matching Content Categories {๐Ÿ“š}

  • Science
  • Education
  • Telecommunications

Content Management System {๐Ÿ“}

What CMS is doi.org built with?

Custom-built

No common CMS systems were detected on Doi.org, and no known web development framework was identified.

Traffic Estimate {๐Ÿ“ˆ}

What is the average monthly size of doi.org audience?

๐ŸŒŒ Gigantic Traffic: 2M - 5M visitors per month


Based on our best estimate, this website will receive around 3,436,036 visitors per month in the current month.

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How Does Doi.org Make Money? {๐Ÿ’ธ}

We're unsure if the website is profiting.

The purpose of some websites isn't monetary gain; they're meant to inform, educate, or foster collaboration. Everyone has unique reasons for building websites. This could be an example. Doi.org might be earning cash quietly, but we haven't detected the monetization method.

Keywords {๐Ÿ”}

cell, proliferation, author, laboratory, submit, thymus, cycle, ahr, kip, dioxins, cold, spring, harbor, press, article, home, subscribe, advertise, info, archive, contact, search, pkip, induction, inhibition, receptor, hepatoma, cells, karlsruhe, regulation, york, transcription, toxic, direct, resistant, fetal, glands, email, full, articles, issue, online, issn, skip, main, page, content, keyword, advanced, intracellular,

Topics {โœ’๏ธ}

p27kip1 cyclin/cdk inhibitor bhlh/pas transcription factor e-mail martin cell cycle regulation issue alert altered cell proliferation affects cell proliferation kip1antisense-expressing cells article ยป table developing thymus mediates dioxin toxicity ongoing protein synthesis authors contributed equally direct transcriptional target intracellular ah receptor dioxin-induced suppression kip1-deficient mice 0890-9369 online issn laboratory cell proliferation author dioxins ah receptor alteringkip1 transcription thymic glands direct mode carsten weiss andrew koff immune system toxic phenomena signaling pathways poorly understood toxic agent severe reduction thymocyte recovery received january 25 kip1 expression 76021 karlsruhe ahr induces search proliferation york induced kip1 ahr skip keyword inhibition institute genetics

Questions {โ“}

  • What's this?

External Links {๐Ÿ”—}(83)

Analytics and Tracking {๐Ÿ“Š}

  • Facebook Pixel
  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {๐Ÿ“š}

  • jQuery

Emails and Hosting {โœ‰๏ธ}

Mail Servers:

  • mx.zoho.eu
  • mx2.zoho.eu
  • mx3.zoho.eu

Name Servers:

  • josh.ns.cloudflare.com
  • zita.ns.cloudflare.com
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