DOI . ORG {
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We began analyzing https://genesdev.cshlp.org/content/10/19/2462, but it redirected us to https://genesdev.cshlp.org/content/10/19/2462. The analysis below is for the second page.
Title[redir]:
TGF-beta1 and Ha-Ras collaborate in modulating the phenotypic plasticity and invasiveness of epithelial tumor cells.
Description:
A biweekly scientific journal publishing high-quality research in molecular biology and genetics, cancer biology, biochemistry, and related fields
Title[redir]:
TGF-beta1 and Ha-Ras collaborate in modulating the phenotypic plasticity and invasiveness of epithelial tumor cells.
Description:
A biweekly scientific journal publishing high-quality research in molecular biology and genetics, cancer biology, biochemistry, and related fields
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Keywords {🔍}
cells, tgfbeta, epithelial, author, rastransformed, submit, fibroblastoid, normal, invasive, copyright, cold, spring, harbor, laboratory, press, article, home, subscribe, advertise, info, archive, contact, search, haras, tumor, vivo, mammary, treatment, growth, articles, issue, online, issn, skip, main, page, content, keyword, advanced, collaborate, modulating, phenotypic, plasticity, invasiveness, peli, rudaz, schwarz, beug, reichmann, affiliations,
Topics {✒️}
ras-transformed cells start ras-transformed cells mammary epithelial cells advanced search tgf-beta1 ha-ras collaborate activated ha-ras epithelial tumor cells epithelial-fibroblastoid conversion epithelial-stromal interactions wound healing processes tgf-beta1 brings article » table article doi tgf-beta results secrete tgf-beta 0890-9369 online issn epithelial characteristics tgf-beta1 cells maintain additional cells fibroblastoid features tgf-beta normal cells issue alert phenotypic plasticity abstract metastasis invade stroma blood vessels collagen gels treatment growth arrest growth inhibition autocrine maintenance vivo tumorigenesis invasive phenotype fibroblastoid search normal vivo invasive skip keyword modulating invasiveness oft peli rudaz schwarz beug wien
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