Here's how DOI.ORG makes money* and how much!

*Please read our disclaimer before using our estimates.
Loading...

DOI . ORG {}

Detected CMS Systems:

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Doi.org Make Money
  6. Keywords
  7. Topics
  8. Social Networks
  9. External Links
  10. Analytics And Tracking
  11. Libraries
  12. Hosting Providers
  13. CDN Services

We began analyzing https://journals.lww.com/shockjournal/fulltext/2012/07000/mechanisms_of_methicillin_resistant_staphylococcus.12.aspx, but it redirected us to https://journals.lww.com/shockjournal/fulltext/2012/07000/mechanisms_of_methicillin_resistant_staphylococcus.12.aspx. The analysis below is for the second page.

Title[redir]:
Shock
Description:
sepsis, we sought to determine whether MRSA pneumonia induces intestinal injury. FVB/N mice were subjected to MRSA or sham pneumonia and killed 24 h later. Septic animals had a marked increase in intestinal epithelial apoptosis by both hematoxylin-eosin and active caspase 3 staining. Methicillin-resistant S. aureus–induced intestinal apoptosis was associated with an increase in the expression of the proapoptotic proteins Bid and Bax and the antiapoptotic protein Bcl-xL in the mitochondrial pathway. In the receptor-mediated pathway, MRSA pneumonia induced an increase in Fas ligand but decreased protein levels of Fas, FADD, pFADD, TNF-R1, and TRADD. To assess the functional significance of these changes, MRSA pneumonia was induced in mice with genetic manipulations in proteins in either the mitochondrial or receptor-mediated pathways. Both Bid−/− mice and animals with intestine-specific overexpression of Bcl-2 had decreased intestinal apoptosis compared with wild-type animals. In contrast, Fas ligand−/− mice had no alterations in apoptosis. To determine if these findings were organism-specific, similar experiments were performed in mice subjected to Pseudomonas aeruginosa pneumonia. Pseudomonas aeruginosa induced gut apoptosis, but unlike MRSA, this was associated with increased Bcl-2 and TNF-R1 and decreased Fas. Methicillin-resistant S. aureus pneumonia thus induces organism-specific changes in intestinal apoptosis via changes in both the mitochondrial and receptor-mediated pathways, although the former may be more functionally significant....

Matching Content Categories {📚}

  • Pets
  • Science
  • Education

Content Management System {📝}

What CMS is doi.org built with?


Doi.org relies on MYBB.

Traffic Estimate {📈}

What is the average monthly size of doi.org audience?

🌍 Impressive Traffic: 500k - 1M visitors per month


Based on our best estimate, this website will receive around 600,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

check SE Ranking
check Ahrefs
check Similarweb
check Ubersuggest
check Semrush

How Does Doi.org Make Money? {💸}

We can't figure out the monetization strategy.

Earning money isn't the goal of every website; some are designed to offer support or promote social causes. People have different reasons for creating websites. This might be one such reason. Doi.org might have a hidden revenue stream, but it's not something we can detect.

Keywords {🔍}

mice, pneumonia, mrsa, apoptosis, intestinal, fig, pathway, septic, aeruginosa, epithelial, fas, compared, animals, bid, sham, mitochondrial, increased, receptormediated, expression, aureus, pathways, subjected, ligand, decreased, gut, methicillinresistant, sepsis, protein, bcl, data, bax, mediators, min, shown, apoptotic, caspase, proapoptotic, length, proliferation, rabbit, study, active, antiapoptotic, bclxl, significant, response, epithelium, cbl, cell, western,

Topics {✒️}

random high-power fields horse anti–mouse immunoglobulin ice-cold homogenization buffer aureus–induced intestinal apoptosis quantifying s-phase cells labeling s-phase cells sepsis-induced lymphocyte apoptosis final live/die decision panton-valentine leukocidin negative oriented crypt-villus units rabbit anti-fas ligand rabbit anti-bcl-xl aeruginosa pneumonia–induced sepsis mrsa pneumonia–induced sepsis bcl-xl inhibits apoptosis sterile phosphate-buffered saline intensive care unit rabbit anti–β-actin receptor-mediated pathways contribute mouse anti–tnf-r1 fas-lgld mice relates intestinal epithelial apoptosis antiapoptotic protein bcl-xl endogenous peroxidase activity gut epithelial apoptosis pneumonia-induced apoptosis e-stained jejunal sections intestinal epithelial cells mrsa-induced apoptosis bax/bcl-2 ratio directly western blot analysis pneumonia–induced sepsis panton-valentine leukocidin rabbit anti–bcl-2 rabbit anti-fas journal authors submit mrsa pneumonia induced increased epithelial apoptosis s-phase cells invasive mrsa annually septic fas-lgld mice fas-lgld mice subjected tris-buffered saline view full text septic fabpl–bcl-2 mice fabpl–bcl-2 mice subjected rabbit anti-bid showed maximal apoptosis staphylococcus aureus mrsa pneumonia induces

Social Networks {👍}(1)

External Links {🔗}(104)

Analytics and Tracking {📊}

  • Comscore
  • Google Analytics
  • Google Tag Manager

Libraries {📚}

  • Bootstrap
  • FontAwesome
  • jQuery
  • Zoom.js

Emails and Hosting {✉️}

Mail Servers:

  • mx.zoho.eu
  • mx2.zoho.eu
  • mx3.zoho.eu

Name Servers:

  • josh.ns.cloudflare.com
  • zita.ns.cloudflare.com

CDN Services {📦}

  • Cookielaw
  • Jsdelivr
  • Scite
  • Wolterskluwer

5.45s.