Here's how DOI.ORG makes money* and how much!

*Please read our disclaimer before using our estimates.
Loading...

DOI . ORG {}

Detected CMS Systems:

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Doi.org Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. Hosting Providers
  14. CDN Services

We began analyzing https://journals.lww.com/cardiovascularpharm/abstract/2012/01000/postconditioning_modulates_ischemia_damaged.13.aspx, but it redirected us to https://journals.lww.com/cardiovascularpharm/abstract/2012/01000/postconditioning_modulates_ischemia_damaged.13.aspx. The analysis below is for the second page.

Title[redir]:
Journal of Cardiovascular Pharmacology
Description:
diac injury. The reversible blockade of electron transport during ischemia prevents damage to mitochondria. We propose that the targets of PC cytoprotective signaling are mitochondria damaged by ischemia. Thus, if ischemia-mediated mitochondrial damage is prevented, PC at the onset of reperfusion will not result in additional protection. Isolated, Langendorff-perfused adult rat hearts underwent 25-minute global ischemia and 30-minute reperfusion. Amobarbital (2.5 mM) was used to reversibly inhibit electron transport during ischemia. PC (6 cycles of 10-second ischemia–reperfusion) was applied at the onset of reperfusion. Subsarcolemmal and interfibrillar mitochondria were isolated after reperfusion. Blockade of electron transport with amobarbital only during ischemia preserved oxidative phosphorylation and decreased myocardial injury. PC, after untreated ischemia, decreased cardiac injury without improvement of oxidative phosphorylation. Blockade of electron transport during ischemia or PC improved calcium tolerance and inner membrane potential in subsarcolemmal mitochondria after reperfusion. In hearts treated with amobarbital before ischemia, PC did not provide further protection. Thus, PC protects myocardium via the regulation of ischemia-damaged mitochondria during early reperfusion....

Matching Content Categories {📚}

  • Insurance
  • Health & Fitness
  • Telecommunications

Content Management System {📝}

What CMS is doi.org built with?


Doi.org is powered by MYBB.

Traffic Estimate {📈}

What is the average monthly size of doi.org audience?

🌍 Impressive Traffic: 500k - 1M visitors per month


Based on our best estimate, this website will receive around 600,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

check SE Ranking
check Ahrefs
check Similarweb
check Ubersuggest
check Semrush

How Does Doi.org Make Money? {💸}

We can't tell how the site generates income.

Earning money isn't the goal of every website; some are designed to offer support or promote social causes. People have different reasons for creating websites. This might be one such reason. Doi.org might be cashing in, but we can't detect the method they're using.

Keywords {🔍}

journal, reperfusion, ischemia, mitochondria, electron, transport, issue, cardiac, injury, access, subscribe, alerts, authors, ischemiadamaged, damage, blockade, amobarbital, log, register, articles, issues, abstract, article, postconditioning, phd, buy, mitochondrial, applied, early, onset, protection, isolated, hearts, minute, subsarcolemmal, oxidative, phosphorylation, decreased, text, subscribers, request, browse, content, submit, service, privacy, policy, logo, advanced, search,

Topics {✒️}

ischemia-mediated mitochondrial damage journal authors submit ischemia-damaged mitochondria full text access decreases cardiac injury decrease cardiac injury decreased cardiac injury ischemia prevents damage ischemic postconditioning decreased myocardial injury artificial intelligence training mitochondria damaged pc cytoprotective signaling pc protects myocardium etoc alerts hearts treated service request 800-638-3030 journal tables register subscribe 30-minute reperfusion individual subscribers log article oxidative phosphorylation untreated ischemia electron transport interfibrillar mitochondria issue journal authors ischemia–reperfusion subsarcolemmal mitochondria damage persists website subscribe center submit melanie bs† ying ba cardiovascular pharmacology 59 finding suggests membrane potential email inbox rights reserved data mining ischemia qun md heng md mitochondria reperfusion chen ludovic phd† early reperfusion additional protection

Questions {❓}

  • Not a Subscriber?

External Links {🔗}(57)

Analytics and Tracking {📊}

  • Comscore
  • Google Analytics
  • Google Tag Manager

Libraries {📚}

  • Bootstrap
  • FontAwesome
  • jQuery
  • Zoom.js

Emails and Hosting {✉️}

Mail Servers:

  • mx.zoho.eu
  • mx2.zoho.eu
  • mx3.zoho.eu

Name Servers:

  • josh.ns.cloudflare.com
  • zita.ns.cloudflare.com

CDN Services {📦}

  • Cookielaw
  • Jsdelivr
  • Scite
  • Wolterskluwer

6.41s.