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Detected CMS Systems:

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Doi.org Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
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We began analyzing https://journals.lww.com/immunotherapy-journal/abstract/2010/02000/tumor_secretion_of_vegf_induces_endothelial_cells.2.aspx, but it redirected us to https://journals.lww.com/immunotherapy-journal/abstract/2010/02000/tumor_secretion_of_vegf_induces_endothelial_cells.2.aspx. The analysis below is for the second page.

Title[redir]:
Journal of Immunotherapy
Description:
ess T-cell functions in vitro. The current studies examined in vitro and in vivo the mechanism by which tumors induce the formation of suppressor endothelial cells and the means by which suppressor endothelial cells disrupt T-cell functions. In vitro studies demonstrated that inhibition of tumor-derived VEGF with neutralizing antibodies or treatment of endothelial cells with the VEGF receptor tyrosine kinase inhibitor, SU5416, prevented endothelial cells from being induced to suppress T-cell functions. Treatment of tumor-bearing mice with SU5416 blocked the development of endothelial cells that are suppressive to CD4+ and CD8+ T-cell functions. We next examined the role of suppressor endothelial cell-derived PGE2 in the inhibition of T-cell functions. Abrogation of endothelial cell PGE2 production in vitro with indomethacin prevented tumor-conditioned media from stimulating endothelial cell production of immune inhibitory activity toward T-cell functions. Similar treatment of endothelial cells from lungs of tumor-bearing mice blocked their capacity to produce T-cell-inhibitory mediators. These studies demonstrate that tumor-derived VEGF induces endothelial cells to upregulate production of PGE2 which, in turn, leads to suppression of T-cell functions....

Matching Content Categories {📚}

  • Science
  • Health & Fitness
  • Education

Content Management System {📝}

What CMS is doi.org built with?


Doi.org operates using MYBB.

Traffic Estimate {📈}

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🌍 Impressive Traffic: 500k - 1M visitors per month


Based on our best estimate, this website will receive around 600,019 visitors per month in the current month.
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We're unsure if the website is profiting.

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Keywords {🔍}

endothelial, cells, journal, functions, tcell, studies, issue, vegf, cell, production, pge, vitro, access, subscribe, alerts, authors, abstract, suppress, immune, examined, suppressor, treatment, log, register, logo, articles, issues, previous, induces, buy, role, suppression, induced, current, inhibition, tumorderived, prevented, tumorbearing, mice, blocked, text, subscribers, request, browse, content, submit, service, privacy, policy, secondary,

Topics {✒️}

produce t-cell-inhibitory mediators cd8+ t-cell functions suppress t-cell functions tumor-bearing mice blocked tumor-induced immune suppression tumor-derived vegf suppressor endothelial cells prevented endothelial cells t-cell functions tumor-bearing mice immune cell functions immune inhibitory activity full text access journal authors submit current studies examined lewis lung carcinoma artificial intelligence training etoc alerts endothelial cells cell functions vitro studies demonstrated service request 800-638-3030 register subscribe individual subscribers log journal tables issue previous studies pge2 mulligan studies demonstrate website subscribe upregulate production journal su5416 blocked center submit potent regulators laboratory showed tumors induce neutralizing antibodies email inbox rights reserved data mining induced similar treatment suppress pge2 content production suppression authors log

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Social Networks {👍}(1)

External Links {🔗}(58)

Analytics and Tracking {📊}

  • Comscore
  • Google Analytics
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Libraries {📚}

  • Bootstrap
  • FontAwesome
  • jQuery
  • Zoom.js

Emails and Hosting {✉️}

Mail Servers:

  • mx.zoho.eu
  • mx2.zoho.eu
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Name Servers:

  • josh.ns.cloudflare.com
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CDN Services {📦}

  • Cookielaw
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