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DOI . ORG {}

Detected CMS Systems:

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Doi.org Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. Hosting Providers
  14. CDN Services

We began analyzing https://journals.lww.com/ajsp/abstract/2007/01000/brca2_mutation_associated_breast_cancers_exhibit_a.15.aspx, but it redirected us to https://journals.lww.com/ajsp/abstract/2007/01000/brca2_mutation_associated_breast_cancers_exhibit_a.15.aspx. The analysis below is for the second page.

Title[redir]:
The American Journal of Surgical Pathology
Description:
mors accrued through the Breast Cancer Family Registry. We examined the tumor morphology and hormone receptor status in 157 hereditary breast cancers with germline mutations in BRCA2 and 314 control tumors negative for BRCA1 and BRCA2 mutations that were matched for age and ethnicity. Tissue microarrays were constructed from 64 BRCA2-associated and 185 control tumors. Tissue microarray sections were examined for HER2/neu protein overexpression, p53 status and the expression of basal markers, luminal markers, cyclin D1, bcl2, and MIB1 by immunohistochemistry. The majority of BRCA2-associated tumors and control tumors were invasive ductal, no special-type tumors. In contrast to control tumors, BRCA2-associated cancers were more likely to be high grade (P<0.0001) and to have pushing tumor margins (P=0.0005). Adjusting for grade, BRCA2-associated tumors were more often estrogen receptor positive (P=0.008) and exhibited a luminal phenotype (P=0.003). They were less likely than controls to express the basal cytokeratin CK5 (P=0.03) or to overexpress HER2/neu protein (P=0.06). There was no difference in p53, bcl2, MIB1, or cyclin D1 expression between BRCA2-associated and control tumors. We have demonstrated, in the largest series of BRCA2-associated breast cancers studied to date, that these tumors are predominantly high-grade invasive ductal carcinomas of no special type and they demonstrate a luminal phenotype despite their high histologic grade....

Matching Content Categories {📚}

  • Education
  • TV
  • Politics

Content Management System {📝}

What CMS is doi.org built with?


Doi.org is built with MYBB.

Traffic Estimate {📈}

What is the average monthly size of doi.org audience?

🌍 Impressive Traffic: 500k - 1M visitors per month


Based on our best estimate, this website will receive around 600,420 visitors per month in the current month.

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How Does Doi.org Make Money? {💸}

We don't see any clear sign of profit-making.

Earning money isn't the goal of every website; some are designed to offer support or promote social causes. People have different reasons for creating websites. This might be one such reason. Doi.org might be earning cash quietly, but we haven't detected the monetization method.

Keywords {🔍}

phd, tumors, journal, brcaassociated, breast, cancers, control, phenotype, issue, brca, access, subscribe, alerts, articles, authors, abstract, tissue, luminal, grade, log, register, logo, issues, morphology, molecular, microarrays, john, cancer, family, registry, buy, examined, tumor, receptor, status, mutations, herneu, protein, expression, basal, markers, cyclin, bcl, mib, invasive, ductal, high, text, subscribers, request,

Topics {✒️}

her2/neu protein overexpression overexpress her2/neu protein 0f buy abstract journal authors submit full text access high histologic grade estrogen receptor positive artificial intelligence training tissue microarray sections hormone receptor status pushing tumor margins basal cytokeratin ck5 breast cancers exhibit breast cancers studied distinguishing phenotype based invasive ductal etoc alerts comprehensively characterize brca2 service request 800-638-3030 register subscribe cyclin d1 expression individual subscribers log special-type tumors high grade american journal journal tables issue breast cancers tissue microarrays cyclin d1 special type website subscribe journal basal markers center submit 185 control tumors control tumors molecular profiles o'malley surgical pathology 31 distinct morphologic molecular phenotype tumor morphology germline mutations p53 status luminal markers luminal phenotype largest series email inbox rights reserved

Questions {❓}

  • Not a Subscriber?

Social Networks {👍}(1)

External Links {🔗}(58)

Analytics and Tracking {📊}

  • Comscore
  • Google Analytics
  • Google Tag Manager

Libraries {📚}

  • Bootstrap
  • FontAwesome
  • Foundation
  • jQuery
  • Zoom.js

Emails and Hosting {✉️}

Mail Servers:

  • mx.zoho.eu
  • mx2.zoho.eu
  • mx3.zoho.eu

Name Servers:

  • josh.ns.cloudflare.com
  • zita.ns.cloudflare.com

CDN Services {📦}

  • Cookielaw
  • Jsdelivr
  • Scite
  • Wolterskluwer

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