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DOI . ORG {}

Detected CMS Systems:

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Doi.org Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. External Links
  10. Analytics And Tracking
  11. Libraries
  12. Hosting Providers
  13. CDN Services

We began analyzing https://journals.lww.com/anti-cancerdrugs/abstract/2007/06000/peroxisome_proliferator_activated_receptor__.2.aspx, but it redirected us to https://journals.lww.com/anti-cancerdrugs/abstract/2007/06000/peroxisome_proliferator_activated_receptor__.2.aspx. The analysis below is for the second page.

Title[redir]:
Anti-Cancer Drugs
Description:
some proliferator-activated receptor-γ antagonists, T0070907 and GW9662, on a panel of hematopoietic and epithelial cell lines. The peroxisome proliferator-activated receptor-γ antagonists and a reference agonist (pioglitazone) were tested in an in-vitro proliferation assay on a panel of seven hematopoietic and nine epithelial cancer cell lines, some of which are chemoresistant. Peroxisome proliferator-activated receptor-γ expression was measured by immunoblotting, as was the effect of treatment with these agents on peroxisome proliferator-activated receptor-γ levels. The effect of exogenous interleukin-6, an antiapoptotic cytokine, on growth inhibition was evaluated as well as the apoptotic effects of these drugs. The peroxisome proliferator-activated receptor-γ antagonists showed significantly greater potency on all cell lines (IC50s of 3.2–29.7 versus 26.5–78.7 μmol/l for pioglitazone) and greater maximum growth inhibition. Peroxisome proliferator-activated receptor-γ levels did not correlate with growth inhibition in this panel of cell lines. Combinations of peroxisome proliferator-activated receptor-γ antagonists and the agonist actually showed schedule-dependent increases in growth inhibition. Exogenous interleukin-6 did not induce resistance to these agents. Both the antagonists and the agonist induced apoptosis, but only the former drugs showed caspase dependence. These two peroxisome proliferator-activated receptor-γ antagonists have significantly more potent in-vitro antiproliferative effects versus hematopoietic and epithelial cancer cell lines. This effect does not correlate with peroxisome proliferator-activated receptor-γ levels, suggesting alternative mechanisms or other targets of action. These findings support further translational studies to explore the mechanism of action and therapeutic potential of this class of agents....

Matching Content Categories {📚}

  • Social Networks
  • Telecommunications
  • Education

Content Management System {📝}

What CMS is doi.org built with?


Doi.org relies on MYBB.

Traffic Estimate {📈}

What is the average monthly size of doi.org audience?

🏙️ Massive Traffic: 50M - 100M visitors per month


Based on our best estimate, this website will receive around 91,705,781 visitors per month in the current month.

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How Does Doi.org Make Money? {💸}

We see no obvious way the site makes money.

While many websites aim to make money, others are created to share knowledge or showcase creativity. People build websites for various reasons. This could be one of them. Doi.org could have a money-making trick up its sleeve, but it's undetectable for now.

Keywords {🔍}

peroxisome, proliferatoractivated, receptorγ, journal, antagonists, cell, lines, issue, effects, hematopoietic, epithelial, growth, inhibition, access, subscribe, alerts, submit, authors, abstract, versus, cancer, anticancer, drugs, panel, agonist, effect, agents, levels, showed, log, register, articles, issues, preclinical, reports, potent, antiproliferative, buy, studies, evaluated, pioglitazone, invitro, exogenous, interleukin, significantly, greater, correlate, action, text, subscribers,

Topics {✒️}

showed schedule-dependent increases epithelial cell lines journal authors submit vitro proliferation assay full text access area address agonists suggesting alternative mechanisms artificial intelligence training clinical anticancer activity anticancer effects agonist induced apoptosis apoptotic effects preclinical reports potent service request 800-638-3030 register subscribe antagonists individual subscribers log cell lines 7 versus 26 journal tables significantly etoc alerts growth inhibition website subscribe center submit journal reference agonist exogenous interleukin-6 antiapoptotic cytokine induce resistance findings support therapeutic potential email inbox rights reserved data mining issue translational studies drugs authors content log hematopoietic agonist subscribers text studies jack castillo

Questions {❓}

  • Not a Subscriber?

External Links {🔗}(56)

Analytics and Tracking {📊}

  • Comscore
  • Google Analytics
  • Google Tag Manager

Libraries {📚}

  • Bootstrap
  • FontAwesome
  • jQuery
  • Zoom.js

Emails and Hosting {✉️}

Mail Servers:

  • mx.zoho.eu
  • mx2.zoho.eu
  • mx3.zoho.eu

Name Servers:

  • josh.ns.cloudflare.com
  • zita.ns.cloudflare.com

CDN Services {📦}

  • Cookielaw
  • Jsdelivr
  • Scite
  • Wolterskluwer

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