Here's how DOI.ORG makes money* and how much!

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DOI . ORG {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Doi.org Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. Hosting Providers

We began analyzing https://pnas.org/doi/full/10.1073/pnas.0910009106, but it redirected us to https://pnas.org/doi/full/10.1073/pnas.0910009106. The analysis below is for the second page.

Title[redir]:
p21CIP1 attenuates Ras- and c-Myc-dependent breast tumor epithelial mesenchymal transition and cancer stem cell-like gene expression in vivo | PNAS
Description:
p21CIP1/WAF1 is a downstream effector of tumor suppressors and functions as a cyclin-dependent kinase inhibitor to block cellular proliferation. Breast tumors may derive from self-renewing tumor-in...

Matching Content Categories {📚}

  • Science
  • Education
  • Telecommunications

Content Management System {📝}

What CMS is doi.org built with?

Custom-built

No common CMS systems were detected on Doi.org, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of doi.org audience?

🚦 Initial Traffic: less than 1k visitors per month


Based on our best estimate, this website will receive around 19 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

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How Does Doi.org Make Money? {💸}

We find it hard to spot revenue streams.

Websites don't always need to be profitable; some serve as platforms for education or personal expression. Websites can serve multiple purposes. And this might be one of them. Doi.org could be getting rich in stealth mode, or the way it's monetizing isn't detectable.

Keywords {🔍}

pcip, cells, cancer, fig, cell, stem, expression, twist, emt, crossref, pubmed, google, scholar, mammary, tumors, tumor, gene, center, cmyc, markers, jefferson, ras, epithelial, mesenchymal, mcfaras, mcfacmyc, view, analysis, articles, biology, breast, features, haras, genes, department, university, author, increased, mice, induced, mcfa, kimmel, thomas, philadelphia, article, human, transgenic, ecadherin, data, sirna,

Topics {✒️}

org/cgi/content/full/0910009106/dcsupplemental tgf-beta/smad gene targets lesser extent mcf10a-c-myc e-box binding site mcf10a-c-myc cells treated mcf10a-c-myc cells transduced mcf10a-c-myc-induced emt cd44+/cd24− mcf10a-ras cells social media facebook cyclin-dependent kinase inhibitor supporting information online mcf10a-c-myc transformed c-myc expression induced c-myc induce epithelial mcf10a-c-myc treated genome-wide expression profiles mammary-targeted ha-ras c-myc-induced tumorigenesis p18inkc-deficiency-dependent tumorigenesis c-myc-induced features ras-dependent cellular migration jan-willem van ittersum c-myc-induced tumors oncogene-induced mammary tumors mammary oncogene-induced tumors c-myc induce features c-myc mammary tumors mcf10a-c-myc cells p21cip1 attenuated ha-ras c-myc-induced emt hsc-long-term hscs normal mammary gland cd44+/cd24−/low cells mmtv-c-myc/p21cip−/− human mammary glands renewing tumor-initiating cells stem/progenitor cell properties c-myc-p21cip1−/− tumors mesenchymal marker n-cadherin protein microarray-based analysis e-cadherin promoter activity p21cip1-induced emt pathways epithelial mesenchymal transition mcf10a-ras cellular migration demonstrated p21cip1-repressed genes ha-ras enhanced expression promotes ras-tumor progression cell surface markers comparing p21cip1-regulated genes gene expression profiles

Questions {❓}

  • H Peinado, D Olmeda, A Cano, Snail, Zeb and bHLH factors in tumor progression: An alliance against the epithelial phenotype?

External Links {🔗}(312)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Tag Manager

Libraries {📚}

  • Dropzone.js
  • Zoom.js

Emails and Hosting {✉️}

Mail Servers:

  • mx.zoho.eu
  • mx2.zoho.eu
  • mx3.zoho.eu

Name Servers:

  • josh.ns.cloudflare.com
  • zita.ns.cloudflare.com
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