Here's how DOI.ORG makes money* and how much!

*Please read our disclaimer before using our estimates.
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DOI . ORG {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Doi.org Make Money
  6. Keywords
  7. Topics
  8. Social Networks
  9. External Links
  10. Analytics And Tracking
  11. Libraries
  12. Hosting Providers

We began analyzing https://pnas.org/doi/full/10.1073/pnas.1003428107, but it redirected us to https://pnas.org/doi/full/10.1073/pnas.1003428107. The analysis below is for the second page.

Title[redir]:
Mitochondrial metabolism and ROS generation are essential for Kras-mediated tumorigenicity | PNAS
Description:
Otto Warburg

Matching Content Categories {📚}

  • Science
  • Education
  • Health & Fitness

Content Management System {📝}

What CMS is doi.org built with?

Custom-built

No common CMS systems were detected on Doi.org, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of doi.org audience?

🚦 Initial Traffic: less than 1k visitors per month


Based on our best estimate, this website will receive around 19 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

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How Does Doi.org Make Money? {💸}

We don't see any clear sign of profit-making.

Websites don't always need to be profitable; some serve as platforms for education or personal expression. Websites can serve multiple purposes. And this might be one of them. Doi.org could be getting rich in stealth mode, or the way it's monetizing isn't detectable.

Keywords {🔍}

cells, mitochondrial, growth, fig, ros, cell, tumor, metabolism, crossref, glycolysis, google, scholar, proliferation, glutamine, cancer, anchorageindependent, cybrids, pubmed, required, levels, pathway, agar, soft, tca, shrna, generation, results, cycle, complex, hct, δcytochrome, view, oxidative, glucose, iii, phosphorylation, phosphate, mice, analysis, lslkras, articles, research, aerobic, atp, pentose, erk, presence, role, control, protein,

Topics {✒️}

lsl-kras g12d mice kras-induced anchorage-independent growth oncogene-induced anchorage-independent growth lsl-krasg12d 3t3 mefs otto warburg observed methods full methods lslkrasg12d3t3 +cre/immortalized lslkrasg12d oncogenic kras-induced growth diminishes anchorage-independent growth colon laurent peyrin-biroulet lsl-kras g12d termed lsl-krasg12d 3t3mefs attenuate anchorage-independent growth subsequent anchorage-independent growth prevents anchorage-independent growth anchorage-independent manner suggests lox-stop-lox krasg12 jan-willem van ittersum kras-induced cell proliferation lsl-kras g12dfl/+ oncogenic kras-transformed cells cell permeable alpha-ketoglutarate 48-hours post-treatment suggesting glucose-free media enriched oncogene-induced transformation depends regulating oncogene-induced tumorigenicity krasv12-p53dn cells/immortalized p53dn wild-type kras allele kras-mediated tumorigenicity proceedings myr-akt-p53dn mefs oncogenic kras induced extracellular signal-regulated kinase pnas direct submission dmk-induced colony growth kras-induced growth complex iii-generated ros xenograft tumor formation lsl-krasg12d allele superoxide dismutase mimetics wild-type 143b cybrids anchorage-independent growth sophisticated auditory–motor learning anchorage-independent manner anchorage independent-manner authors declare kras-mediated tumorigenicity oncogenic kras mutation oncogene-induced tumorigenicity citation information map kinase pathway

External Links {🔗}(299)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Tag Manager

Libraries {📚}

  • Dropzone.js
  • Zoom.js

Emails and Hosting {✉️}

Mail Servers:

  • mx.zoho.eu
  • mx2.zoho.eu
  • mx3.zoho.eu

Name Servers:

  • josh.ns.cloudflare.com
  • zita.ns.cloudflare.com
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