Here's how DOI.ORG makes money* and how much!

*Please read our disclaimer before using our estimates.
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DOI . ORG {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Doi.org Make Money
  6. Keywords
  7. Topics
  8. Social Networks
  9. External Links
  10. Analytics And Tracking
  11. Libraries
  12. Hosting Providers

We began analyzing https://pnas.org/doi/full/10.1073/pnas.0805009105, but it redirected us to https://pnas.org/doi/full/10.1073/pnas.0805009105. The analysis below is for the second page.

Title[redir]:
Brk is coamplified with ErbB2 to promote proliferation in breast cancer | PNAS
Description:
Amplification of the receptor tyrosine kinase ErbB2 is frequently observed in breast cancer. Amplification of erbB2 is also associated with multiple genomic gains and losses; however, the importanc...

Matching Content Categories {📚}

  • Science
  • Education
  • Telecommunications

Content Management System {📝}

What CMS is doi.org built with?

Custom-built

No common CMS systems were detected on Doi.org, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of doi.org audience?

🚦 Initial Traffic: less than 1k visitors per month


Based on our best estimate, this website will receive around 19 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

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How Does Doi.org Make Money? {💸}

We're unsure if the website is profiting.

Websites don't always need to be profitable; some serve as platforms for education or personal expression. Websites can serve multiple purposes. And this might be one of them. Doi.org might have a hidden revenue stream, but it's not something we can detect.

Keywords {🔍}

brk, erbb, cells, breast, cell, proliferation, cancer, kinase, scholar, crossref, pubmed, google, expression, activation, fig, tumors, mammary, erbbinduced, activity, signaling, tyrosine, pathway, epithelial, cold, spring, harbor, cyclin, levels, coexpression, mcfa, min, view, human, tumor, observed, overexpression, lapatinib, determine, acbbrk, cycle, mice, articles, article, information, growth, expressing, increase, data, generated, analysis,

Topics {✒️}

org/cgi/content/full/0805009105/dcsupplemental mcf-10a-based cell lines serum-free dmem/f12 medium social media facebook anne-lise borresen-dale jan-willem van ittersum median-polished log ratios clinically relevant link ligand-independent receptor homodimerization anti-rabbit antibody conjugated supporting information online increases anchorage-independent growth parental erbb2-expressing cells receptor protein-tyrosine kinase heregulin-induced cell proliferation β-actin immunoblot serves anti-fade prolong reagent inhibit erbb-induced proliferation cold spring harbor regulates heregulin-induced activation inhibit erbb2-induced proliferation erbb2-induced cell proliferation wild-type erbb2 receptors horizontal dotted lines anti-erbb2 therapeutic strategies alexa fluor dyes promote egf-independent proliferation sophisticated auditory–motor learning supporting information induce egf-independent proliferation erbb2-postive breast cancer immortalized egf-dependent erbb2-brk signaling axis full-length erbb2 brk-transplanted mice started growth factor-starved cells erbb2-induced maximal activation cleared fat pads authors declare tumor cell–specific function solid straight line promotes cell proliferation mammary epithelial cells promote erbb2-induced activation c-terminal tyrosine 447 murine mammary tumorigenesis epidermal growth factor enhances downstream signaling librarian recommend pnas p38 map kinases

External Links {🔗}(311)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Tag Manager

Libraries {📚}

  • Dropzone.js
  • Zoom.js

Emails and Hosting {✉️}

Mail Servers:

  • mx.zoho.eu
  • mx2.zoho.eu
  • mx3.zoho.eu

Name Servers:

  • josh.ns.cloudflare.com
  • zita.ns.cloudflare.com
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