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DOI . ORG {}

Detected CMS Systems:

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Doi.org Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. External Links
  10. Analytics And Tracking
  11. Libraries
  12. Hosting Providers
  13. CDN Services

We began analyzing https://journals.lww.com/hep/abstract/2002/09000/effect_of_tauroursodeoxycholic_acid_on_endoplasmic.10.aspx, but it redirected us to https://journals.lww.com/hep/abstract/2002/09000/effect_of_tauroursodeoxycholic_acid_on_endoplasmic.10.aspx. The analysis below is for the second page.

Title[redir]:
Hepatology
Description:
n calcium homeostasis in an ER stress-induced model of apoptosis. The human liver-derived cell line, Huh7, was treated with thapsigargin (TG) to induce ER stress. Typical morphologic changes of ER stress preceded development of apoptotic changes, including DNA fragmentation and cleavage of poly (adenosine diphosphate-ribose) polymerase (PARP), as well as activation of caspase-3 and -7. Elevation of intracellular calcium levels without loss of mitochondrial membrane potential (MMP) was shown using Fluo-3/Fura-red labeling and flow cytometry, and confirmed by induction of Bip/GRP78, a calcium-dependent chaperon of ER lumen. These changes were accompanied by procaspase-12 processing. TUDCA abolished TG-induced markers of ER stress; reduced calcium efflux, induction of Bip/GRP78, and caspase-12 activation; and subsequently inhibited activation of effector caspases and apoptosis. In conclusion, we propose that mitochondria play a secondary role in ER-mediated apoptosis and that TUDCA prevents apoptosis by blocking a calcium-mediated apoptotic pathway as well as caspase-12 activation. This novel mechanism of TUDCA action suggests new intervention methods for ER stress-induced liver disease. E-mail:[email protected] *Address reprint requests to: VAMC (151B), 2002 Holcombe Blvd., Houston, US 77030. hyphen;28871, Alcalá de Henares, Spain. fax: 713-794-7870. Received February 07, 2002; Accepted June 11, 2002; previously published online December 30, 2003 Copyright © 2002 American Association for the Study of Liver Diseases....

Matching Content Categories {📚}

  • Health & Fitness
  • Video & Online Content
  • Non-Profit & Charity

Content Management System {📝}

What CMS is doi.org built with?


Doi.org utilizes MYBB.

Traffic Estimate {📈}

What is the average monthly size of doi.org audience?

🏙️ Massive Traffic: 50M - 100M visitors per month


Based on our best estimate, this website will receive around 76,504,851 visitors per month in the current month.

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How Does Doi.org Make Money? {💸}

We see no obvious way the site makes money.

Earning money isn't the goal of every website; some are designed to offer support or promote social causes. People have different reasons for creating websites. This might be one such reason. Doi.org might have a hidden revenue stream, but it's not something we can detect.

Keywords {🔍}

activation, aasld, liver, caspase, log, journal, stress, tudca, apoptosis, access, hepatology, subscribe, issue, alerts, abstract, stressinduced, apoptotic, calcium, disease, register, member, secondary, logo, articles, browse, submit, effect, tauroursodeoxycholic, acid, endoplasmic, reticulum, buy, mitochondrial, pathway, role, induction, bipgrp, copyright, american, association, study, diseases, text, subscribers, members, request, content, service, usa, privacy,

Topics {✒️}

fluo-3/fura-red labeling er stress-induced model aasld members log calcium-mediated apoptotic pathway open access policy individual subscribers log liver diseases induce er stress institutional users access endoplasmic reticulum including dna fragmentation adenosine diphosphate-ribose intracellular calcium levels calcium-dependent chaperon reduced calcium efflux tudca action suggests artificial intelligence training subsequently inhibited activation common apoptotic pathways mitochondrial membrane potential er-mediated apoptosis full text access tudca prevents apoptosis secondary role journal tables er stress register subscribe service request 800-638-3030 caspase-12 activation calcium homeostasis er lumen journal website subscribe mitochondrial damage tauroursodeoxycholic acid death receptors typical morphologic flow cytometry bip/grp78 procaspase-12 processing effector caspases mitochondria play intervention methods email inbox rights reserved data mining stress issue content er

Questions {❓}

  • AASLD Member?
  • Not a Subscriber?

External Links {🔗}(66)

Analytics and Tracking {📊}

  • Comscore
  • Google Analytics
  • Google Tag Manager
  • Mouseflow

Libraries {📚}

  • Bootstrap
  • FontAwesome
  • jQuery
  • Video.js
  • Zoom.js

Emails and Hosting {✉️}

Mail Servers:

  • mx.zoho.eu
  • mx2.zoho.eu
  • mx3.zoho.eu

Name Servers:

  • josh.ns.cloudflare.com
  • zita.ns.cloudflare.com

CDN Services {📦}

  • Cookielaw
  • Jsdelivr
  • Mouseflow
  • Scite
  • Wolterskluwer

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