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Keywords {🔍}

pubmed, article, nature, google, scholar, tumour, cancer, cas, data, cell, tumours, cells, heterogeneity, clonal, growth, central, extended, polyak, model, access, figure, subclonal, kornelia, evolution, ads, massachusetts, usa, fig, content, subclones, mathematical, cookies, information, marusyk, tumor, boston, initiated, ilrα, privacy, noncellautonomous, altrock, dynamics, institute, research, department, harvard, mixtures, parental, supplementary, doxorubicin,

Topics {✒️}

nature portfolio permissions reprints privacy policy limit cancer development advertising shannon index author information authors social media stat1 mediate il-11-dependent dana-farber cancer institute wnt-driven mammary cancers nature rev author correspondence nature med human-specific reference amplicon sample kolmogorov–smirnov test fluorescent-activated cell sorting enabling inter-clonal interactions nature 481 nature 501 nature 469 nature 463 nature 508 nature 514 nature springerlink instant access permissions intra-tumour clonal heterogeneity tumour volume–density relation extended data figures total tumour size tumour-growth promoting effects il11-expressing clones derived supplementary references privacy article marusyk green fluorescent protein issue learn metastasis competent subclones competing financial interests 3d culture systems explore content subscription content branched evolution revealed cell-autonomous driving single-cell transcriptomics cell death occurs cell-autonomous mechanism vanessa almendro overcoming environmental constraints

Questions {❓}

• Challenging the axiom: does the occurrence of oncogenic mutations truly limit cancer development with age?
• Why don't we get more cancer?

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